HLTH 2501: congenital heart defects Flashcards

1
Q

cardiac anomalies

A

structural defects in the heart that develop during the first 8 weeks of embryonic life

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

examples of cardiac anomalies

A

altered or missing valve, septal defects, shunts or abnormalities or the large vessels, or combinations of these

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

how common are congenital heart defects

A

8/1000 infants in the US; these are the major cause of death in the first year of life

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what can most heart defects be detected by?

A

the presence of heart murmurs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what is the major concern with congenital heart defects?

A

inadequate O2 delivery to tissues

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

left-to-right shunt

A

blood from the left side of the heart is recycled to the right side and to the lungs, resulting in increased volume in the pulmonary circulation and overall decreased CO

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

right-to-left shunt

A

unoxygenated blood from the right side of the heart bypasses the lungs and enters the left side

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

acyanotic conditions

A

disorders in which systemic blood consists of oxygenated blood

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

cyanotic disorders

A

venous blood mixes with arterial blood, permitting significant amounts of unoxygenated hemoglobin in the blood to bypass the lungs and enter the systemic circulation; produces a blue colour in the skin and mucous membranes, commonly in the lips and nails

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

genetic causes of congenital heart defects

A

can be associated with chromosomal abnormalities like down syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

environmental causes of congenital heart defects

A

viral infections such as rubella, maternal alcoholism, and maternal diabetes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

compensation mechanisms for congenital heart defects

A

the heart increases it rate and force through a SNS stimulation, however, this causes the heart to become hypertrophied; this also increases O2 needs for the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what does acidosis result in?

A

increased lactic acid in the body

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what may develop with chronic hypoxia?

A

secondary polycythemia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

signs and symptoms of large congenital heart defects

A

pallor, cyanosis, tachycardia, dyspnea, infants tend to sit in a comfortable squat position, clubbed finger, intolerance for cold, and delayed growth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

tests for congenital heart defects

A

cardiomegaly observed on radiography, diagnostic imaging, cardiac catheterization, ECGs, and echnocardiograms

17
Q

treatment for congenital heart defects

A

surgical repair to close abnormal openings or to replace valves; supportive measures and drug therapy may also be used

18
Q

what is the most common congenital heart defect?

A

ventricular septal defect

19
Q

ventricular septal defect

A

is an opening in the interventricular septum, in which large openings permit a left-to-right shunt of blood (left ventricle is higher in pressure so it moves to the right ventricle)

20
Q

result of ventricular septal defect

A

CO and SV are decreased, more blood enters the pulmonary circulation, damaged pulmonary vessels, adn pulmonary hypertension

21
Q

treatment for ventricular septal defect

A

surgical repair or medications that focus on increasing the strength of contractions, keeping a regular heartbeat, or decreasing the amount of fluid in the circulation

22
Q

valvular defects

A

malformations of the valves commonly affect the semilunar valves; can be the narrowing of the valve or the valve causing blood to leak backwards

23
Q

stenosis

A

narrowing of the valve

24
Q

incompetence

A

a failure of a valve to close completely, allowing blood to leak backwards

25
Q

two classifications of valve defects

A

stenosis or incompetence

26
Q

mitral valve prolpase

A

abnormally enlarged floppy valve leaflets that ballon backwards with pressure

27
Q

what do valve defects result in?

A

decreased efficiency of the heart pump and decreased SV; the myocardium will eventually hypertrophy and fail

28
Q

treatment for valve defects

A

surgical replacement by mechanical, animal, or tissue engineered valves

29
Q

what is the risk for valve replacements?

A

thrombus may develop in the future and endocarditis infections are at risked; therefore ASA and antimicrobial drugs may be used

30
Q

tetralogy of fallot

A

includes four defects that alter pressure within the heart, thus altering blood flow

31
Q

what is the most common cyanotic congenital heart condition?

A

tetralogy of fallot

32
Q

four defects in tetralogy of fallot

A

pulmonary valve stenosis, ventricular septal defect, dextroposition of the aorta to the right, and right ventricular hypertrophy

33
Q

how does tetralogy of fallot work?

A

the pulmonary valve restricts outflow from the right ventricle, leading to right ventricular hypertrophy and high pressure in this region; this pressure leads to a right-to-left shunt of blood through the ventricular septal defect; this flow causes deoxygenated blood from the right ventricle directly into the systemic circulation and is promoted by the position of the aorta

34
Q

result of tetralogy of fallot

A

the pulmonary circulation receives a small amount of deoxygenated blood from the right ventricle and the systemic circulation receives a larger amount of mixed blood

35
Q

what is treatment for tetralogy of fallot?

A

surgery that closes the VSD, repairs the pulmonary valve, and widens the pulmonary arteries