Atrial Dysrhythmias Flashcards

1
Q

what are the atrial dysrhythmias

A

-Afib
-Aflutter
-premature atrial contraction (PAC)
-paroxysmal supraventricular tachycardia (PSVT)

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2
Q

what is PAC

A

ectopic pacemaker in atrium discharges before SA node fires, usually a one time beat
isolated premature atrial beat

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3
Q

what causes a PAC

A

-usually benign but if new onset or increases in occurrence -> check electrolytes (mg, ca, k)
-stress
-caffeine or stimulants
-may indicate atrial pathology

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4
Q

an increase in frequencies of PAC may indicate what

A

patient is about to convert to afib

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5
Q

if you notice new onset of PACs, what is your next best nursing action

A

connect the provider b/c we can give meds to prevent them from converting to afib

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6
Q

how to treat benign PAC

A

do not need to treat

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7
Q

what is Afib

A

total disorganization of atrial electrical activity due to multiple ectopic foci, resulting in loss effective atrial contraction (“kick”, p wave)
atria is quivering b/c SA node is no longer in charge of pacing

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8
Q

characteristics of afib

A

-no p wave
-R to R intervals are irregularly irregular
-atrial rate > 400 bmp
-ventricular rate >100-175 bmp
-most common dysrhythm
-increases w/ age

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9
Q

can you live w/ afib

A

yes as long as ventricular rate is controlled

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10
Q

what causes afib

A

person usually has underlying heart disease
-electrolyte imbalance
-hypoxia
-cardiac surgery

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11
Q

main risk if a person if in afib

A

afib leads to a fibrillating atria -> pooling of blood -> clot formation -> risk for embolus -> can throw clot to the brain causing a stroke

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12
Q

what do symptoms of afib/aflutter depend on

A

ventricular rate
how long rhythm has been present
patient’s CV status

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13
Q

S/s of Afib & aflutter

A

see key features

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14
Q

what is Aflutter

A

a tachydysrhythmia that originates from a single ectopic focus -> re entry impulse is repetitive & cyclic

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15
Q

characteristics of aflutter

A

-ID by recurring regular, saw tooth shaped flutter waves
-R to R is regular or irregular
-atrial rate may be >250 bmp
-ventricular is lower than atrial bc not every impulse is getting to the ventrical

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16
Q

what are the waves called in between the QRS in aflutter

A

F waves

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17
Q

what causes aflutter

A

rarely occurs in a healthy heart
electrolyte imbalance

18
Q

what is a big risk of being in aflutter

A

emboli formation

19
Q

what is the goal of treating afib/aflutter

A

-control ventricular rate (priority)
-rhythm control
-prevent embolic stroke

20
Q

what are the rate control drugs used for afib/aflutter

A

-beta blockers
-CCB (diltiazem, verapamil)
~digitalis
give IV first

21
Q

what are the rhythm control drugs used for afib/aflutter

A

-amiodarone
-dofetilide
~digitalis
give IV first

22
Q

drugs to prevent clots

A

warfarin

23
Q

how to treat stable afib/aflutter

A

IV medications using a bolus then maintenance drip

24
Q

how to treat unstable afib/aflutter

A

synchronized cardioversion

25
Q

how to treat afib/aflutter that has been occurring for >48 hrs

A

put patient on anticoagulant for 3 to 4 weeks before cardioversion and for 3 to 4 weeks after successful cardioversion

26
Q

what needs to be done before a cardioversion if the patient has a history of afib

A

perform an TEE to confirm there is no clots in the atrium

27
Q

if treatment for afib/aflutter doesn’t work

A

long term use of anticoag is required (warfarin or new class)

28
Q

what are the new class of anti coags

A

dabigatran, apixanabn, rivaroxaban, eboxaban

29
Q

if you are on long term warfarin

A

have to monitor INR regularly

30
Q

if you are on long term new class anti coag

A

routine testing not required but there is no antidote for the drug, cost & contraindicated for a pt w/ impaired renal function

31
Q

what limits drug therapy for afib/aflutter

A

-high failure rates
-potential for drug toxicity

32
Q

non pharmacology treatments for afib/aflutter

A

-catheter ablation
-maze procedure
both in cath lab

33
Q

what does PSVT usually start with

A

a PAC

34
Q

what is paroxysmal supraventricular tachycardia (PSVT)

A

originates in ectopic focus anywhere above bifurcation of bundle of his

35
Q

characteristics of PSVT

A

-runs of repeated premature beats
-abrupt onset and termination (unless sustained and then it doesn’t terminate)

36
Q

what causes PSVT

A

-overexertion
-emotional stress
-stimulants
-digitalis toxicity
-various forms of heart disease

37
Q

what does symptoms of PSVT depend on

A

how long it lasts and how fast the ventricular rate is

38
Q

S/s of PSVT

A

look at key features

39
Q

how to treat PSVT (primary)

A

ensure they are connected to monitor
-valsalva/bear down (most effective)
-coughing
-carotid sinus massage (MD only)
-diving reflex/cold water immersion (rarely used)

40
Q

what medications can be used to treat PSVT if primary treatment did not work

A

adenosine IVP to slow HR

41
Q

adenosine facts

A

-very short half life so follow w/ rapid NS flush
-onset: 10 to 40 seconds, duration 1-2 mins
-may cause a pause (asystole) on rhythm strip, pt may be aware

42
Q

if all treatments (primary & drug) treatment fail for PSVT and patient becomes hemodynamically unstable, what is the next best action

A

cardiovert the patient