BLOCK 6: AIRWAY MANAGEMENT Flashcards
how long can brain cells survive without oxygen before permanent damage occurs
6 minutes
the upper airway includes all structures above ___
the glottis
what is the glottis
space between the vocal cords
what is the first and largest anatomic structure to manipulate when managing patient’s airway
tongue
if the tongue is not managed during airway what does it tend to do
fall back into the posterior pharynx
2 things the uvula does
prevents food we eat from going up your nose and triggers gag reflex when stimulated
what is the muscular tube that extends from nose and mouth to the esophagus and trachea
pharynx
the pharynx starts and ends where
starts: nose and mouth
ends: esophagus and trachea
what composes the pharynx (3 in order from top to bottom)
nasopharynx, oropharynx, laryngopharynx
where does the lower airway start and end
starts: glottis
ends: pulmonary capillary membrane
what marks where the upper airway ends and lower airway begins
larynx
shield-shaped structure palpable on the anterior neck
thyroid cartilage
superior part of the thyroid cartilage that forms a V shape
thyroid notch
what is laryngeal prominence also known as
Adam’s apple
where is the laryngeal prominence
immediately inferior to the thyroid notch
where is the thyroid cartilage located
directly anterior to glottic opening and vocal cords
what does the cricoid cartilage form
the lowest portion of the larynx
what is special about the cricoid cartilage
only circumferential ring of the trachea
which structure is more prominent in males?
which structure is more prominent in females?
laryngeal prominence in males
the cricoid ring in females
where are cricothyrotomies performed
the cricothyroid membrane
what is the narrowest protion of the adult airway
glottis
what is the leaf-shaped cartilaginous structure
epiglottis
what closes over the trachea during swallowing
epiglottis
what 3 things must be visualized before inserting ET tube
epiglottis, glottis, and vocal cords
physical act of moving air into and out of the lungs
ventilation
process of loading oxygen molecules onto hemoglobin molecules in the bloodstream
oxygenation
exchange of oxygen and carbon dioxide in the alveoli and tissues of the body
respiration
what is the active part of ventilation?
what is the passive part of ventilation?
active: inhalation
passive: exhalation
what is external respiration also called
pulmonary respiration
what is internal respiration also called
cellular respiration
what is external respiration
exchanging oxygen and carbon dioxide between alveoli and blood in pulmonary capillaries
what is internal respiration
exchanging of oxygen and carbon dioxide between systemic circulation and cells of the body
what type of problem is an overdose on CNS depressors
ventilation problem
what type of problem is a person trapped in a place devoid of oxygen
oxygenation problem
circulation of blood within an organ or tissue in adequate amounts to meet the current needs of the cells
perfusion
what is a dangerous condition in which the tissues and cells do not receive enough oxygen
hypoxia
early signs of hypoxia (5)
restlessness, irritability, apprehension, tachycardia, anxiety
late signs of hypoxia (3)
change in mental status, weak/thready pulse, cyanosis
how to assess cyanosis in dark skinned patients
mucous membranes (lips, gums, inner eyelids, and nailbeds)
what is dyspnea
shortness of breath
how does gas exchange occur
simple diffusion
what is a failure to match ventilation and perfusion called
V/Q mismatch
what contributes to most abnormalities in oxygen and carbon dioxide exchange
V/Q mismatch
what is normal resting minute ventilation
6L/min
what is normal resting alveolar volume
4L/min
what is normal pulmonary artery blood flow
5L/min
what is the ratio of ventilation to perfusion
4:5L/min or 0.8L/min
the ventilation-to-perfusion ratio is highest where? lowest where?
highest: apex of the lung
lowest: base of the lung
what is the most common airway obstruction in an unresponsive patient and what are the two indicators
tongue - improper head/neck position and snoring respirations
what two neuromuscular disorders can affect the ability of the CNS to control breathing
muscular dystrophy and poliomyelitis
how does muscular dystrophy affect the CNS’s ability to control breathing
degeneration of muscle and its contractility as well as curvature of the spine
how does poliomyelitis affect the CNS’s ability to control breathing
affects the nerves that regulate ventilation and result in paralysis
what three conditions are bronchoconstriction associated with
allergic reactions, asthma, and COPD
intrinsic factors that cause airway obstruction (3)
infection, allergic reactions, unresponsiveness
extrinsic reactions that cause airway obstruction (2)
trauma and foreign body airway obstruction
what is respiratory splinting
purposely breathing shallow to alleviate pain caused by injury such as flail chest
hypoventilation
CO2 production exceeds body’s ability to eliminate it or CO2 elimination is depressed and can’t keep up with normal metabolism
hyperventilation
CO2 elimination exceeds carbon dioxide production
if the pH of blood is too high, what happens to the patient’s breathing and why
shallow/slow breaths in attempt to retain carbon dioxide
if the pH of blood is too low, what happens to the patient’s breathing and why
hyperventilation to expel more carbon dioxide
what two examples can cause hyperventilation/pH acidity
hyperglycemic ketoacidosis and aspirin OD
decrease in minute volume leads to ____
hypercapnia
increase in minute volume leads to ____
hypocapnia
what is hypercapnia
buildup of CO2 in the blood
what is hypocapnia
decrease of CO2 in the blood
how does hypoventilation affect minute volume
goes down
how does hypoventilation affect CO2 elimination
goes down
how does hypoventilation affect CO2 levels
goes up (hypercapnia)
how does hyperventilation affect minute volume
goes up
how does hyperventilation affect CO2 elimination
goes up
how does hyperventilation affect CO2 levels
goes down (hypocapnia)
what two things have key roles in the process of respiration
atmospheric pressure and partial pressure of oxygen
what happens to percentage of oxygen, partial pressure, and total atmospheric pressure at higher altitudes
oxygen - stays the same
partial pressure - decreases
total atmospheric pressure - decreases
what is PaO2
partial pressure of oxygen
what makes it difficult to breathe at high altitudes
low PaO2
carbon monoxide has a ___x higher affinity for hemoglobin than oxygen
250
what is intrapulmonary shunting
blood entering the lungs from the right side of the heart bypasses the alveoli and returns to the left side of the heart in an unoxygenated state
what does fluid accumulation in the alveoli lead to
anaerobic respiration and increase in lactic acid accumulation
what is anemia
deficiency of red blood cells
hemorrhagic vs vasodilatory shock
hemorrhagic: decrease in blood volume caused by internal/external bleeding
vasodilatory: increase in size of blood vessels causing decrease in BP and blood flow
what is the fastest way the body can eliminate excess H+ ions
creating water and carbon dioxide
four main clinical presentations of acid-base disorders
respiratory acidosis and alkalosis
metabolic acidosis and alkalosis
side effect of positive pressure ventilation in patients
decreased cardiac output and preload, increased afterload, and hypotension
what draws air into the chest cavity
negative-pressure ventilation
normal adult resp rate
12-20
normal child resp rate
12-37
normal infant (1month-1year) resp rate
30-53
what is a clear indicator of a depressed or absent gag reflex
pooling of secretions in patient’s mouth
what is the gag reflex
spastic pharyngeal and esophageal reflex triggered by stimulating the uvula or posterior pharynx
low oxygen level in arterial blood
hypoxemia
deficiency of oxygen at the tissue and cellular levels
hypoxia
lack of oxygen that results in tissue and cellular death
anoxia
difference between hypoxemia and hypoxia treatment
hypoxemia can be reverse by administering supplemental oxygen and hypoxia require more aggressive oxygenation and ventilatory support
what is adventitious breath sounds
abnormal
what is the upright sniffing position
patient is sitting up with head moved forward until the earlobes are on the same vertical plane as the manubrium of the sternum
what is the tripod position
patient is sitting up and leaning forward with elbows bent
proper airway management order (4)
opening airway
clearing airway
assessing breathing
providing appropriate interventions
what is orthopnea
positional dyspnea
what is breathing retractions
skin pulling between and around the ribs during inhalation
what muscles are used during accessory muscle use breathing
sternocleidomastoid muscles, pectoralis major muscles, abdominal muscles
what are staccato speech patterns
one or two word dyspnea
what is paradoxical motion and what does it indicate
opposite of normal chest movements (inward movement of chest segment during inhalation and outward movement of chest segment during exhalation) indicating flail chest
what is pulsus paradoxus and what does it indicate
drop in systolic BP of more than 10, change in pulse quality, or disappearing of pulse during inhalation indicating decompensating COPD, pericardial tamponade, or other increase in intrathoracic pressure
what is sneezing caused by
irritation of the nasal cavity
what is hiccupping
sudden inhalation caused by spasmodic contraction of the diaphragm cut short by closure of the glottis
what creates breath sounds
air moving through the tracheobronchial tree
what type of respiratory pattern is gradual increasing rate and depth of respirations followed by gradual decrease of respirations with intermittent periods of apnea and what is it associated with
Cheyne-Stokes respirations
brainstem injury
what type of respiratory pattern is deep, rapid respirations and what is it associated with
Kussmaul respirations
diabetic ketoacidosis
what type of respiratory pattern is irregular pattern, rate, and depth of breathing with intermittent periods of apnea and what is it associated with
Biot (ataxic) respirations
intracranial pressure
what type of respiratory pattern is prolonged, gasping inhalation followed by extremely short, ineffective exhalation and what is it associated with
Apneustic respirations
increased intracranial pressure
what type of respiratory pattern is slow, shallow, irregular or occasional gasping breaths and what is it associated with
agonal gasps
cerebral anoxia (shortly after heart has stopped but brain is still sending signals to the muscles)
what type of respiratory pattern is tachypneic hyperpnea (rapid, deep respirations) and what is it associated with
central neurogenic hyperventilation
increased intracranial pressure or direct brain injury
what is the inspiratory/expiratory ratio
1:2 (expiration time is twice as long as inspiration)
the I/E in patients with lower airway obstruction or asthma may be what ratio
1:4 or 1:5
what is the I/E ratio in patients with tachypnea
1:1
wheezing is what pitch and represents what
continuous high-pitched
constricted lower airway (asthma)
rhonchi is what pitch and represents what
continuous low-pitched
mucus or fluid in larger lower airways (pulmonary edema)
what is crackles and represents what
also known as rales
discontinuous
airflow causes mucus or fluid into the smaller lower airways
what is stridor and represents what
high-pitched during inspiration
foreign body aspiration, infection, swelling or trauma immediately above the glottic opening
what causes pleural friction rub
inflammation causing pleurae to thicken, allowing visceral and parietal pleurae to rub together
what does a pulse oximeter measure
percentage of saturated hemoglobin in arterial blood and patient’s pulse
when auscultating breath sounds emergently, where do you auscultate
bilaterally at the third or fourth intercostal space in the midaxillary line
normal oxygenated patient should have what SpO2 level
greater than 95%
less than 95% SpO2 in nonsmoker suggests what
hypoxemia
SpO2 less than 90% accompanied with respiratory distress signals a need for what
aggressive oxygen therapy
hemoglobin loaded with oxygen
oxygemoglobin
hemoglobin from which oxygen has been released to the cells
reduced hemoglobin
compound formed by oxidation of iron on the hemoglobin
methemoglobin
hemoglobin loaded with carbon monoxide
carboxyhemoglobin
peak expiratory flow rate in healthy adults
350-750mL
what is used to evaluate bronchoconstriction and with what tool
peak rate of forceful exhalation with peak expiratory flowmeter
what ABG measurements are used to evaluate patient’s acid-base status and what is the normal range of both
pH and Hco3-
pH: 7.35-7.45
Hco3-: 21-28mEq/L
what ABG measurement is used to evaluate patient’s effectiveness of ventilation and what is the normal range
PaCO2
35-45mm Hg
what ABG measurements are used to evaluate patient’s oxygenation and what is the normal range of both
PaO2 and SaO2
PaO2: 80-100mm Hg
SaO2: above 95%
what is the “smoke of metabolism”
carbon dioxide
how many ATP does aerobic metabolism create from each glucose molecule
38
how many ATP does anaerobic metabolism create from each glucose molecule
2
what is the recommended method of monitoring placement of advanced airway device
capnography
what reading approximates the arterial PaCO2 level and by how close
ETCO2 level is usually 2-5mmHg lower than arterial PaCO2 levels
what happens to CO2 levels after ROSC
abrupt and sustained increase
colorimetric carbon dioxide detector function
treated paper turns from purple to yellow during exhalation signifying presence of carbon dioxide
Phase 1 (A-B) of capnography waveform
respiratory baseline
initial stage of exhalation (dead space gas free of CO2)
Phase II (B-C)
expiratory upslope
alveolar gas mixes with dead space gas
Phase III (C-D)
alveolar plateau
all alveolar ga, max CO2 level
Phase IV (D-E)
inspiratory downstroke
displaces CO2 causing waveform to return to base level
what happens to waveform during hypoventilation
high waveforms, prolonged alveolar plateau, and longer-than-normal intervals between waveforms
what happens to waveform during hyperventilation
small waveforms, short alveolar plateau, shorter-than-normal intervals between waveforms
what is “shark fin” waveform
caused by bronchospasms
gradual upsloping phase II (B-C)
how is rebreathing CO2 shown on waveform
waveforms elevate and never return to the baseline at the end of the inspiratory downstroke phase IV (D-E)
cause of too high CO2 in apneic with a pulse patient
positive-pressure ventilation is too slow
cause of too low CO2 in apneic with a pulse patient
positive-pressure ventilation is too fast
cause of too high CO2 in apneic and pulseless patient
positive-pressure ventilation is too slow or could indicate ROSC
cause of too low CO2 in apneic and pulseless patient
misplaced ET tube, prolonged arrest, inadequate chest compressions, positive-pressure ventilation is too fast