HLTH angina and MI Flashcards

1
Q

what does coronary heart disease include?

A

angina pectoris or myocardial infarction

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2
Q

what is the result of coronary heart disease?

A

damage to the myocardium due to obstruction in the coronary arteries, causing low O2 delivery which can lead to heart failure, death, or serious arrhythmias

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3
Q

what is chest pain the result of?

A

angina

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4
Q

angina and four causes

A

is chest pain when there is impaired supply of O2 to the heart or when the heart is working harder than usual; caused by obstructions such as atherosclerosis, arteriosclerosis, vasospasm, or myocardial hypertrophy

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5
Q

atherosclerosis

A

build up of materials in the vessel walls

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6
Q

arteriosclerosis

A

refers to arterilar disorders such as hardening of the vessel wall

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7
Q

patterns of chest pain in angina

A

classic, variant, or unstable

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8
Q

variant pattern of angina

A

is a vasospasm at rest

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9
Q

unstable pattern of angina

A

the most serious type and occurs when there is a break in an atheroma

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10
Q

causes of reduced O2 in angina?

A

tachycardia associated with hyperthyroidism or increased force of contractions due to hypertensions (stress, exercise, respiratory infection, or weather extremes)

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11
Q

signs of angina

A

usually are recurrent and is chest pain that may radiate to the neck or left arm, pallor, excessive sweating, or nausea

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12
Q

diaphoresis

A

excessive sweating

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13
Q

treatment for angina

A

vasodilators such as nitroglycerin

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14
Q

myocardial infarction

A

a heart attack that occurs as a result of the death of myocardial tissue to due ischemia

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15
Q

risk for those who have had a recent MI?

A

reoccuring MI or stroke

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16
Q

two classifications of MI’s

A

ST-elevation and non-ST elevaton

17
Q

5 subtypes of MI’s

A

type 1 is associated with an atherosclerosis; type 2 is a mismatch in myocardial O2 supply; type 3 is fatal; and type 4 and 5 are the result of medical procedures such as angioplasty

18
Q

three ways an infarction may occur in the coronary arteries

A

the thrombus obstructs the artery, vasospasm may occur, leading to total obstruction, or part of the thrombus breaks away and blocks a vessel

19
Q

what are most MI’s?

A

transmural meaning that are three heart layers are involved, as well most involve the left ventricle

20
Q

what happens when obstruction of coronary vessels occurs?

A

the heart tissue may become necrotic, leading to inflammation, ischemia, and enzymes released

21
Q

what are myocardial fibres replaced with?

A

fibrous, nonfunctional tissues

22
Q

warning signs of a MI

A

pressure, heaviness, or burning in the chest that radiates to the left arm or neck, shortness of breath, pallor, sweating, weakness, fatigue, low-grade fever, nausea, hypotension, anxiety and fear

23
Q

what can help prevent damage in someone who has had a MI?

A

thrombolytic therapy; if administered in 20 minutes, permanent damage is not sustained

24
Q

what enzymes are released during an MI?

A

aspartate aminotransferase and creatine phosphokinase

25
Q

serum levels for a MI

A

will have high enzyme levels, elevated troponin and myosin, abnormal sodium and potassium, high WBCs, and high erythrocyte sedimentation rate

26
Q

why do MIs cause death?

A

arrhythmias and fibrillation, impaired conduction, and spontaneous impulses at an ectopic site

27
Q

how does cocaine cause MIs?

A

it can interfere with cardiac conduction and cause vasospam

28
Q

less severe complications of a MI?

A

necrosis of heart tissue or thromboembolism

29
Q

treatment for a MI?

A

O2 therapy, analgesics for pain, anticoagulants (aspirin or warfarin), medication to reduce dysrhythmias, a pacemaker, bypass surgery, or other drugs