Ventricular arrhythmias Flashcards

1
Q

3 types of vent arrythms

A
  1. V. Tachy
    - monomorphic
    - poly morphic
  2. V. flutter
  3. V. fib
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2
Q

what is most important question to ask

A

does VT occur in setting of a normal heart

- organized VAs almost never caused by ischemia

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3
Q

4 things seen on ECG in monomorphic VT

A
  1. wide complex, originated from Vent
  2. 120-250bpm, at least 3 beats
  3. sustained = > 30 secs
  4. consitent beat-beat QRS morphology
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4
Q

2 main causes of VT

A
  1. reentry
    - scarring
    - His-purk disease
    - idiopathic
  2. automatic or triggered
    - peri-infarction
    - normal heart - catecholamine sensitive
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5
Q

3 methods to diff. VT from SVT w/aberancy

A
  1. HX
  2. PhX
  3. ECG
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6
Q

what are Hx predictors of VT

A
  1. age>40
  2. history of CAD, previous MI
  3. previous VT
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7
Q

2 Phx signs

A
  1. hemodyanamic stability
    - not too helpful, because young ppl can stand it
  2. AV dissociation
    - cannon A waves in JVP
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8
Q

5 main ECG criteria to differentiate

A
  1. AV dissociation
  2. rate - not helpful
  3. QRS axis
  4. QRS duration
  5. QRS morpho
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9
Q

what does AV dissociation mean and 3 things to look for

A

diagnostic of VT

  1. fusion beats - part of one uses H/P system
  2. capture beats - one fully down H/P
  3. p -waves marching
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10
Q

what does axis tell you

A

high prob of VT if axis is right superior (+ in AVR)

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11
Q

what does QRS duration tell you

A
  • > 140ms is 100 spec. for VT
  • wider in myocardial disease, metabolic abnormal
  • narrower with focus close to H/P system
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12
Q

what are 3 questions about QRS morphology

A

ANY yes = VT, all 3 NO = SVT

  1. absence of RS in all precorial leads
  2. onset R to nadir S > 100ms in any V lead
  3. morphology criteria in both V1-2 and V6
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13
Q

what are morphology criteria

A
LBBB like QRS
V1or2
- R>30ms wide
- >60ms to nadir of S
- notched S
V6
- QR or QS
- monophasic R

Need both 1-2 and 6 to be met

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14
Q

3 points to remember

A
  1. in WCT, Vt is far more common than SVT
  2. if structrual heart diease present, PPV for Vt = 95
  3. can make correct diagnosis in most patients if apply criteria
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15
Q

what is poly morphic VT

A

not a nice repeating wave- need a code blue

  • ischemia most common cause
  • may be assoc. with long QT, but not always
  • signature is usually sinus rythm and onset of poly VT with short couple PVC (
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16
Q

3 other important cause of poly VT

A
  1. bradycard
  2. hypokalemia
  3. hypomagnesia
17
Q

what is long QT syndrome

A
  • genetic defect in K channed
  • prolonged repolarization
    QT>450 in men
    >460 in women
18
Q

3 ECG signs of V flutter

A
  1. monomorphic
  2. rate>250
  3. usually hemo unstable and treated as fib
19
Q

3 ECG signs of V fib

A
  1. polymorphic
  2. rate > 250
  3. can be very fine
20
Q

Tx of VT

A

if unstable -shock
- do NOT give AV node blockade to PTs with WCT unless previous documented benefit or are sure this is not pre-ecxited Afib

21
Q

5 ER scenarios

A
  1. mono VT with structural HD
  2. poly VT without long QT
  3. mono VT without structural HD
  4. poly VT with long QT
  5. vent. fib/flutter
22
Q

what is and TX of xmono VT with structural HD

A
  • related to reentryaround scar of old MI
  • shock, amiodarone
  • need to R/O ischemia as precipitant
23
Q

what is and TX of poly VT without long QT

A

may be ischemia, lyte abnormal, catecholamine
TX
- amiodarone, IV Mg, shock
- then ischemia mgmt and eval

24
Q

what is and TX of xmono VT without structural HD

A
  • unlikely ischemia related
  • most likely idiopathic benign
    TX
  • very rare
  • procainamide and or amiodarone
25
Q

what is and TX of poly VT with long QT

A
  • torsades de pointes
  • may be secondary to drugs (many) or genetics
    Tx
  • IV Mg
  • correction of lytes
  • removal of meds
  • isoproterenol
26
Q

Tx of flutter/lib

A
shock
treat underlying cause
- metabolic
- drugs
- ischemia