Tintinellis EMED Ph 2 Flashcards
Cardiogenic Shock MC results from ?
These are more likely to occur from ? type of injury
What causes coronary artery hypoperfusion to occur
LV infarct induced pump failure leading to decreased tissue perfusion despite adequate volume avail in circulation
STEMI- leading cause of death in PTs w/ AMI
Lack of systemic resistance= dec DBP
Why does pulmonary edema occur during cardiogenic shock
Augmenting ? can improve coronary artery perfusion
What is the risk of this augmentation
Inc after load/dysfunction increases LV end diastolic pressure
Vasoconstriction
Dec CO
Why does cardiogenic shock induce systemic vasodilation
Euvolemic cardiogenic shock occurs subacutely in ? Pts
Finding of diaphoresis on these Pts indicates ?
Triggers systemic inflammatory response= inc NO w/ negative inotropic effects
Heart failure
Activation of sympathetic nervous system
Shock is indicated by SBP <90 or a MAP below ?
? type of view on E-FAST is used for suspected tamponades/effusions
Pericardial effusion and aortic root bigger than ? suggest ascending aortic dissection
<65mmHg
Sub-costal 4 chamber view
> 3cm
? is the most important definitive intervention for acute ischemia related cardiogenic shock
Add O2 to keep sats above ?
What issue happens after intubation w/ pos-pressure ventilation and how is this countered
Emergent revascularization via PCI/CABG
> 91%
Dec preload and CO= worsened HOTN;
Fluid bolus
How is HOTN during cardiogenic shock Tx
How is congestion w/out HOTN Tx
What is the nest step if these meds fail to correct the shock
Vasopressor- Dopamine, NorEpi
Inotrope- Dobutamine: inc contractility; if SBP <70: NorEpi
Intra-aortic balloon pump/assist device
Pts taking BBs and suffering from cardiogenic shock may not have appropriate response to dobuatmine, in which case ? med is used
Acute HF management is improved by ? therapies
? new medication is FDA approved for use but does not improve outcomes
Milrinone- selective phosphodiesterase 3 inhibitor
BB ACEI Resynch therapy Spironolactone
Nesiritide
What are the 3 cardinal Sxs of HF
MI induced reduction of CO causes the neurohormonal release of ?
What are the counter-regulatory responses to this activation
SOB Edema Fatigue
NorEpi Endothlin ADH TNF-a: activate RAAS/Symp-NS
Natriuretic peptide- inhibit RAS/sympathetic NS
A: atrial B: ventricle C: endothelium
EF normally is ?
? dysfunction is seen early on in ischemic induced HF cascade
What Sxs have the highest sens/spec for HF
60%; Systolic failure <50%
Diastolic dysfunction leading to CADz
Sensitivity: dyspnea w/ exertion
Specificity: PNDyspnea, Orthopnea, Edema
What PE finding has the highest positive likelihood for acute HF
What CXR findings are the most specific for Dx of Acut HF
? E-FAST finding suggests elevated central venous pressure
S3 sound
Pulm congestion, Megaly, Interstitial edema
IVC >2cm, Collapse <50%
Acute HF Pts need to keep O2 levels above ?
? intervention can help avoid intubation for these Pts
What is the next step for persistent HOTN after Nitro use
95%
Dec after load w/ vasodilators- Nitro then Nitroprusside
Dec volume or RV infarct- give NS bolus
? is the best predictor for recurrence of syncope
Prolonged QTc is defined as ?
Myocarditis can lead to ? and is definitively Dx by ?
Syncope w/in preceding year
> 470msec
Dilated myopathy, Biopsy
? is the MC Sx of Pericarditis
? is a distinguishing feature
? is the MC and most important PE finding
Chest pain worse w/ supine, relieved w/ sitting
Pain referred to L trap
Friction rub w/ diaphragm at LLSB
What rare CXR finding may aid w/ Dx pericarditis
? is the image modality of choice for detection and confirmation
How do Pts die from PE
Epicardial fat pad sign
Echo
PEA/Asystole or RV failure and circulatory shock
? joint are most and least likely to be the origin of PEs
? combo causes the greatest risk for VTEs
? surgical procedures have the highest rates of VTE formation
Least: Elbow Shoulder Ankle Knee Hip- most
Same sided hip-knee immobile w/ non-weight bearing
Abdominal to remove cancer
Joint replacement
Brain/spine w/ neuro deficits
When are cancer Pts at highest risk for developing VTEs
? are the two MC Sxs of PE
What are the two rare CXR findings of PE
During chemo Tx
Dyspnea, Chest pain
Westermark- lobar artery obstruction
Hampton- pulmonary infarction
Leg size difference of ? suggests PE
Where are these measurements taken
PE R/o Criteria
≥2cm
10cm below tibial tubercle
HAD CLOTS:
Hormones Age >50 +DVT/PE Hx
Coughing blood Leg swollen O2 <95 Tachy >100 Surgery/Trauma <4wks
Well’s Score for DVT
EAT CHIPS: Edema/Pain/DVT Sx- 3 Alternative Dx unlikely- 3 Tachy- 1.5 Ca- 1 Hemoptysis- 1 Immobile >3 days: 1.5 Previous DVT/PE: 1.5 Surgery <1mon: 1.5
MC imaging modality for PE
Criteria for Pre-E
What are the 4 etiologies of catecholamine induced HTN emergencies
CT angiography w/ contrast
≥140/≥90 after 20wks
Clonidine d/c or withdrawl
Autonomic dysfunction
Pheo
Sympathetic drug use- cocaine, meth, PCP
How quickly is BP lowered during HTN Emergency
What is the therapeutic goal for aortic dissections
? is the mainstay of Tx
Dec SBP x 25% in first hour
<160/100 over 2-6hrs
To normal <48hrs
SBP 100-20 w/ HR ≤60bpm w/in first hour
Nitrates
How are HTN Emergencies induced by cocaine/meth Tx
What is used if Pts don’t respond to this Tx
What is used as 3rd line agents
IV Benzos, avoid BBs (except labetalol- +A-blocker effect)
Nitro or Phentolamine
CCBs
First line Tx for Pheo induced HTN Emergency
? medication is used pre-op
Aortic dissections occur d/t damage to ?
Phentolamine
Phenoxybenzamine
Intima- blood enters between intima and adventitia
How are aortic dissections Dx
If properly trained, ? is equally sensitive and specific
Criteria for P-HTN
CT w/ and w/out contrast
TEE
Aterial pressure >25mm at rest, >30 w/ exertion
Definitive method to Dx P-HTN
Why does P-THN lead to RV failure
? is the MC EKG finding
R-sided catheter
RCA perfusion depends on diastolic, RA pressure during systole and diastole. Dz dec systolic perfusion, CO
RAD