ABGs - patho 406 Flashcards
purpose of ABGs
to assess acid base status and to determine adequacy of oxygenation & ventilation
primary event
the problem that initiates the acid base imbalance
(ex: hypovent, hypervent, vomiting, diarrhea)
primary disorder
what results from the primary event
(ex: resp acidosis, metabolic alkalosis)
compensation mechanisms
physiologic processes that adjust the pH back to the normal range
(ex: lung problem -> kidney will comp)
general causes of imbalance: metabolic
-HCO3 level changes secondary to metabolic alterations (kidneys)
general causes of imbalance: respiratory
-H2CO3 level changes secondary to respiratory alterations (lungs)
respiratory acidosis or alkalosis
increases or decrease in CO2
change in ventilation
metabolic acidosis or alkalosis
changes in H+ or bicarb ions
acid base mnemonic: ROME
R espiratory
O pposite
inc pH, dec PCO2 = alk; dec pH, inc PCO2 = acid
M etabolic
E qual
inc pH, inc HCO3 = alk; dec pH dec HCO3 = acid
metabolic acidosis: what is it & what is our comp
dec HCO3, dec Ph
comp: lungs blow off CO2 to decrease levels (dec pCO2)
respiratory alkalosis: what is it & what is our comp
dec CO2, inc pH
comp: kidneys get rid of bicard (HCO3) to decrease levels
(dec HCO3)
respiratory acidosis: what is it & what is our comp
inc pCO2, dec pH
comp: kidneys hold on to bicard to increase levels
(inc HCO3)
metabolic alkalosis: what is it & what is our comp
inc HCO3, inc pH
comp: lung hold on to CO2 to increase levels
(inc pCO2)
metabolic alkalosis: ABG
too much bicard or not enough carbonic acid
pH > 7.48 (B)
PaCO2: 35-45 (N)
HCO3 >29 (B)
metabolic alkalosis: causes
-taking excess baking soda, alka-seltzer (H shifts out of the cell & K+ goes in causing hypoK)
-prolonged vomiting
-NG tube suctioning
-diuretics (lose H but bicard stays)
metabolic alkalosis: clinical manifestations
CNS over excitability
confusion
tremors
muscle cramps
parethesias
coma
N/V/D
resp depression (to try to hold on to CO2)