mitral stenosis and regurg Flashcards

1
Q

what is etiology for mitral stenosis

A
  • almost always rheumatic

- murmur appears 20 years later

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2
Q

what happens in rheumatic

A
  • fibrotic rigid mitral leaflets
  • fused valvular commissures
  • thick short fused chordae
  • calcification of leaflets
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3
Q

what is patho in mitral stenosis

A
  • L atrial Pressure
  • pulm HT and interstitial/alveolar pulm edema due to back transmission
  • large atrium = thrombus risk, Afib risk
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4
Q

what is use of cath

A

normal Pulm wedge pressure = 6-12mmHg
> 25 causes transudation
- incr. pulm venous and cap pressure

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5
Q

what happen on Left heart

A
  • reduced mitral inflow

- low output

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6
Q

4 possible complications

A
  1. pulm edema and hemoptysis
  2. A fib and cardioembolism
  3. R heart failure
  4. endocarditis
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7
Q

** what is murmur in mitral stenosis

A

opening snap and decrescendo in diastole

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8
Q

what might be seen on ECG

A
  • afib

- R vent hypertrophy with strain pattern

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9
Q

what is seen in CXR

A
  • splayed carina
  • straight left heart border
  • pulm venous congestion
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10
Q

4 possible medical therapies

A
  1. reduce pulm edema
    - diuetics
  2. prevent/treat afib
    - antiarrhtymics
  3. prevent embolism
    - anticoag
  4. prevent Tx endocard
    - oral hygene
    - early recog and Tx
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11
Q

3 indications for intervention

A

definite
- NYHA 3-4 and valve60-80
prob
- Asx with moderate or worse stenosis

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12
Q

what is intervention

A

valve replacement

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13
Q

3 parts of mitral regurg etiology

A
  1. leaflet chordae
    - endo, rhuem, congenital
  2. annulus
    - LV dilatation
  3. papillary muscles
    - iscehmia or infart
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14
Q

what is pathphys in chronic

A
  • state of LV and LA vol ume overload
  • systolic function may remain normal or years until fibrosis
  • low P LA outlet may mask clinical deterioration
  • may have intact EF - reduced EF is bad sign
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15
Q

what is patho in acute

A
  • sudden vol overload
  • LV and LA not ready
  • high preload, low afterload
  • high contractility
  • poor outflow - pulm edema and shock
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16
Q

Sx of regurg

A
  • fatiuge
  • exertional dyspnea
  • orthopnea
  • edema
  • PND
  • palpitation
17
Q

4 parts of workup

A
  1. ECG
    - LA enlarged, LV hypertrophy
  2. CXR
    - megaly, atrial dilatation
  3. ECHO
  4. cath
18
Q

5 parts of medical ther

A
  1. TX underlying cause if possible
  2. aftreload reduction - ACEi
  3. preload reduction - diuretics
  4. endo recog and Tx
  5. antiarryhtmi and anticoag
19
Q

indications for surge

A

definint
- acute Sx
- NYHA 2-4 and normal vent function
Contraindicated in severe vent dysfunction

20
Q

what is patho of endo

A

exposure (bacteremia) + diathesis (preexisting lesion)

21
Q

2 main types of phenommena seen with it

A
  1. vasc
  2. iummunological
    - glomeruloneph
    - RF +ve
    - osler nodes
22
Q

*** duke criteria for endo

A
2 major, or 1 major 3 minor, or 5 minor
MAJOR
-blood culture with typical organisms
- evidence of endocardial involvment
MINOR
- predisposition
- fever
- vasc. phenom
- immuno phenom
- microbiol evidence
23
Q

3 Tx of endo

A
  1. targeted ABx
  2. elim portal of entry
  3. valve surgery
24
Q

3 groups to prophylax

A
  1. prosthetic heart valves
  2. previous endo
  3. unrepaired complex congential heart disease
25
Q

4 common errors in endo

A
  1. failure to recog. asymptomatic LV dys in mitral regurg
  2. failure to anti-coag in Afib
  3. failure to provide bridging anti-coag for vavlular prostheses
  4. failure to recognize and Tx endo
26
Q

3 groups not ti echo from choosing wisely

A
  1. asymtomatic PT with innocent murmurs
  2. asymptomatic PT with mild stenosis more than 3-5 years
  3. asymptomatic PT with mild mitral regurg and normal LV function