PVD & VD <3 - patho E3 Flashcards
risk factors for PVD
smoking
diabetes
high chol
heart disase
stroke
increase age >50
etiology of PVD
-atherosclerosis
-a thrombus
-inflammation (thromboangitis obliterans)
-vasospasm (raynauds)
clinical manifestations of PVD
-pain (calf & buttock)
-numb, burning, intermittent claudication
-wounds that do not heal
-diminished sensation in the extremities
-trophic skin changes (shiny, thick toenails, loss of leg hair, diminished pulses, pallor/cyanosis, red legs when they hang, erectile dysfunction)
intermittant claudication
consistent pain precipitated by consistent level of exercise
always when walking & stops w/ rest
5 P’s of PAD
-pain
-pulselessness
-palpable coolness
-paresthesias
-paresis
how to dx PVD
ankle - brachial index: BP in leg vs arm, normal is >1
PAD S/s
-intermittent claudication pain
-no edema
-no pulse
-no drainage
-round, smooth sores on toes & feet
-black eschar
PVD S/s
-dull, achy pain
-lower leg edema
-pulse present
-drainage
-sores w/ irregular borders on ankles
-yellow slough or ruddy skin
CVI causes & treatment
blood to pool or collect in veins -> venous stasis
treat w/ anti platelets & coags, thrombolytics & lipid lowering agents
CVI s/s
lower extremity edema
achiness or tiredness in legs
leathery looking skin
stasis ulcer on ankles
flaking or itching skin
new varicose veins
pharm treatment of PVD/PAD
-anti platelet (aspirin)
-anti coag (warfarin)
-antiilipemics
-antihtn
the AV valves are
tricuspid & mitral
open during systole & close w/ diastole
the ventricular valve are
pulmonic & aortic
valve abnormalities: pannus
failure to for the valve leaflets to close all the way
endocarditis usually leads to what
a valve replacement