Medsurg final 5 Flashcards
DKA or HHS: Polyuria
both
DKA or HHS: Polydipsia
both
DKA or HHS: Polyphagia
DKA
DKA or HHS: Weight loss
DKA
DKA or HHS: GI effects (nausea, vomiting, abdominal pain)
DKA
DKA or HHS: Blurred vision, headache, weakness
both
DKA or HHS: Orthostatic hypotension
both
DKA or HHS: Fruity odor of breath
DKA
DKA or HHS: Kussmaul respirations
DKA
DKA or HHS: Metabolic acidosis
DKA
DKA or HHS: Mental status changes
Both
DKA or HHS: Seizures, myoclonic jerking
HHS
DKA or HHS: Reversible paralysis
HHS
Cause of DKA manifestation: Cells are unable to use glucose because of insulin deficiency. The body is placed in a catabolic state.
Weight loss
Cause of DKA manifestation: the breakdown of stored glucose, protein, and fat to produce ketone bodies
Metabolic acidosis
Cause of HHS manifestation: Related to serum osmolarity greater than 350 mOsm/L
Seizures and Paralysis
tx of DM complications: maintain perfusion to vital organs by
giving rapid isotonic fluid (NS) replacement
tx of DM complications: When serum glucose levels approach 250 mg/dL, add ____ to IV fluids to minimize the risk of cerebral edema associated with drastic changes in serum osmolarity and prevent hypoglycemia
glucose
tx of DM complications: When serum glucose levels approach 250 mg/dL, add glucose to IV fluids to minimize the risk of cerebral edema associated with
drastic changes in serum osmolarity and prevent hypoglycemia
tx of DM complications: Administer regular insulin (Humulin R) 0.1 unit/kg as an IV bolus dose and then follow with a
continuous IV infusion of regular insulin at 0.1 unit/kg/hr.
tx of DM complications: Blood glucose of less than ___ mg/dL is the goal for resolution
200
tx of DM complications: monitor for what electrolyte
K
What electrolyte levels will initially be increased with insulin therapy
K
Potassium levels will initially be increased with insulin therapy, but
potassium will shift into cells and the client will need to be monitored for hypokalemia.