Top Feline Shelter Medicine Diseases Flashcards

1
Q

what is the classic case presentation of a cat with otodectic mange?

A

mostly seen in kittens - scratching at the ears, shaking head, inflammation inside the pinna, dark brown waxy debris in the ears, & barely visible white specs moving in debris (may see mild alopecia, redness, or pruritus on head/neck)

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2
Q

what is the etiology of otodectic mange?

A

otodectes cynotis

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3
Q

how are ear mites diagnosed?

A

otoscopic exam seeing white moving specs & microscopic exam of mites in debris in mineral oil

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4
Q

how are ear mites treated in the scope of shelter medicine?

A

preferably, all shleter animals should be treated, clean the external ear canal with ceruminolytic cleanser, & systemic treatment with topical selamectin is cost effective for kittens if prepackaged dosages for large dogs are divided among kittens by weight

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5
Q

T/F: ear mites are very contagious between puppies & kittens

A

TRUE

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6
Q

what is the zoonotic potential of ear mites?

A

very low but they can bite humans

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7
Q

T/F: approximately 1/3 of all shelter cats are symptomatic carriers for feline trichomoniasis

A

TRUE

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8
Q

what clinical signs are seen in shelter cats with trichomoniasis?

A

chronic diarrhea that may progress from large to mized bowel diarrhea that is yellow green, malodorous, hematochezia, fecal incontence, tenesmus, & flatuelence

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9
Q

what cats are commonly affected by trichomoniasis?

A

usually densely housed kittens/cats under 2 years old

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10
Q

what is the etiology of trichomoniasis?

A

tritrichomonas foetus - protozoa

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11
Q

how is trichomoniasis diagnosed?

A

PCR is the gold standard but can also do a protozoal culture in house with the InPouch feline test which is less expensive than PCR

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12
Q

what is remission like for cats with trichomoniasis?

A

remission may occur in 5-24 months but relapses are common - infection may persist & be lifelong

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13
Q

how is trichomoniasis prevented?

A

decrease housing density by increasing the amount of space per cat

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14
Q

is metronidazole effective againts trichomoniasis?

A

nope

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15
Q

what is used to treat trichomoniasis?

A

ronidazole

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16
Q

what are the disadvantages of using ronidazole for trichomoniasis?

A

narrow margin of safety, drug-resistance is becoming more common, don’t use in pregnant queens, & it is expensive

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17
Q

T/F: hygiene & disinfection of facilities is not nearly as effective as decreasing cat housing density when treating trichomoniasis

A

TRUE

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18
Q

what is fading kitten syndrome?

A

occurs secondary to vertical transmission of FeLV with hypothermia, dehydration, inability to nurse, & early mortality

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19
Q

what are the clinical signs seen with FeLV?

A

pale/icteric mucus membranes, lymphadenopathy, fever, neuro signs depending on location of lymphoma, chronic ulcerative proliferative gingivostomatitis

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20
Q

what cats are commonly infected with FIV?

A

outdoor male cats

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21
Q

what are the early signs of FIV? what are the chronic signs?

A

early - fever & lymphadenopathy, chronic -

22
Q

what are the 4 subtypes of FeLV?

A

FeLV-A: original virus, source of natural infection, other types mutated from this virus, FeLV-B: high incidence of neoplasia, FeLV-C:increases likelihood of erythroid hypoplasia & severe anemia, & FeLV-T: causes destruction of t lymphocytes & immunodeficiency

23
Q

how is FeLV diagnosed?

A

snap test antigen test

24
Q

what is seen on a CBC of a cat with FIV?

A

non-regenerative normochromic anemia due to bone marrow suppression

25
Q

how is FIV/FeLV prevented?

A

FeLV vaccination, spay/neuter pets, keep infected cats separated, keep surfaces clean

26
Q

how is FeLV related anemia treated?

A

erythropoietin or darbopoietin +/- steroids if the others aren’t effective & if severe, blood transfusion

27
Q

how is FIV stomatitis treated?

A

AZT, antibiotics, & if persistent, removal of all teeth

28
Q

how is FeLV transmitted?

A

horizontal through social contact or biting or vertical transmission through utero

29
Q

how is FIV transmitted?

A

transmitted through biting - high incidence of FeLV negative b-cell lymphoma

30
Q

what is the classic case presentation of a cat with an upper respiratory tract infection from calicivirus or herpes virus?

A

usually seen in kittens, stomatitis/gingivitis/oronasal ulceration in stomatitis, anorexia, lethargy, ocular discharge, nasal discharge, sneezing, & in herpes virus, dendritic corneal ulcers

31
Q

how are feline upper respiratory tract infections prevented?

A

vaccination with modified-live or killed FCV/FHV starting at 6-9 weeks of age & then every 3-4 weeks until kitten is atleast 16 weeks old, then annually

32
Q

how are feline upper respiratory tract infections treated?

A

mainly supportive care, oxygen/fluids if needed, broad spectrum antibiotics to prevent secondary bacterial infections, nebulization, & keeping eyes/nose clean

33
Q

what bacteria can cause feline upper respiratory infections?

A

bordetella bronchiseptica & chlamydophila felis

34
Q

what is pseudomycetomas?

A

pyogranulomatous draining nodules in generalized dermatophytosis

35
Q

what is the most common etiology of dermatophytosis in cats?

A

microsporum canis

36
Q

what is the classic case presentation of a shelter cat with ringworm?

A

kittens most commonly affected on head/extremities, patchy alopecia, often circular or ring like, scaling, crusting, & sometimes pruritus

37
Q

how is ringworm treated in shelter cats?

A

clip long haired cats, combo miconazole/chlorhexidine rinses, lime sulfur dips, itraconazole

38
Q

what is the potential complication of ringworm in devon rex cats?

A

eosinophilic/mastocytic dermatitis

39
Q

T/F: ringworm is difficult to eradicate from shelters & expensive to treat

A

TRUE

40
Q

if a cat is put on griseofulvin for ringworm, what must be monitored?

A

CBC every 2 weeks, then monthly - don’t use in pregnant or FIV positive cats!!!

41
Q

what is the classic case presentation of a cat with panleuk?

A

fever or hypothermia, depression, anorexia, vomiting, rapid dehydration, thick bowel loops, diarrhea, +/- sudden death (fading kitten syndrome)

42
Q

why will cats get cerebellar hypoplasia from panleuk?

A

in utero or perinatal infection

43
Q

how is panleuk diagnosed?

A

leukopenia/neutropenia (50-3,000 WBC), can do a snap parvo test

44
Q

what treatment is used for panleuk?

A

supportive care only - isolation, aggressive iv fluid, broad spectrum antibiotics

45
Q

what cats have the highest mortality from panleuk?

A

kittens under 5 months

46
Q

T/F: a WBC count under 2,000 from panleuk Is associated with a worse prognosis

A

TRUE

47
Q

T/F: long haired cats with ringworm tend to have persistent infections (years) where infections in in short haired cats may be self-limiting

A

TRUE

48
Q

what signs are seen with cerecellar hypoplasia cats infected by panleuk in utero?

A

hypermetria, ataxia with no weakness, intention tremors, & head bobbing

49
Q

how is panleuk prevented?

A

vaccination with the inactivated or modified live starting at 6-9 weeks, booster every 3-4 weeks until 16 weeks of age, & then annually

50
Q

T/F: a pregnant queen cannot be vaccinated with the modified live vaccine for panleukopenia

A

TRUE