Exotics Top Topics - Camelids Top 6 Diseases Flashcards

1
Q

what are the most common congenital/inherited diseases of camelids?

A

atresia ani, choanal atresia, & wry face

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2
Q

what is the classic case presentation of atresia ani?

A

tenesmus in a newborn, abdominal pain, & abdominal distension

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3
Q

what is the classic case presentation of choanal atresia?

A

very common - unilateral or bilateral, respiratory distress in a neonate especially when nursing, & aspiration pneumonia

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4
Q

what is the classic case presentation of wry face?

A

laterally deviated maxilla, dental malocclusion, tear staining due to obstruction of nasolacrimal duct, and potentially nasal occlusion

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5
Q

how is atresia ani diagnosed?

A

failure of dorsal membrane between the rectum & anus to rupture during development

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6
Q

how is choanal atresia diagnosed?

A

choane fail to open during embryonic development

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7
Q

how are the most common inherited/congenital disorders of camelids prevented?

A

do not breed affected animals or parents of affected animals

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8
Q

how are the most common inherited/congenital disorders of camelids treated?

A

not treated because rarely successful - euthanasia

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9
Q

what two inherited/congenital disorders can occur together in camelids?

A

choanal atresia & wry face

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10
Q

what defects are fairly common in south american camelids?

A

urogenital, ocular, & cardiac

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11
Q

what is the purpose of C1?

A

fermentation compartment, esophageal groove present, together with C2 similar to rumenoreticulum in ruminants

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12
Q

what is the purpose of C2?

A

smallest compartment, mostly glandular epithelium, & divisions that form a reticular pattern

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13
Q

what is the purpose of C3?

A

mostly glandular, distal 5th secretes acid similar to a monogastric stomach/abomasum, & fluid reabsorption

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14
Q

what is the classic case presentation of gastric ulcers in camelids?

A

decreased appetite, colic, frequent kushing, depression, melena, hypersalivation, & bruxism

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15
Q

how are gastric ulcers diagnosed in camelids?

A

usually occur in distal C3 or proximal duodenum - presumptive diagnosis based on history & clinical signs

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16
Q

how are gastric ulcers in camelids treated?

A

oral meds are generally unsuccessful, parenteral omeprazole, parenteral abx, & stress reduction

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17
Q

when do clinical signs related to gastric ulcers in camelids usually occur?

A

3-5 days after stressful event

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18
Q

T/F: gastric ulcers of camelids are likely stress-related to things such as environmental changes, isolation, injury, or illness

A

TRUE

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19
Q

what is the classic case presentation of mites in camelids?

A

alopeica (especially between the toes with chorioptic mange), pruritus, hyperkeratosis, & scaling

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20
Q

what is the classic case presentation lice in camelids?

A

biting lice - ragged fiber coat with matting/fiber loss, rubbing against fences/trees due to pruritus, & scaling around base of tail/sides of neck/sides of body

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21
Q

what is the classic case presentation of sucking lice in camelids?

A

anemia, pruritus, & scaling around the flanks, head, neck, & withers

22
Q

what is the classic case presentation of ticks in camelids?

A

lower motor neuron paralysis (flaccid), horner syndrome, & head tilt/nystagmus

23
Q

what mites often affect camelids? how are they diagnosed?

A

sarcoptes, psoroptes, & chorioptes - deep skin scrapes or biopsy

24
Q

what lice affect camelids? how are they diagnosed?

A

damalinia breviceps (biting) & microthoracius cameli (sucking) - remove with transparent tape & observe with magnifying glass or microscope

25
Q

how are mites on camelids treated?

A

parenteral ivermectin every 10-14 days, chorioptes/sarcoptes may require additional topical therapy, prevention includes treating new herd additions & animals returning from travel

26
Q

how are sucking lice treated in camelids? how are biting lice treated in camelids? how are they prevented?

A

sucking: parenteral ivermectin, biting: topical synthetic pyrethrins, & prevention is the same used for mites

27
Q

how are ticks on camelids treated?

A

manual removal, pyrethrin spray, & ivermectin

28
Q

why are topical products not as helpful for ectoparasites on llamas/alpacas?

A

they don’t contain lanolin which hinders the spread of topical products

29
Q

what is the classic case of mycoplasmosis in camelids?

A

pale mucus membranes, chronic weight loss, lethargy, & depression

30
Q

what is the etiology of mycoplasmosis in camelids?

A

mycoplasma haemolamae

31
Q

how is mycoplasmosis in camelids diagnosed?

A

anemia, organisms visualized on blood smear, & PCR

32
Q

how is mycoplasmosis in camelids treated?

A

long acting tetracyclines

33
Q

how is mycoplasmosis transmitted in camelids?

A

insects, contaminated needles, & transplacental

34
Q

T/F: a chronic carrier state of mycoplasmosis is common in camelids once they are infected

A

TRUE

35
Q

T/F: many camelids are infected with mycoplasmosis but they only become clinical after stress or immunosuppression

A

TRUE

36
Q

what is the classic case presentation of coccidiosis in camelids?

A

diarrhea, weight loss, lethargy/weakness, depression, colic, tachycardia, ascites, neuro signs secondary to cerebral edema, & sudden death

37
Q

what is the etiology of coccidiosis in camelids? how is it diagnosed?

A

eimeria macusaniensis (large coccidiosis) - multiple sugar fecal floats, hypoproteinemia, & hypoalbuminemia

38
Q

how is coccidiosis in camelids treated?

A

oral ponazuril & parenteral sulfadimethoxine

39
Q

what is the most pathogenic coccidia in camelids?

A

e. macusaniensis

40
Q

what is the classic case presentation of hepatic lipidosis in camelids?

A

anorexia/weight loss, icterus, depression/lethargy, ascites, diarrhea, hepatic encephalopathy, recumbency, & sudden death

41
Q

what lab values are elevated in camelids with hepatic lipidosis?

A

non-esterified fatty acids, beta-hydroxybutyrate, bile acids, GGT, SDH, AST, bilirubin, ammonia, & sometimes BUN/creatinine

42
Q

what lab values are decreased in camelids with hepatic lipidosis?

A

albumin, total protein, glucose

43
Q

how is hepatic lipidosis in camelids diagnosed?

A

liver biopsy is definitive

44
Q

what supportive care is used for camelids with hepatic lipidosis?

A

iv fluids, glucose, analgesia, parenteral omeprazole, provide a companion animal, & frequent walks

45
Q

what nutritional support is used for camelids with hepatic lipidosis?

A

increase oral food/fluid intake (high carb low fat), for severe cases, tube feeding, parenteral (must have higher concentrations of amino acids than other species), & yogurt

46
Q

how is hepatic lipidosis prevented in camelids?

A

check BCS 4-6 times a year, check pregnant/lactating females monthly, regular forage testing, clostridial vaccination, & make sure food intake is adequate in sick animals

47
Q

hepatic lipidosis in camelids is associated with what type of diet?

A

diet high in mature grass forage

48
Q

T/F: hepatic lipidosis in camelids may be stress related from things such as illness, isolation, & pregnancy

A

TRUE

49
Q

T/F: in a camelid with hepatic lipidosis, continuous rise of BUN & creatinine in spite of supportive care are poor prognostic indicators

A

TRUE

50
Q

what is the prognosis of hepatic lipidosis in camelids?

A

guarded - recovery may take weeks to months