Phase 2 EMED w/ Book Flashcards

1
Q

Define Acute Chest Pain

This term includes ? two Dxs that are defined as ?

A

Recent onset of pain/pressure/tightness in anterior thorax between xyphoid, suprasternal notch and both mid-axillary lines

AMI: necrosis proven w/ elevated markers (classified into N/STEMI)
UA: chest pain/equivalent (neck/UE pain) from de perfusion w/ inc freq/less activity/at rest

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2
Q

What type of nerve fibers innervate the chest

What type of pain/description does these fibers offer

A

Somatic- chest wall from dermis to pleura

Enter spine at corresponding dermatome level= precise, easily described

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3
Q

Why/how does ACS present w/ referred pain

What words may be used to describe this type of pain stimulation

A

Visceral fibers map from spine to parietal cortex w/ corresponding somatic fiber

Vague, difficult
Discomfort Pressure Ache Tight Heavy

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4
Q

Pts presents w/ chest pain need to be in bed/on monitor/w/ EKG in ? min and add O2 if ?

What are the associated Sxs that can be present w/ these Pts

A

<10min
SPO2 <94%

Diaphoresis
Dyspnea
Dizzy
N/V
Palpitations
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5
Q

AMI Pts w/ pain located ? have 2x M/M risk increase

Define Classic Chest Pain

A

Pain out of chest cavity

Retrosternal, left anterior chest w/ Crushing Squeeze Tight Pressure that inc w/ exertion/improves w/ rest

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6
Q

Standard time limits for Anginal, UA and AMI pain

What are the “classic” ACS features?

A

A: 2-10min
U: 10-30min
AMI: >30min

Radiating Jaw/Arm/Neck pain
Diaphoresis
Dyspnea
N/V

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7
Q

Define “Non-Classic Chest Pain”

What populations are more likely to present with this

A

Pain lasting sec-hrs w/out wax/wane or worsened by movement

DM
Psych
Female
AMS
Minority
Elderly
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8
Q

Pre/Early menopause females w/ non-classic chest pain are more likely to present w/ ? pain features

Female ACS associated Sxs

Male ACS associated Sx

A

Palpitations w/out pain
Unrelieved w/ Nitro/AAcids
Non-exercise related
C/C fatigue

N/V
Jaw/Neck/Back pain

Diaphoresis

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9
Q

MI + Diaphoresis= ? inc sudden death risk

Epigastric pain, even if relieved w/ AAcids, is suspicious for ACS especially in ? populations

How does this presentation change the Pts work up

A

4x than ASx
2x than classic angina

> 50y/o
Hx CADz

EKG for abdominal pain

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10
Q

Why is ACS considered for new palpitations

What are 3 RFs that can increase the rate of atherosclerosis leading to MI

A

Ischemia= inc automaticity, irritability leading to dysarrhythmia

Chronic cocaine
HIV/retroviral use

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11
Q

What Sxs are most strongly associated w/ ACS

How do MIs cause Tachy

How do MIs cause Brady

A

Pain radiating to arm/shoulder, worsened w/ exercise

Inc sympathetic tone, dec LVSV

Ischemia/infarct in conduction system altering activation to SA/AV node

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12
Q

AMI can present w/ ? murmurs

A new murmur can indicated ? two issues occurring

Ischemia induced heart failure will have ? PE finding

A

S3/S4
Dec S1
Paradoxical split S2

Aortic dissection
Ruptured tendonae

Crackles in lungs

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13
Q

What Sxs are most associated w/ AMIs?

? type of MI will benefit from rapid reperfusion efforts

A

HOTN S3 Dizzy

New ST elevation <1mm in two contiguous leads

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14
Q

What EKG finding indicates PTs are at risk for MI

What other conditions can present w/ ST elevations

A

ST depression w/ t-wave inversion

Peri/Myocarditis
LVH
Early Repol
Ventrical aneurysm

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15
Q

Define cardiac troponin marker

What marker is assessed for MIs in Pts w/ RF

When/why is BNP elevated

A

cTn: protein needed for contraction held by actin/myosin filaments in myocyte cytoplasm

cTnT, also inc after dialysis

Any ventricular dysfunction

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