10 - Obs - Fetal Growth, Compromise and Surveillance - Fetal Growth and Terminology Flashcards
Between 24wks – term, ?% babies die. Further 1/500 dev ? ?.
>1/20 req admission to ????. Growing evidence in utero health and growth influences ? ?
Care of fetus in preg directed to causes.
Also prominent is ????– ID and mgmt of compromised fetus is hard, limited resources and potential for over ? of preg.
1% cerebral palsy nicu later life IUGR overmedicalization
Prinicipal causes of Perinatal mortality
- unexplained
- ? delivery
- ????
- ? abnorms
- Intrapartum, incl ?
- ? abruption
preterm IUGR congen hypoxia placental
Major associations of cerebral palsy
- ?
- ????
- Infection
- ?-?
- ? abnorms
- Intrapartum ‘fetal ?’
- ? events
premat IUGR preeclampsia congen distress postnatal
Small for Dates (SFD) aka ???
-weight of fetus less than ?th centile for gestation (?kg at term). Most just constitutionally small, have grown ? and not compromised. Assessment of fetal weight better at identifying ???? if customized to expectation for ?.
SGA 10 2.7 consistently IUGR individual
IUGR
Fetuses failed to reach own growth ?. Growth in utero is ?, many end up ???, some don’t. Many stillbirths or fetuses distressed in labour are of ‘?’ weight. If fetus ? determined to be ?kg at term and delivers at term weighing 3kg = ????, may be ? dysfunction. Similarly an ill, malnourished, tall adult may weight more than a healthy short one. I.e a proportion of IUGR babies do not appear to be SFD.
potential slowed SFD normal genetically 4 IUGR placental
Fetal Distress
Acute situation eg ?, resulting in fetal damage or ? if not ?, or fetus ? urgently. Usually in ?. Most babies w CP were ?
born hypoxic.
Fetal Compromise
? situation when conditions for normal growth and ? dev are not optimal. Cause mostly: poor ? transfer through ?(dysfunction). Commonly ????
hypoxia death reversed delivered labour not
chronic neuro nutrient placenta IUGR
