CAD Flashcards
atherosclerosis starts with (NOT inflammation for this one)
a fatty streak sticks
total cholesterol (cholesterol has your fav numbers)
< 200 low risk
200-239 borderline
>239 high risk
HDL (H High at 35)
> 35 low
< 35 high
LDL (29, 30, and 59 are liddle old)
<129 low
130-159 medium
>159 high
triglycerides (try 201 units to get high)
<200 low
201-399 high
400-1000 very high
>1000
how atherosclerosis forms
inflammation, formation of fibrous cap, if thrombus, it’s just a ruptured plaque.
blood sugar with athleroscoloris should be
right at 100
signs of CAD (Caddy is 4th) - AND BP and heart rhythm? (CAddy can be high or low)
4th heart sound, tachycardia, hypotension, HTN, angina
signs of CV disease (my CV has some pulses and headaches)
Diminished carotid pulses, carotid artery bruits, focal neurological deficits, headaches
signs of PVD
Decreased peripheral pulses, peripheral artery bruits, pallor, peripheral cyanosis, gangrene, ulceration, difficulty ambulating, pain with ambulation
signs of AAA
Pulsatile abnormal mass, peripheral embolism, circulatory collapse, pre/syncope, weakness
Atheroembolism (if Athens is blocked, it will fall off the map)
Gangrene, cyanosis, ulceration
athero risk factors (DH LAGGS in athens)
Risk factor assessment (diabetes, HLPD, gender, LV function, provocation of angina, genetics, stress)
athero - Echocardiography (The echo in athens is my liver EF)
to assess LV function, EF, predicts survival
athero diagonostics (the 2 Es and a C in Athens)
ECG, CXR, Echo, labs
CAD
RCA, L main, LAD (left anterior descending), circumflex
cause of supply demand imbalance
thromus or embolus, spasm, hypovolemia, anemia, HR up or down, BP up or down
prevention of CAD
Control cholesterol, diet, physical activity, medications, quit smoking, manage HTN, control diabetes
angiography risk assessment (Angie is high, med, low - that’s it)
just know that there is risk assessment for high, medium, and low
angina pectoris - cause?
A syndrome characterized by episodes or paroxysmal pain or pressure in the anterior chest caused by insufficient coronary blood flow
angina pectoris - Physical exertion or emotional stress
increases myocardial oxygen demand, and the coronary vessels are unable to supply sufficient blood flow to meet the oxygen demand
Chronic Stable Angina (stable will disappear with rest)
Decreased blood flow to myocardium usually caused by CAD
Temporary pain/pressure
Predictable, long term, familiar pattern
Resolves with nitroglycerin, oxygen or rest. This ALWAYS goes away with rest, etc.
exertion angina (thanksgiving ppl) - how long does it last? (exert for less than 15 min)
Resolves with rest or NTG (nitrogycerin)
Lasts < 15 minutes
May radiate to arm, shoulder, back, jaw, neck, wrists
Variant or atypical or Prinzmetal angina
Not caused by exertion
Often caused by coronary artery spasm
Often there is no coronary artery blockage or atherosclerosis
angina described as
May be described as tightness, choking, or a heavy sensation
symptoms of angina - and what time of day usually?
dyspnea, SOB, dizziness, nausea, vomiting. will often wake up feeling this way.
angina pain is where? (Not chest)
retrosternal and may radiate to neck, jaw, shoulders, back or arms (usually left)
does angina pain subside with rest?
yes, or NTG
anxiety and angina?
usually happen together
unstable angina requires
medical intervention!
if ppl have to take 2 TNG,
call EMS.
assessment of chest pain
Scale of 1-10
Quality
Severity
Frequency
Location and radiation
Duration
Precipitating factors
Relieving factors