Phosporus Flashcards
Primary anion found in
intracellular fluid
Normal serum levels (floss 1.8 times a day)
1.8-2.6mEq/L (or 3-4.5mg/dL)
85% phosphorus found in
bone & teeth
Combined in 1:2 ratio with
Ca++ 1, phosphate 2
14% found in (floss is soft)
1% found in
14% found in soft tissue
1% found in ECF
Plays a role in
cell membrane activity, muscle and neurological function
phosphate delivers 02 from what to what? (floss dragging blood to tissues)
O2 from RBCs to tissues
Buffers
acids and bases
Promotes energy transfer to
cells through ATP
Regulation - parathyroid?
Dietary intake - meat, fish, milk
Intestinal absorption -via gi tract, promoted by vitamin D and Parathyroid hormone
Calcium Levels
promotes EXCRETION by Kidneys (does opposite with ca)
inverse relationship with Ca
Hypophosphatemia - intra or extracellular shifts? (too much phosphate shifting into cell)
Transient intracellular fluid shifts -
hypophosphatemia clinical manifestations (low flossssss makes tu sssssslur)
Sx may be acute or develop gradually
Majority of sx due to decreased ATP
higher Ca
mental status, slurred, seizures, muscle weakness, anemia, monitor for dysrthmias.
hypophosphatemia -Diagnostic Tests - and what will be high? think inverse…
Serum phosphorus less than 1.7mEq/L (or less than 3mg/dL)
PTH will be elevated with hyperparathyroidism
Serum Ca++ will likely be increased
hypophosphatemia interventions (all the same, apical, I/O, heart, etc)
Assess & document LOC, orientation, neurologic status with each VS
Assess muscle strength and deep tendon reflexes
Administer Phosphate as ordered
as - po or - IV. monitor IV carefully. must have good kidney function. monitor I/O before and after. Arrhytmias are possible so monitor apical HR.
Encourage intake of foods rich in phosphorus
Medicate for pain
Monitor for signs of cardiac or pulmonary complications
hyperphosphatemia - causes- (drowning in floss on the toilet) and what is the MAIN cause
Excessive intake of phosphates (laxative)
Extracellular fluid shifts (lactic acidosis)
Cellular destruction - crush injuries
Decreased urinary losses (renal failure MAIN cause of hyperphosphatemia**)