cardiac defects peds Flashcards
Structural and Functional Differences
Infants and children < 7 years of age
lies more horizontally
Between ages 1 and 6 years
4x birth size (size of fist)
Between 6 and 12 years of age,
10x birth size
However, the heart is smaller proportionally at this time than at any other stage in life.
During the school-age years
grows more vertically within the thoracic cavity
During adolescence
continues to grow in relation to the teen’s rapid growth
Cardiac output
Amt of blood ejected by heart in 1 min
Determined by multiplying the HR in 1 min by stroke volume
Stroke volume
The amt of blood ejected by the heart with each heartbeat (or contraction)
Affected by 3 factors
Preload
Stretch of myocardial fibers
Circulating blood volume
Afterload
Resistance against which the ventricles pump during contraction
Affected by BP (increases as BP increases, heart pumps harder)
Contractility
Force of left ventricle ejection
Pediatric Indicators of Cardiac Dysfunction
Poor feeding/fatigue with feeding
Tachypnea/tachycardia
Failure to thrive
poor weight gain
activity intolerance
Frequent respiratory tract infections
Developmental delays
Prenatal history
Family history of cardiac disease
Diagnostic Cardiac Catheterization
(cath is on sat)
Provides important information about:
Oxygen saturation of blood within the chambers and great vessels
Pressure changes
Changes in cardiac output or stroke volume
Anatomic abnormalities
Interventional Cardiac Catheterization Procedures
ASD/VSD
Transposition of great vessels
Some complex single-ventricle defects
Pulmonary artery stenosis
Balloon dilatation
Valvular pulmonic stenosis
Recurrent coarctation of aorta
Congenital mitral stenosis
Nursing Considerations with Cardiac Cath - Pre-procedure - what about extremities?
Pre-procedure
Describe room and equipment
Explain that the contrast might make him feel warm
Assess color, temp, pulses of extremity distal to procedure site
Nursing Considerations with Cardiac Cath - post procedure - how long to keep extremity immobile?
Keep extremity immobilized for 4-6 hrs post
Keep site clean
Monitor for bleeding and hematoma at site
Compare pre and post assessment of extremity
Ensure fluid intake
Medicate as needed
Causes of CHD (congenital heart disease)
Maternal or environmental = 1% to 2%
Maternal drug use
Fetal alcohol syndrome—50% have CHD
Maternal illness
Rubella in first 7 weeks of pregnancy → 50% risk of defects including PDA and pulmonary branch stenosis
CMV, toxoplasmosis, other viral illnesses → cardiac defects
IDMs = 10% risk of CHD (VSD, cardiomyopathy, TGA most common)
CHF in Children - Impaired myocardial function
(my tachycardia)
Tachycardia; fatigue; weakness; restlessness; pale, cool extremities; decreased BP; decreased urine output
CHF in Children - Pulmonary congestion - symptoms - think pulmonary
(pull my cyanosis)
Tachypnea, dyspnea, respiratory distress, exercise intolerance, cyanosis
CHF in Children - Systemic venous congestion
(my veins have edema)
Peripheral and periorbital edema, weight gain, ascites, hepatomegaly, neck vein distention
Chest tube - when to use? - think of your patient
After open heart surgery, pneumothorax
chest tube - problems
If tube becomes dislodged from container, the chest tube must be clamped immediately to avoid further air entry into the chest cavity. Alternatively, the end may be immediately placed into a container of sterile water or saline to create a water seal.
Pacing- when to do?
Bradyarrhythmias, heart block, cardiomyopathy, sinoatrial or atrioventricular node malfunction
pacing - problems - can they play sports?
Provide close observation of the child, pacing unit, and ECG.
Maintain asepsis at pacing lead insertion site.
Explain to child and family that the permanent pacemaker may be felt under the skin.
Advise against participation in contact sports
Digoxin (cardiac glycoside, antiarrhythmic agent) - action - does it speed up or slow down HR?
Slows heart rate
Increases contractility of the heart muscle by decreasing conduction and increasing force
Used for heart failure, atrial fibrillation, atrial flutter, supraventricular tachycardia
digoxin - what apical pulse to hold for infants and adolscent?
Prior to administering each dose, count apical pulse for 1 full minute, noting rate, rhythm, and quality. Withhold if apical pulse is <60 in an adolescent, <90 in an infant.
Avoid giving oral form with meals, as altered absorption may occur.
Monitor serum digoxin levels (therapeutic range: 0.8–2 ng/mL).
Notes signs of toxicity: nausea, vomiting, diarrhea, lethargy, and bradycardia.
Ginseng, hawthorn, and licorice intake increases risk for drug toxicity.
Note contraindications (ventricular fibrillation and hypersensitivity to digitalis).
Avoid rapid IV administration, as this may lead to systemic and coronary artery vasoconstriction.
Digoxin and Heart Rate Parameters - numbers for infants and school age
(dig for 100)
Infant
100 beats/minute
Preschool/School-age
70 beats/minute
Adolescent
60 beats/minute
Angiotensin-converting enzyme (ACE) inhibitors - action
(Ace is after the fact)
Competitive inhibition of ACE for management of hypertension
Heart failure management in conjunction with digitalis and diuretics
Reduces afterload
Angiotensin-converting enzyme (ACE) inhibitors (captopril, enalapril) - - implications
Monitor BP, renal function, WBC count, and serum potassium.
Discontinue if angioedema occurs.
Captopril: administer orally on empty stomach 1 hour before or 2 hours after meals.
Enalapril: may administer orally without regard to food.
Calcium Channel Blockers - actions
Decrease the excitability of heart muscle
treats certain types of abnormally rapid heart rhythms
Reduce pressure in arteries
dilates the arteries
makes it easier for the heart to pump blood
result, the heart needs less oxygen
calcium channel blockers - implications
Short and long acting forms
Can cause swelling in extremities
β-Adrenergic blockers - actions
(just B cool)
Competitively block response to β-adrenergic stimulation, decreasing heart rate and force of contraction
Used for management of hypertension, arrhythmias, and prevention of myocardial infarction
β-Adrenergic blockers (propranolol, atenolol, sotalol) - implications
Monitor ECG and BP.
Propranolol: administer with food.
Atenolol, sotalol: administer without regard to food. Do not stop drug abruptly.
May result in bradycardia, dizziness, nausea and vomiting, dyspnea, and hypoglycemia (propranolol).
Contraindications: heart block, uncompensated heart failure, cardiogenic shock, asthma, or hypersensitivity.
Vasodilator (Hydralazine) - action - just for HTN
(realize it dilates)
Direct vasodilation of arterioles to manage moderate to severe hypertension, heart failure
Vasodilator (Hydralazine) - implications
Monitor heart rate and BP.
Closely monitor BP with IV use.
Administer oral dose with food.
May cause palpitations, flushing, tachycardia, dizziness, nausea, and vomiting.
Notify physician or nurse practitioner if flu-like symptoms occur.
Contraindicated in rheumatic valvular disease.
Nursing : Assess for fluid overload - right and left side
Nursing : Assess for fluid overload
Left sided failure has pulmonary clinical manifestations
Right sided failure has systemic clinical manifestations….central body edema
Abdominal girth measurement
Furosemide (loop diuretic) - action - think 1st semester
Inhibits resorption of sodium and chloride
Used to manage edema associated with heart failure, and hypertension in combination with antihypertensives
Furosemide (loop diuretic)- implications
Administer with food or milk to decrease GI upset.
Monitor BP, renal function, electrolytes (particularly potassium), and hearing.
May cause photosensitivity.
Spironolactone (potassium sparing diuretic)- implications - what about the kidneys?
Administer with food.
Monitor serum potassium, sodium, and renal function.
May cause drowsiness, headache, and arrhythmia.
May cause false elevations in digitalis level.
Teach children to avoid high-potassium diets, salt substitutes, and natural licorice.
Contraindicated in hyperkalemia, renal failure, and anuria.
Alprostadil (prostaglandin) - actions
(prosty dilates)
Direct vasodilation of the ductus arteriosus smooth muscle
Indicated for temporary maintenance of ductus arteriosus patency in infants with ductal-dependent congenital heart defects
Alprostadil (prostaglandin) - implications - contraindicated in who?
Apnea occurs in 10–20% of neonates within first hour of infusion.
Monitor arterial BP, respiratory rate, heart rate, ECG, temperature, and pO2; watch for abdominal distention.
Fresh IV solution required every 24 hours.
Reposition catheter if facial or arm flushing occurs.
Use with caution in neonate with bleeding tendency.
Contraindicated in respiratory distress syndrome or persistent fetal circulation.
Indomethacin (nonsteroidal anti-inflammatory agent) - actions
(indo my ducto)
Inhibits prostaglandin synthesis in order to close patent ductus arteriosus
Indomethacin (nonsteroidal anti-inflammatory agent) - implications -
(endo my edema)
Monitor heart rate, BP, ECG, and urine output; monitor for murmur.
Monitor serum sodium, glucose, platelet count, BUN, creatinine, potassium, and liver enzymes.
May mask signs of infection.
Note development of edema.
Heparin (anticoagulant) - action
Interferes with conversion of prothrombin to thrombin, preventing clot formation
Indicated for the prophylaxis and treatment of thromboembolic disorders, especially after cardiac surgery
Heparin (anticoagulant) - implications - where to administer? and what is the antidote?
Administer SQ, not IM.
Dose is adjusted according to coagulation test results.
Monitor for signs of bleeding, platelet counts.
Ensure that the antidote, protamine sulfate, is available.
Do not administer with uncontrolled bleeding or if subacute bacterial endocarditis is suspected.
Hypoxemia - what causes it?
(hypo mixing)
Low oxygen saturation of Hemoglobin
May be due to
“mixing of blood” in the cardiac chambers
OR
Desaturated blood returning to systemic circulation (Pulmonary edema)
Congenital Heart Disease (CHD) - Hemodynamic characteristics
Hemodynamic characteristics
Increased pulmonary blood flow
Decreased pulmonary blood flow
Obstruction of blood flow from the heart
Mixed blood flow
INCREASED PULMONARY BLOOD FLOW DEFECTS
Atrial septal defect
Ventricular septal defect
Patent ductus arteriosus
Atrioventricular Canal
Increased Pulmonary Blood Flow Defects
Abnormal connection between two sides of heart
Either the septum or the great vessels
Increased blood volume on right side of heart
Increased pulmonary blood flow
Decreased systemic blood flow
thrills
The sound of a thrill is a soft vibration over the heart that reflects the transmitted sound of a heart murmur