calcium Flashcards
calcium - cation or anion?
One of body’s most abundant ions (major ECF cation)
Primarily combined with phosphorus to form mineral salts of bone & teeth
Exerts sedative effect on (milk before bedtime calms my nerves)
(drink milk before bedtime) nerve cells
Functions to develop cardiac action potential and contraction of muscle
what regulates Ca? and what does it activate? (Ca goes from bones to ECF, to kidney for reabsoption)
Parathyroid Hormone
- Released from parathyroid gland
- Promotes transfer of Ca from bones to ECF
- Activates vitamin D
- Promotes Kidney reabsorption
vitamin D is a major source of
calcium through oral intake of foods rich in Ca++
calcitonin - what is it and where is it produced? (mom needs to tone her thyroid)
- Hormone produced by thyroid gland.
hypocalcemia causes - (when the albino is low, the milk is low)
Due to reduction of total body Ca++ or due to % of Ca++ that is ionized (look at albumin levels; low albumin is most common cause)
Increased Ca++ loss
Altered intestinal absorption
Altered regulation
Increased phosphorus or decreased Mg levels
hypocalcemia - Clinical Manifestations (low ca makes me french)
Related to diminished function of
neuromuscular, cardiac and renal systems
Numbness, tingling of hands, muscle spasms,
+ Truosseau (20 mm above systolic and cramping of fingers) , +Chvosteks sign (twitching of cheek - Ch for cheek), increased DTRs (deep tendon reflex)
Muscle cramps, can progress to tetany
Cardiac Arrhythmias
hypocalcemia - what numbers on blood test (Selene without Ca) and what number is serum albumin?
Diagnostic Tests
Serum Ca++ less than 8.5
Look at serum albumin less than 3.5
Xray findings may show osteoporosis, bone cavitation
hypocalcemia signs and monitor what? (everyone else is doing it)
Monitor for signs of hypocalcemia - altered sensorium, assess orientation. call light within reach, etc. deep tendon reflexex, apical heart rate for arrythemias.
hypercalcemia typically develops when there is an…(Ca moves from what to what)
increased movement of calcium from bone to ECF (hyperparathyroid, matestis of bone)
Shift of Ca++ from Bone to ECF
Increased Intake & Absorption
Decreased Ca++ Excretion
hypercalcemia clinical manifestions (too much ca makes me weak)
Many effects related to excess Ca++ in cells, which causes a decrease in cell membrane excitability. Especially in skeletal and heart muscle and nervous system:
Weakness, decreased or absent DTRs
Bone pain
Constipation
Potential for renal calculi (flank pain, abdominal pain)
Dysrythmias
hypercalcemia - labs (not 10 exactly)
Total serum Ca++ > 10.5mg/dL (also look at albumin level)
hypercalcemia - monitor for what? (buffy drinking milk)
Monitor for altered sensorium
Provide safe environment (fall risk)
hypercalcemia from taking what meds?
tums, maalox
Ca is necessary for…
blood clotting process
where is Ca located? and where is most of it?
Less than 1% of Ca++ is located in ECF & ICF compartments (of which approx 40% is bound to albumin)
99% located in bone
what has an inverse relationship with Ca?
Phosphorus & calcium have inverse relationship.