Ortho 2.2 Flashcards
OA Pts are more likely to report ? issue during a flare up
What would an OA joint effusion result look like?
What are the MC OA findings in the hand
Stiffness > Pain
Mild pleocytosis
Elevated protein
Normal viscosity
PIP- Bouchard
DIP- Heberden
First CMC
What are three common locations for OA to develop, especially in the foot?
What is the MC form of OA in the knee
If these Pts develop a Baker’s Cyst, it is due to the joint cavity communicating between ? two structures
First MTP joint
Calc/Talus/Navi articulation
Valgus/rigidus
Subtalar joint
Varus- bow legged
Gastroc/Semi-membrane
What will be seen on x-rays of OA
The severity of these findings are based on ? scale
What are the 4 grades
Lost joint space
Osterophytes
Sclerosis
Subchondral cysts
Kellgren Lawrence
0: none
1: doubt
2: minimal
3: moderate
4: severe
Non-pharm Tx of OA
Pharm Tx of OA
What opioids can be used for short-term relief w/ NSAIDs
Avoidance Reassure Education Weight-loss
NSAIDs, then Acetaminophen
Codeine Hydrocodone Oxy Tramadol
? COX-2 selective NSAID has s/e similar to Tylenol and used in Pts w/ no cardiac risk but are not achieving pain relief w/ Acetaminophen
What therapy can PTs utilize who are unable to tolerate weight bearing exercises
What are the indications surgical repair is needed for joints w/ OA
Celecoxib
Isometric exercises
Lost function
Pain at night
Non-surg failure
Define RA
What are common Sxs of RA
What joints are more commonly involved symmetrically
Chronic synovium inflammation causing erosion
2+ swollen joints in AM >1hr x 6wks or,
+RF/anti-CCPs
Feet Hands Ankle Wrist Knee
To receive an official Dx of RA, Pts need 6 out of 10 points based on ?
If they present w/ ? finding, they meet the definition
What joints are affected first and which ones are spared
Joint involvement
Acute phase reactants
Pt self report
Serology results
Characteristic erosive changes
First: hand/feet
Spared: DIP
Extra-articular Sxs of RA are more common in Pts w/ ?
These rarely occur in the absence of ?
? tendons can be ruptured by the Dz process
+RF
Clinical arthritis
EPL= no thumb extension
What are the predominant early PE findings of RA
What two findings are not predominant findings
? is an early result and what is a late result of the Dz process
Pain w/ pressure
Swelling
Dec ROM
Warmth, Erythema
Early: PIP
Late: joint deformity
? is the MC site for subcutaneous RA nodule
What is Rheumatoid Factor
What lab result is as sensitive and more specific
Elbow
IgM against Fc of IgG
Anti-CCP Abs
What lab result correlates to the degree of RA joint inflammation
? will CBC results look like
Which one correlates to Dz activity
ESR/CRP
Dec serum albumin
Inc ESR/CRP
Platelets
Albumin
What part of the body does Ankylosing Spondylitis affect?
What other conditions are associated with this Dx?
What is the Tx plan?
SI joint
Iritis Aoritis Carditis Enthesitis Uveitis
NSAIDs, Exercise
What parts of the body are affected by arthritis associated w/ IBS
What other conditions can be present w/ this Dx
What is the Tx
Asymmetric/oligoarticular
SI Ankle Knee
Crohns Enthesitis UColitis
NSAIDs
What parts of the body are involved w/ Psoriatic arthritis
What other conditions can also exist
What is the Tx
Wrist Ankle SI Hands
Dactylitis Iritis Nails Enthesitis Skin lesions
NSAIDs Biologics Methotrexate
What imaging results are seen in PTs w/ Ankylosing Spondylitis
What finding correlates to severity of Dz
What is different about this type of arthritis compared to other seronegative arthritis’?
Sacroiliitis, Kyphosis
Hip Ankle Shoulder
Less severe
What microbe pathogens can cause Retiers?
What are the 5 patterns of psoriatic arthritis
What differentiates one of these manifestations from RA
Clostridium Campylobacter Chlamydia Shigella Salmonella Yersinia
DIP Arthritic mutilans Asymmetric oligo Symmetric poly Sacroilitis
Symm Poly: DIP involvement w/ absent RA nodules
? form of IBDz is more likely to develop arthritis
? is the presenting Sx in all seronegative arthropathies
What do Pts w/ Reiters present w/
Crohns
Back pain
Conjunctivitis Asymmetric oligoarthritis of LE large joints Dactylitis Urethritis Enthesitis: Achilles, Plantar Sacroilitis
What is the ‘usual’ clinical presentation of Psoriatic Arthritis
Pts w/ joint problems commonly have ? d/o
Pts w/ IBDz arthritis commonly have ? -itis’?
DIP pain
Scaly cutaneous lesions
Nail- Pits Oncolysis Ridging
Sacroillitis
Spondylitis
Knee/Ankle arthritis
IBDz associated arthritis has ? worsen during flare ups
What type of back measurement needs to be done for Pts w/ Ankylosing Sine?
What is the AKA name for this measurement’s starting point
Peripheral Sxs
Spondylitis Sxs remain same
Post Iliac Spine midline to upper lumbar
Dimples of Venus
? is a common x-ray finding of Psoriatic Arthritis in the hands
What causes the ‘bamboo/poker’ appearance on x-ray of ankylosing spondylitis
Which NSAID is particularly successful at controlling the Sxs from seronegative spondyloarthropathies
Proliferative bone reaction
Terminal phalanges resorption
Bamboo: anulus fibrosus enthesitis
Poker: ALL ossification, Facet autofusion
Indomethacin
What drugs are used for Sxs of AnkSpond not controlled by NSAIDs
What drug may be used for chronic reactive arthritis?
What is best for the Tx of Psoriatic Arthritis
TNF-a: Etanercept, Infliximab, Adalimumab
Sulfasalazine
DMARDs
Skin lesions: photo therapy
? type of gunshot wounds are particularly susceptible for compartment syndrome?
? are the MC compartments to be affected by
What are the 4 compartments of the leg
Prox tibia
Leg/Forearm
Ant/Lat/Sup-Deep posterior
What are the 3 compartments of the forearm
What are the 3 compartments of the thigh?
How long can muscles withstand compartmental pressure before beginning necrotic break down and what happens if relief is not achieved in that time?
Volar: flexor, pronator, supinator
Dorsal: extensors
Wad: radialis, extensors
Medial Ant Post
4hrs
6hrs- possible reversal
8hrs: irreversible necrosis
What are the seven Ps of Compartment Syndrome
? Sx is present at the onset of this condition
What is the most specific test to rule in Compartment syndrome and what are two are extremely late findings
Pain Pallor Paresthesia Paresis Poikilothermia Pulselessness #7: pressure
Altered sensation in effected compartments
PooP w/ passive stretch-
Pulseless
Paresis
CRPS is a clinical Dx composed of ?
What are the two types
? types of injuries can precipitate this Dx
Functional impairement
Autonomic dysfunction
Trophic changes
Pain
1: RSD/Algo- no lesion
2: causalgia- + nerve lesion
Distal radius Fx
Injury to infrapatellar branch
? is the most important step for CRPS prognosis outcomes
What is the exception to this rule
What happens after this exception
Early, appropriate Tx
Distal radius Fx w/ poor finger function 3mon after Fx
Algodystrophy after 10yrs
? prophylaxis Rx can be used after distal radius Fx to reduce chances of developing CRPS
What is first line Tx for Complex Regional Pain Syndrome
What off-label meds are used for pain control
Vitamin C 500 IU/d
Parenterals
Counseling
Sympatholytics
Therapy
CCB (a-2 agonists)
Anti-HTN/convulsants
TCA
Steroids
CRPS Tx therapy program utilizes PROM but ? is stressed more
? adaptive modalities are used for CRPS Tx
? medication is used for stellate blocks
AROM w/ stress loading
TENS Iontophoresis Contrast bath
Bupivacaine
Continue intrathecal administration of ? drugs are used for refractory CRPS Sxs
? are the possible sequelaes of CRPS
What are the possible s/e of stellate ganglion blocks for these Pts
Clonidine (a-2 agonsit) and Ziconotide (snail venom creates CCB)
Deformity
Neuromas
Contractures
Compression neuropathy
Seizures Hoarseness Arm numbness Weakness PTHx
How is gout Tx
How is CPDD Tx
What are the 3 stages of urate crystal deposition
Indometha/Naproxin (Sx<48hrs)
Colchicine/Glucocorticoids
Allopurinol/Probenecid
Aspiration- Dx and Thx
Steroid injection- 1-2joints
NSAID/Colchicine if multiple joints during acute attack
3 or > attacks/year= Colchicine prophylaxis
Acute Interval Chronic
Gout affecting 1st MTP is AKA ?
What other areas can be affected
If Pt has gout in the back they have ? type
Podagra
Ankle Tarsal Knee
Tophaceous
After initial gout attack, Pts can expect to remain ASx for ? long
Where can tophi development occur
Define Chondrocalcinosis
2yrs
Hand tendon sheath
Olecranon
Forearm extensor
Achilles
MC in women >80y/o:
CPPD X-ray findings of punctate/linear calcifications of articular cartilage/internal joints
? joint is affected by CPPD more than half the time
What joint involvement come after this MC site
How can CPDD be differentiated from OA/RA
Knee
Wrist MCP Hip Shoulder Elbow SPine
OA: synovial fluid, x-ray
RA: no bony erosion or tenosynovitis
CPDD Pts w/ severe neuropathic joint Dz usually have ? two underlying conditions
What 4 metabolic d/os are associated w/ CPDD
What is a rare adverse outcome of the CPDD Dz process
DM
Tabes Dorsalis
Hyperparathyroid
Hemochromatosis
Hypophosphatasia
Hypothyroidism
End stage arthritis
? is the most preventable cause of death for in-hospital PTs and 3rd MC cause of death in PolyTrauma
Define Virchows Triad
? surgical positions increase DVT risk
PE
DVT identification:
Stasis
Damage
Hypercoagulable
Supine
Abduction
Internal rotation
Flexion
How does general anesthesia increase DVT risk
? bioactive chemicals are released during this time and can increase the risk further
Post-hip arthroplasty Pts have been noted to have decreased levels of ? putting them at DVT risk
Dilation, Stasis
Histamines
Leukotrienes
Anti-thrombin 3
? are classified as high risk surgeries for DVT development
Clinically, what two sings are indicative of DVTs
What post-op findings are indicative
Total joint arthroplasty
Internal fixation of hip fx
Polytrauma
Spinal cord injury
+ Homan
Edematous
Painful
Fever/Leukocytosis
Pts that die of PE do so w/in ? time frame
What are the current mainstays of prophylaxis
What is the FDA approved drug for VTE prevention
<30min
Fondaparinux
LMWH
ASA
Warfarin
Desirudin- hirudin derivative
Indirect inhibition of ? factors can help reduce DVT formation
What is the MC used DVT prophylaxis for hip/knee arthroplasty
What is this drugs s/e
2a 9a 10a
Enoxaparin
Renally cleared- swithc to heparin if dec renal clearance
? is the MC used anticoagulation w/ INR goal of ?
This MC is better at preventing ? clots for Pts having total hip arthroplasty
Mechanical prophylaxis reduces VTE Dzs secondary to ? and ?
PO Warfarin: INR 2-2.5
Proximal
Increased fibrinolysis
Decreases stasis
How long is DVT prophylaxis continued after hip/knee/plasty surgery continued
Spinal trauma Pts may need a IVC filter placed if ? 3 criteria are met
Why is Warfarin bridged w/ Heparin
7-10 days
Hx of VTE w/ prophylaxis
Prolonged immobilizaiton
Chemical anti-coag is c/i
Warfarin reduced Protein C/S causing Pt to be hypercoagulable
How long is Warfarin used for?
Therapeutic heparin is monitored w/ ? lab results
How is Enoxaparin monitoring achieved
3mon
Activated partial thromboplastin
Anti-factor 10a levels
What is the Tx for acute PEs
Oral anticoagulation is then recommended for ? long after first PE
What is an adverse outcome of Tx
Admit w/ heparin, O2 and then Warfarin
6mon
HIT Type 2 (AKA White Clot Syndrome)
DISH is predominant in ? Pt population
How do these Pts present
Since this condition can also lead to spinal fusion, how is this differentiated from Ankylosing
White male >60y/o
Spine stiffness in AM/PM
Red hip ROM
Knee arthritis
Normal posterior apophyseal/SI joints
What landmark does dish follow in the in the cervical region
What are the two MC causes of cervical myelopathy
What may be seen on x-rays of the pelvis and ribs
PLL
1st: cervical spondylosis
2nd: cervical DISH
Whiskering- shaggy hyperostotic bone
What are the two MC Dxs encountered by Rheumatologists
What are the two suspected etiologies for Fibromyalgia
What type of cardiac murmur may develop fibromyalgia
1st: RA
2nd: Fibromyalgia
Genetics
Environmental
MVP- mid-systolic click w/ late systolic murmur
How is FMS Tx per FDA recommendation
Why do these Pts need to be started at the lowest dose possible
? meds need to be avoided
Pregabalin Duloxetine Milnacipran
Usually have hypersensitivity to Rxs
Steroids
What two meds can be used at bedtime for fibromyalgia
What is recommened for use if Pt develops/has depression
What meds are good for sleep maintenance
Amytriptyline
Cyclobenzaprine
Fluoxetine
Initiatiion: Trazadone
Maintain: Gabapentin, Tiagabine
? meds are added for Fibromyalgia PTs w/ Restless Leg Syndrome or MVP?
What are the most beneficial injections for pain relief?
? type of fitness program is recommended for these pts
Clonazepam
Pramipexole
Lidocaine
Saline if allergic
Aerobic 20-30min/day x 5 days/wk
What is an adverse effect Pts w/ fibromyalgia can develop in response to long term Amytriptyline or Cyclobenzaprine usage
Where/how does osteomyelitis usually affect Peds/Adults
How can neonates present w/ this
Tachyphylaxis- decreased responses
Peds: Hematogenous to metaphysis of long bones (can lead to septic arthritis)
Adult: open Fx/post-ORIF
Pseudoparalysis
How does osteomyelitis present in adolscents/adults
What images can be used for Dx of osteomyelitis but what is the best method for Dx
What do lab results look like in cases of osteomyleitis
Post-op: drainage, failed/delayed healing
MRI
Open biopsy/aspiration
Acute- elevated leukocyte, ESR/CRP
Chronic/ImmSupp- normal
ESR/CRP- Dz process marker
What are the MC organism to cause osteomyelitis in Peds and adults
What is an indication of surgical site infection in Pt w/ AIDS
What is a rare adverse outcome if chronic osteomyelitis develops
Peds: Staph A > GBS > HInflu
Adults: Staph A, Pseudomonas
CD4 <200
Malnutrition (albumin <2.5g)
Marjolin Ulcer- SCC metaplasia
Since surgical debridment is required for osteomyelitis Tx, what type of ABX usage is recommended
What are the 3 methods of septic arthritis development
What microbe is the MC cause of septic arthritis in PTs >2y/o
Parenteral/impregnated methyl methacrylate beads after surgery
Direct
Hematogenous
Extension
Staph A
IV drug users can develop septic arthritis in ? unusual locations
? microbe is their culprit
Septic arthritis in kids is MC spread by ? route
Sternoclavicular
Sacroiliac
Pseudomonas
Hematogenous
? Pt populations are at increased risk for developing septic arthritis
? is a common scenario for Peds to present w/ that have a septic arthritis Dx
What will older children present w/
Systemic Lupus
ImmComp
RA
Previously ambulatory, no refusing to bear weight
Anorexia
Fever
Irritable
Tachy
What are the hallmark PE findings of septic arthritis
If the hip is affected, it will usually be held in ? position
? lab marker is used for monitoring response to therapy
Tenderness/Effusion/Erythema w/ painful PROM
Flexed, Abducted
CRP
What lab result of a native joint indicates Dx of septic arthritis
What if it’s a prosthetic joint
What will glucose/protein results look like
WBC >50K
> 1,100 w/ neutrophils >64%
Low glucose
High protein
When assessing septic arthritis, if ? 3 microbes are suspected, the lab needs pre-notification
? form of imaging is useful to identify the location of infection
What are the next best steps after Dx of septic joint has been made
H Influenza
Gonorrhoeae
Kingella
Tc-99
Synovial/blood culture
IV ABX
Surgical decompression/lavage
? is the cornerstone of successful Tx of septic joint
Once transitioned to PO AB, how long is the regiment continued
What microbe and type of microbe causes Lyme Dz
Emergent surgical decompression
4-6wks
Spirochete: Borrelia burgdorferi
Lyme Dz is the most ?
What types are endemic to Europe and Asia?
What are the 3 phases of Lyme Dz
Prevalent vector-borne illness in US
B afzelli
B garinii
Local: viral Sxs
Disseminated: cardiac/neuro- Meningitis Rediculopathy Cranial neuropathy
Late: arthritis/neuro- paresthesia encephalopathy radiculopathy joint pain
? is the most important non-op Tx for Lyme Dz
Lyme Dz risk remains low if tick is removed w/in ? time frame
How are these PTs Tx w/ ABX
Skin/Clothing checked for ticks
<36hrs
Doxy 100mg BID x 28days
Amox 500mg TID x 28 days
<8y/o: Amox 20mgg/kg
What are the 3 types of osteoporosis
What types of Fxs can Pts present w/ that indicate the type they have
Osteoporosis is usually unnoticed until Pts present complaining of ? four issues
Primary 1: post-menopausal (6x F>M)
Primary 2: senile (2x F>M)
Secondary: M>F steroid Hx, MM, OM, OI, hyperpara/thyroid
Type 1: compression vertebral, distal radial
Type 2: hip, pelvis
Type 3: men w/ low energy Fx
Back pain
Fx
Lost height >2”
Spine deformity
? is the 10yr Fx risk assessment for Osteoporosis and what two factors are as important as low bone density
What is the name of the thoracic kyphosis these PTs can develop
What are the two DEXA scores provided and what are the ranges
FRAX: bone density + RFs
Old age + prior low energy Fx
Dowagers Hump
Z: peers
T: healthy, young PTs
0- -1: normal
- 1 - -2.5: osteopenia
- 2.5 or more: osteoporosis
Where does DEXA scan measure density at
What is DEXA best used for ?
Who needs to have DEXA scans d/t RFs
Spine FemNeck Trochanter Femur
Monitoring osteoporosis Tx
Female >65y/o
Post-menopause <65y/o
Men w/ Hx low-trauma Fx/prostate Ca
Primary Hyperparathyroid
What are the two sub-types of osteoporosis
When does bone mass density reach peak levels during life
What recommendations are given to reduce risk for osteoporosis development
High-turnover: high NTx score
Low formatoin: low NTx score
<28y/o
Ca/Vit D
Avoid alcohol/tobacco
Impact loading- walk, strength, Tai Chi
How much Ca intake is recommended for osteoporosis prevention
What are the two main forms of Ca for ingestion
What is the difference in these two method of breaking down during metabolism
750-1000mg/day
(range 25-45)
Carbonate
Citrate
Carbonate reqs acid
Citrate dissolves at all pH levels w/ dec risk for stone development
How much Vitamin D is needed for proper Ca absorption
How much Vitamin D intake is recommended
Fx from fall can be avoided w/ as little as ? Vit D intake/day
> 15ng to avoid insufficiency
30 recommended
800-1200 IU/day
2-4K if Vit D deficient
800
What is the 3rd MC cause of delayed Fx healing
How is Osteoporosis Tx w/ Rx
Low Vit D
High NTx= antiresoptives (disphosphonates, -ate)
SERM: Denosumab
Low NTx: Anabolic (intermittent PTH)
? medication is used to increase spinal bone mass but doesn’t dec risk for hip Fxs but w/ ? s/e
? is used in Pts w/ dec renal function
? medication has mild spinal Fx protection and possible pain relief but w/ 2 s/e
Raloxifene- inc DVT risk and hot flash occurance
Denosumab- inhibits osteoclast formation
Calcitonin
No non-vertebral Fx protection
Inc Ca risk
When are anabolic agents recommended for use against osteoporosis
When is their use c/i
Prolonged diphosphonate usage is associated w/ ? 2 adverse outcomes
Pre-menopause
Impaired Fx healing
Diphosphonate failure
Low turnover
Radiation Hx
Paget Dz
Children
Atypical femur Fx
Jaw necrosis
What are the three parts assessed for overuse syndromes during PE
+ Phalen test means ? Dx
How are overuse syndromes Tx and what type of rehab program is useful in Tx
Inspect: Atrophy Pallor Erythema Swelling
Palpate: Point of max tenderness
Strength for pain w/ resistance
De Quervains (APL, EPL)
Protection Rest Ice NSAID Cream Eccentric
Sprains are uncommon in ? population
Instead of sprains, these Pts usually end up w/ ? Dx
What are the 3 degrees of Sprains
What are the 4 grades of Strains
Open growth plates
SALTR Harris Fx
1: partial w/ no instability
2: partial w/ laxity
3: complete w/ laxity
1: <10% muscle, intact fascia
2: 10-50% muscle, intact fascia
3: 50-100% muscle, intact fascia
4: 100% tear w/ disrupted fascia
What is assessed in strains or sprains during PE and what imaging modality is best
All sprain/strain need x-rays if pain is still present after ? days
How are Sp/trains Tx
Point of max tenderness
Sprain: joint stability
Strain: stretch injured muscle for defect
MRI: confirm/grade/rupture
7-10
PRICE- mainstay Cryotherapy NSAIDs
Minor sprain- compression, immobilize
Minor strain- immobilize w/ muscle stretched
When do sprain/strains need to be referred to Ortho
What are the 3 stages of benign tumors
What are 3 terms used for benign growths
Grade 4 strain, all Grade 3, Sev Grade 2
Latent Active Aggressive
Well-defined
Non-aggressive
W/out cortical destruction
W/out periosteal reaction
? Sx indicates a bone tumor has weakened the structural integrity
What is a critical part about evaluating bone tumors on x-ray?
Most benign tumors don’t weaken the underlying bone w/ ? exceptions
Sxs exacerbated w/ activity
Pattern recognition
Osteoid osteoma
Osteochondroma
What Sxs are absent in Pts w/ benign bone tumors
If these Sxs are present, ? Dx should be suspected
? is the MC scenario for bone tumors to be found
Constitutional Sxs
Mets Infection Lymphoma
Incidental finding
What is the best imaging modality for suspected bone tumor assessment
What other imaging modalities are used and what are the pros
What do bone scans use for imaging
Radiograph
MRI better: soft tissue/marrow
CT better: bone detail
Tc-99m: isotope bound to ligand methylene-diphosphonate
PTs >40y/o w/ aggressive bone lesions will probably have ? types of Ca
If a primary site can’t be located after detailed Hx/PE, what is the next step
What blood tests may be done to help w/ Dx in Pts >40y/o
Metastases
Myeloma
CT- chest abdomen pelvis
Light Immunoglobin Microglobulin Electrophoresis
Quant immunoglobin
Protein electrophoresis
Free light chain assay
B-2 microglobulin factor
What are the two MC methods for obtaining bone biopsies for suspected neoplasms
What type of benign tumors can cause pathological Fx and loss of function
These Pts are at risk for ? two things if they become immobile
Closed needle, Open bone
Active/Aggressive
HyperCa
Pneumonia
How are benign bone tumors Tx
? drug plays a vital role in managing established bone mets
What is the theorized etiology of growing pains in ? population MC
Active/Aggressive: surgery
Primary tumor, Peds: Chemo, Surgery
Mets: Rad/Chemo/Surgery
Disphosphonates
Over activity- muscle strain/fatigue
Boys 2-5y/o w/ ligamentous laxity
What may be found on PE in suspected growing pains
What part of the body is MC affected
what is done for management/Tx
Pain w/ deep pressure
Flexible flatfeet
Calves
Stretching Education Analgesics
Pt w/ suspected growing pains may need metabolic work up if ? two Dxs are possible
What is more common about CRPS in Peds
Where do these Pts MC have skin color changes
Leukemia
Endocrinopathy
Type 1: MC extremeties in Peds 9-15y/o
Ankle/Feet
What are the S/Sxs of long term CRPS in Peds
What two meds are used for Ped Pts that don’t respond to rehab
Most cases of child abuse occur in PTs ? old w/ ? populations at higher risk
Muscle wasting/contracture
Coarse hair, extremity
Thick nails
Amitriptyline
Gabapentin
<3y/o
First Handicapped Stepchildren Premature
Toddlers commonly have bruises located ?
What is the next step in evaluation if a child’s mental status is abnormal
What is the name and age criteria for the series of x-rays done for these suspected abuse cases
Brow Elbow Chin Knee Shin
Subdural hematoma
Retinal hemorrhage
Skeletal survey: <2y/o
Fx highly suspicious for abuse
Fx moderately suspicious for abuse
Bone scans can be used to assess for rib fxs in suspected abuse, what would be seen in healed Fxs
Post ribs Corner long bone metaphysis Scapular Process, spinal Chip long bone metaphysis Sternum
Multiple/Bilateral/Aged/ Fxs Fingers Epiphyseal separation Vertebral body Skull, complex
Fusiform thickening
How is the age of a Peds Fx assessed by imaging
How are Fxs older than 6wks best assessed
? type of wrist Fx is not associated w/ Fx
7-14d: new periosteal/callus
14-21d: lost Fx line, mature callus/trabecula
21-42d: dense callus
>42d: sublte fusiform sclerotic thickening
Eval for thickening by comparing to contralateral side
Buckle
What system is used to describe Peds Fxs involving the physis
? type of x-ray increases the ability to view these Fxs
What are the adverse outcomes from these types of Fxs
Salter-harris
Oblique
Premature growth arrest
Dec bone length
What are the Tx goals of Peds Salter-harris Fxs
How are these Fxs Tx
Mild displacement is allowed in ? gender that are ? age
Reduction/Avoiding arrest x 6wks
Type 1-2: closed reduction, cast immobilization
3-4: reduction w/ ORIF
Boys 15 and >
Girls 13 and >
Kids younger than 13y/o should not have any Fx older than ? reduced
Salter Fx Types 3-4 require surgery d/t ? structures involved
These require reduction to ensure congruent surfaces to prevent ? formation
> 7 days
Cartilage of growth plate and articular surface
Physeal bars
How long do Peds w/ Salter-harris Fxs need f/u
? types of Fxs require longer f/u process
How is JIA named differently in the US and Europe
12mon, less if they reach skeletal maturity before f/u appointment
Femur, Tibia
JRA: USA
JCA: Europe
There are at least seven types of JIA, but they all have ? two things in common
What are the types of facts of each
Chronic arthritis x 6wks
Pt is <16y/o
Systemic: Fever Arthritis Rash Adenopathy Hepa/Spleno-megaly Pericarditis
Oligoarticular: 4 or < joints, high risk ASx uveitis
RF neg-poly: RF-,, 5 or > joints
RF pos-poly: RF+, 5 or > joints
Psoritatic: first degree relative
Enthesitis: SI, enthesitis, HLA-B27
Udifferentiated: doesn’t fit elsewhere
What joint needs to be palpated when assessing suspected JIA in ASx Pts
What are adverse outcomes from this Dz
How is this Tx
TMJ
Joint arthritis/destruction
Blindness from un-Tx uveitis
NSAID- first line
Few joints: intra-articular CCS
Unless arthritis is mild, DMARD (Methotrexate) or a-TNF (Etanercept, A/I-umab)
What two meds are used for Ped PTs w/ refractory JIA uveitis
When would splinting be recommended
What is an adverse outcome to Tx for these Pts that are on a-TNF meds
Inflixiamab
Adalimumab
At night for contractures
Fungal/TB infection
Define Osteochondritis Dissecans
Where does this d/o MC occur and where can it occur
Where does it rarely develop
Osteonecrosis of subchondral bone
MC- posterolateral medial femoral condyle
Talus Humerus Elbow Femur
Patella
What do Pts w/ OCD in the knee report as a pain relieving maneuver
What test may be positive on PE for this condition
What is the goal of Tx and how are these Tx
Walking toe out
Wilson Test
Let lesion heal
Non-op: Peds w/ lesion <1cm, LLD, crutches, refractory due to noncompliance= immobilization
Surgery: mature/cartilage has separated or lesion >2cm
Why is hematogenous spread of microbes in Peds most likely to infect metaphysis of long bones
What is the sequence of infection progression
What is the difference between osteomyelitis sequestrate and involucrum
Circulation creates u-turn, slowing flow down
Canal, Cortex, Abscess formation
Seq: abscess inc pressure= bone fragment
Persistence leads to chronic osteomyelitis
Involucrum: periosteum remains, new bone growth
How does subacute osteomyeltits present in Peds and d/t ? microbes
X-rays of these Pts can show ? finding that can mimic ?
How do these Pts appear in clinic
Indolent- bacteria/TB
Lytic- aggressive appearance like a tumor
Pain Malaise
Warmth Erythema Swelling Tenderness
What is an early Sx of Ped osteomyelitis if the infection is in the pelvis or spine
What is seen if the upper extremity is involved
How does chronic osteomyelitis present
Refusal to walk/limp
Pseudoparalysis
Sepsis
Sinuses w/ chronic drainage
? is typical PE finding for Peds w/ Acute Hematogenous Osteomyelitis
What lab results will be elevated earliest during the Dz process
What are the MC joints involved
Fever >100.4
Tenderness over bone
Effusions
CRP, <8hrs of infection onset
Intra-articular:
Prox- Femur Humerus Radius
Distal-Fibula
How does subacute osteomyelitis appear on x-ray
How does AHO appear differently on x-ray
? is the imaging modality of choice for Dx AHO
Lytic lesions w/ thin sclerotic rim and crosses physis
Physis sparing
MRI
What are the next steps of Tx once a case of Peds osteomyelitis is suspected
? microbe is MC the cause
? other microbes need to be covered when considering ABX coverage
Culture/biopsy
IV ABX
Staph A
GBS
Neonate: enteric rods
6-48mon: H influenza
What special step is needed if Kingella kingae is the suspected culprit of Peds osteomyelitis
When are these Pts switched from IV to PO ABX
How long are ABX recommended
PCR and special culture media
7 days
6wks
What is almost always needed to Dx subacute osteomyelitis
? Tx step provides the best likelihood for Dz eradication
Why is immobilization recommended and for ? long
Biopsy from surgical debridment
Removal of all infected material
3-6wks
Dec pain
Reduces chance of pathological Fx
Septic arthritis in kids is usually d/t ? route and microbes
Septic joints will have ? lab results
What joints are most likely to be affected
Hematogenous seeding of synovium from:
Skin infections
Impetigo
Pneumonia
ESR >30
WBC >15K
Synovial WBC >50K
Knee Hip
Septic arthritis needs to be ID’d early in Peds and can be done so by ?
What signs may be present when this Pt arrives
If the hip/elbow/knee is involved, Pts hold it in ? position
Hyaline cartilage damage from lymphocytic enzymes <72hrs from inoculation
Guarding Fever Anorexia Malaise
Hip: Flex Abducted External rotation
Knee/Elbow: slight flexion
When assessing Peds Pt for septic arthritis, how is their presentation different if the underlying Dx is Transient Synovitis or Legg-Calves Dz
? lab marker is best for monitoring Ped Septic Arthritis
Septic joints will have ? lab results
TS/LC: discomfort instead of pain
CRP
ESR >30
WBC >15K
Synovial WBC >50K
How is Ped Septic Arthritis Tx
Pediatric Seronegative Spondyloarthropathies have ? 4 characteristics in common
? c/c presentation suggests a case of anklyosing spondylitis
Joint aspiration/drainage
IV ABX
HLA-B27
Inflammation of tendon/fascia/enthesitis
Pauciarticular arthritis in LE
Extra-articular inflammation
Asymmetric peri-articular arthritis of the lower extremeties in kids 9 or >
Peds Reiters Syndrome is a triad of ? three Dx
This conditions can be triggered by diarrhea caused by ? microbes
What can cause the non-gonorrhea urethritis in adolescents
Conjunctivitis Enthesitis Urethritis
Yersinia Campylobacter Salmonella Shigella
Trachoma
Chlamydia
Peds w/ Reiters in what two locations are particularly painful
How does Peds w/ Psoriatic arthritis tend to present
Peds IBDz arthritis usually presents prior to ? and ? prevelance is twice as common
Achilles or Plantar Fascia
Female <15y/o w/ skin problems before arthritis
<21y/o w/ arthralgia w/out effusion
What PE finding is a distinguishing feature of juvenile spondyloarthropathies
Peds w/ Ankylosing Spondylitis may have enthesitis in ? locations
What extra-articular Sxs do Peds w/ Reiters present w/
Purple discoloration around joint
Patellar Achilles Plantar
Conjunctivitis
Anterior uveitis
Photophobia
What is the most common manifestation for Peds w/ Psoriatic arthritis
Involvement of ? three locations is more common w/ Psoriatic than other Spondylonegatives
What lab result supports a Dx of juvenile Reiters Syndrome
Monoarticular knee
Digit tenosynovitis
UExtremity involvement
Nail pitting
Sterile pyuria
How are Peds w/ Spondyloarthropathies Tx
What is the name of the distal end of the spinal cord that ends at ? level
Anything below is AKA and if compressed presents as ?
Muscle strengthening
Orthoses
Activity modification
NSAIDs
Conus medullaris ending at L1-2
Cauda equina: L2-S4
Paralysis w/out spasticity
How does Cuada Equina present on PE
What special tests are done for suspected Cauda Equina Syndrome
How can Pts seen in the ER for back pain be mis-Dx w/ Cauda Equina
Bilateral radiculopathy
Incontinence
Foot drop
Stumbling gait
Inability to rise from chair (quad/extensor test)
Inability to walk on heels (ankle dorsiflexion, plantar flexion)
If given narcotic injection, causes acute urinary retention
How is Dx of Cauda Equina confirmed w/ imaging
What is a possible error that could be an adverse outcome to this Dz
What is the usual cause of cervical radiculopathy in young/older PTs
Compressed thecal sac on CT/Myelogram
Blame bladder Sxs as Cystocele/Prostatism w/out considering sphincter paralysis
Young: herniation traps root in foramen
Older: stenosis/arthritis
What will usually be seen on PE of cervical radiculopathy
Stenosis of the cervical spine commonly present w/ ? Sxs
Pts will report ability to relieve Sxs w/ ? maneuver
Radicular pain w/ UE numbness/paresthesia (deltoid to thumb)
Changed grip/handwriting
Trunk/leg dysfunction
Gait disturbance
Incontinence
Place hands on top of head
What type of neck malformation may be present and restrict movement in cervical radiculopathy
? motor/sensory tests need to be done for these PT
How is this Dx confirmed w/ imaging
Reduced cervical lordosis
C5-T1
UE reflexes
CT/Myelogram w/ contrast
How is Cervical Radiculopathy Tx
What two Txs are avoided in this population
Define Cervical Spondylosis
Most spontaneous x 8wks
NSAIDs + traction
Opioids
Manipulation
Degenerative disc dz of the cervical spine
What causes the Cervical Spondylosis dz process
What are the MC Sxs of Cervical Spondylosis
What Pt is morelikely to have spinal stenosis and myelopathy w/ this condition
Herniation
Osteophyte growth
Thick ligamentum flavum
Limited mobility
Pain worse w/ upright
Older men
What are 3 Sxs of early cervical mylopathy from cervical spondylosis
What PE findings will be abnormal
This type of abnormality suggests ? structure is involved
Palmar paresthesis
Altered gait (heel-toe)
Difficult dexterity
Lost vibration/proprioception in the feet
Posterior column
? sensory and motor function tests are needed for PTs w/ Cervical Spondylosis
What two special neuro tests may be positive in cervical spondylosis PTs
X-rays may reveal osteophytes originating from ? landmark
C5-T1 and L1-S1
Lhermitte sign Hoffmann Clonus Hyper-reflexia Babinksi
Zygoapophyseal joints
Where are cervical spondylosis/age-related degenerative findings MC seen
How is this Tx
What two meds can be used for sleep aids
C5-7
Cervical pillow
NSAIDs
Surgical decompression
Doxepin
Amitriptyline
What is the classic mechanism for whip-lash injury
What is the MC findings on PE
How are these Pts Tx
Stopped car that is rear ended= flexion/extension
Non-focal/radicular neck pain
NSAIDs Soft collar Muscle relaxants Cervical pillows Doxepin/Amitriptyline
What 3 x-rays are ordered for cervical strains and what is a normal measurement obtained
What is the next step and measurement if severe pain is present
What image should not be ordered until after eval by specialist
AP/Lat/Odontoid- pre-vertebral tissue width at C3 should be <1/3 width of C3
Vertebral body translation 3.5mm or more and/or 11* of angulation
Flex/Extension images
What type of rehab is recommended post-cervical strains
What is the most important x-ray obtained for multiple injury trauma Pt
What are the MC missed injuries
Walking early
Isometric exercises when tolerated
Cross-table lateral view of C1-T1
Injury to upper/lower C-spine
What type of x-ray is needed for trauma Pt to evaluate the cervical-thoracic junction
How are neck injured Pts Tx if cleared by imaging but pain persists
Flexion-distraction injuries of the T/L spine usually alos have ? injuries too
Swimmer view
Cervical collar x 7-10 days
Abdominal- bowel lac
What secondary issue can develop in Pts w/ lumbar spine Fxs
What are the hallmark PE findings of an unstable T/L flex-distract or burst Fx
Burst Fxs tend to involve ? column of the spine and best seen w/ ? image
Ileus- dec bowel motility
Hematoma w/ step off
Middle, CT
Any vertebral Fx other than ? requires additional imaging
Isolated transverse process Fxs need to be inspected for injury to ? and can be Tx w/ ? to dec Sxs
Compression Fxs w/ ? measurements are also tx the same method x 8wks
Single compression
Kidney, thoracolumbar corest
Wedging <20*
No posterior involvement
? is the MC cause of disability and lost time at work for Pts <45y/o
What causes the irritation process for this condition
What is used to monitor progress
Acute low back pain
Injury to anulus fibrosus= nucleus pulposus leak= irritation
Lumbar flexion
Ease of extension
Acute LBP that need x-rays need to have ? landmark in the picture
What are the two phases of Tx for acute lower back pain
When does this type of back pain become reclassified to chronic lower back pain
T10
Initial: Sx relief
Secondary: return to activity
Pain >3mon
All Pts w/ chronic lower back pain need to be evaluated by ? providers
Identifying ? underlying issue w/ Chronic LBP can help Sx resolution
Often there is ? sign seen on x-rays
GYN Internist FamMed Spine
Depression
Vacuum- Nitrogen in air space
What type of material is found in the nucleus pulposus
What are the 3 parts of the intervertebral disc
What two movements increase pressure on the nucleus pulposus
Collagen Type 2
Nucleus pulposus
Anulus fibrosus
Sup/Inferior end plates
Twisting
Lifting
Where do lumbar herniations MC occur
What nerve root is irritated
Herniations located in ? areas tend to NOT have radiculopathy below the knee and have ? Sx
L4-S1
L5-S1
L1-4, pain in anterior thigh
? PE test has high correlation to lumbar herniation
What test is even more specific though
When performing the supine straight leg raise, this maneuver pressures ? area
Seated leg raise
Crossed straight leg raise
L5-S1 is stretched
What will be seen in L3-4 herniation onto L4
What will be seen on L4-5 herniation onto L5
What will be seen on L5-S1 herniation onto S1
Weak anterior tibialis, asymmetric knee reflex
Great toe extensor weakness, numb dorsal foot/lateral calf
Unable to toe walk, lateral foot pain, asymmetric ankle relfex
When is an MRI ordered for suspected lumbar herniations
How many epidural injections can these PTs receive
When should the injections be avoided
Sxs >4wks
3 in 6mon
Substantial neuro deficit
Motor, Reflex and Sensation for L4 nerve root
Motor, Reflex and Sensation for L5 nerve root
Motor, Reflex and Sensation for S1 nerve root
Anterior tibialis / Patellar / Medial foot
Extensor hallucis longus / NONE / Dorsal foot
Gastroc soleus (toe raise) / Achilles / Lateral foot
Why would a PT younger than 60y/o experience lumbar stenosis
Where does stenosis typically develop
? type of movement tends to narrow the lumbar region
Achondroplasia
L2-5
Spine extension
What special tests should be done for suspected lumbar stenosis?
What is uncommon and ? area is rarely affected by this condition
Lateral x-ray views need to have ? landmark included
Proprioception/Romberg/Neurovascular
Leg muscle weakness
Uncommon sphincter tone decrease
T10
How is lumbar stenosis Tx non-op
When do these Pts become surgical candidates
What is the goal of Tx
Water exercise
Epidural injections
Non-ambulatory/Dec quality of life
Prevent progression
What type of malignant tumors of the spine are considered rare/common
Highest incidence of spinal carcinoma is d/t ? and via ?
How are Cas to the spinal column spread via hematogenous
Primary- rare
Metastatic- common
BLT KPC by hematogenous spread
Batson’s plexus- connects w/ inferior vena cava
Malignant tumors of the spine can present in ? ways
? is the MC presenting issue for these Pts
What is the first manifestation these appear as on x-ray
Pain as presenting c/c
Incidental
Neuro findings
Known primary tumor
Pain, usually from minor vertebral Fxs
Lost pedicle integrity (winking owl)
What is the best screening study for widespread mets after suspected spinal neoplasm
This test will usually be negative in Pts w/ ? Dx
What are the most severe sequelae of pathological Fxs induced by these mets
Tc-99m bone scan
Multiple myeloma
Quad/Paraplegia
How are ASx spinal neoplasms found during the search for mets Tx non-op
How are painful metastasis Tx
When is surgery indicated
Chemo/Rad/Hormones
Radiation if no deformity/neural compression
Pain w/ neurodeficit- decompression and stabilization w/ post-op radiation
What is a common adverse outcome after surgical decompression of spinal neoplasms?
Define Scoliosis
What is the MC presenting Sx
Wound complication if surgery is post-radiation/steroid
Coronal curvature of spine >10* using Cobb method
Pain in region of deformity
What is the MC overlapping condition seen w/ scoliosis
If these PTs have radiculopathy it’s because of ? compression
Neurological findings are rare but ? is the MC
Degenerative spondylosis
L4-5
Hallucis Longus
How is decompression in scoliosis assessed
How is adult scoliosis Tx
What are the red flags for referral in these Pts
Plum line- C7 to gluteal cleft
NSAIDs
Water/swimming therapy
Neuro deterioration
Can’t walk 2 blocks d/t pain
Respiratory dysfunction
Trunk exercise
Define Degenerative Spondylolisthesis
What is the opposite direction of slippage called
What nerve roots need to be evaluated
Female >40 L4-5 body slips fwd d/t deteriorated facets/disc leaving lamina/pars interarticularis intact
Retrolisthesis- posterior slippage
L1-S4
What neuro findings are seen in Pts w/ Degenerative Spondylolisthesis
Narrowing of ? two spaced in degenerative spondylolistesis causes ? types of pain
Where does pediatric isthmic spondylolisthesis usually develop
Dec knee reflexes, also seen in geriatrics
Weak toe/heel walking
Weak toe dorsiflexion
Lateral recess= radiculopathy
Central canal= claudication
L5-S1
Isthmic Spondylolisthesis develops at ? junction
This form of the condition is more likely to represent ? event
If only the defect is present, and no slippage has occurred? the PT has ? Dx
Lamina w/ pedicle (pars interarticularis)
Cyclic loading AKA- fatigue Fx that fails to heal
Spondylosis
? activities put Pts at higher risk for developing Isthmic Spondylolisthesis
How do Pts w/ isthmic spondylolisthesis present to clinic
What may be seen on PE
Gymnastic/Football
Posterior pain radiation below knees, worse w/ standing
Dec lordosis/flat buttocks
Vertebral step off
Hamstring spasm w/ forward extension/leg raise
What area of the lumbar spine can become compressed during Isthmic Spondylolisthesis
What is the x-ray finding name for this condition
Since Peds are considered skeletally immature, what imaging test is ordered to assess metabolic activity at site of defect
L5
Collar on scotty dog
Single Photon Emission Test- SPECT CT
How are cases of Isthmic Spondylolisthesis Tx
? is the MC cause of thoracic and lumbar pain in kids?
Spinal hyperextension loads posterior spine to test for ? while flexion loads anterior spine to test for ?
Metabolically active and skeletal immature= rigid brace
Surgery: refractory, high grade slip
Skeletal mature: no fixation, NSAID, exercise
Muscle strains
Extend: posterior- spondylolysis
Flex: ant- discitis, compression Fx
Abnormal abdominal reflexes may be the only sign Peds Pt has ? Dx
What is the initial imaging method of choice for Peds w/ back pain
Discitis and vertebral osteomyelitis in Peds are issues involving ? d/t ?
Syringomyella
Weight bearing PA/Lat x-ray
Discitis: MC Staph A in anterior spine in kids <5y/o
Osteo: Staph A in vertebral column in Pts >5y/o
Where is discitis MC seen in Peds
Other the MC microbe causing discitis, what other 3 microbes can cause this Dx
What special tests are performed and what is the imaging modality of choice
Low thoracic/Lumbar
Kingella E coli GAS
Percussion- localizes
Passive flex- pain due to anterior element compression
MRI
What provacative test can be done for Peds Pts w/ suspected discitis
? other Dx test should be considered in these populations
? abnormal lab results may be seen in Peds w/ discitis/vertebral osteomyelitis
Pick up- will avoid bending back to retrieve item
TB skin test
Normal WBC w/ inc ESR/CRP
What are common adverse outcomes in Peds w/ discitis/vertebral osteomyelitis
How are these PTs Tx by non-op methods
When is surgery/biopsy indicated
ASx persistent disk narrowing and spontaneous vertebral fusion
Empiric bed rest, LLD, analgesics
IV ABX x 2wks then PO x 4wks
Orthosis worn x 6wks
Non-responsive to empiric Txs