EXAM 2: Motor System 1 Flashcards

1
Q

feedforward

A

the anticipatory use of sensory information to prepare for movement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

feedback

A

the use of sensory information during or after movement to make corrections either to ongoing movement or to future movements

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Automatic movements require continuous integration of:

A
  • visual
  • somatosensory
  • vestibular info
  • motor processing
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

In the absence of vision, reaching depends on this to locate objects

A
  • somatosensation

- proprioception

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What disrupts positioning of limbs in individuals with complete deafferentation?

A

Loss of somatosensation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What does neural activity begin with?

A

with a decision made in the anterior part of the frontal lobe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

follow the path of neural activity

A
  • frontal lobe
  • motor planning areas activated
  • control circuits activated (in UMN tracts)
  • UMN tracts deliver signals to interneurons and LMNs
  • LMNs transmit signals to skeletal muscles
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are control circuits?

A

consist of cerebellum and basal ganglia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What do control circuits do?

A
  • regulate activity in UMN tracts

- activation results in excitation or inhibition of motor neurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

UMN tracts deliver signals to

A
  • interneurons

- LMNs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

classification of UMN tracts

A
  • postural/gross movement tracts
  • fine movement tracts
  • nonspecific UMNs
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

postural/gross movement tracts control:

A

automatic skeletal muscle activity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

fine movement tracts control:

A

fractionated movements of limbs and face

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

nonspecific UMNs control:

A

all motor neurons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What do LMNs do?

A
  • transmit signals directly to skeletal muscles

- elicit contraction of muscle fibers that move the upper limbs and fingers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How is voluntary movement controlled?

A

top down

  • brain
  • spinal cord
  • muscle
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

properties of skeletal muscle

A
  • excitable
  • contractile
  • extensile
  • elastic
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

t-tubules

A
  • transverse tubules

- projections of muscle cell membranes that extend into the muscle

19
Q

sarcoplasmic reticulum

A

series of storage sacs for Ca2+ ions

20
Q

myofibrils

A

individual muscle fibers

21
Q

sarcomeres

A

arrangement of proteins in a myofibril

22
Q

two protein types in sarcomeres

A
  • structural

- contractile

23
Q

structural proteins of sarcomeres

A
  • Z line
  • M line
  • titin
24
Q

M line

A

anchors fibers in center of sarcomere

25
titin
- connects Z line with m line - maintains position of myosin relative to actin - prevents sarcomere from being pulled apart
26
contractile proteins of sarcomeres
- myosin - actin - tropomyosin - troponin
27
contraction
- Produced when active slides relative to myosin | - Repeated attachment, swiveling, and detachment of myosin heads produce contraction of the muscle
28
What do muscles behave like?
springs
29
______ springs generate more resistance to stretch than the same spring when it is _______
- stretched | - shortened
30
What determines total resistance to muscle stretch?
- active contraction - titin - weak actin-myosin bonds
31
muscle tone
resistance to stretch in a resting muscle
32
How is muscle tone assessed?
assessed clinically using PROM
33
When tone is normal, resistance to passive stretch is
minimal
34
Normal resting muscle tone provided by
- titin | - weak actin-myosin bonds
35
weak actin-myosin bonds
- attached, but myosin heads don't swivel | - no muscle contraction, but there is resistance
36
What happens to actin-myosin bonds if muscle is immobile for a prolonged period?
- bonds continually form | - broken by stretching the muscle
37
stretch velocity and actin-myosin bonds
The faster the stretch, the greater the resistance
38
What happens when healthy innervated muscle is continuously immobilized in a shortened position for a prolonged period?
sarcomeres disappear from the ends of myofibrils
39
Why do sarcomeres get lost when immobilized in a shortened position?
Loss of sarcomeres is a structural adaptation to the shortened position so the muscle generates optimal force at the new resting length
40
What happens when a structurally shortened muscle is stretched?
it quickly reaches the limits of its elasticity and is resistant to stretching
41
What happens if a muscle is immobilized in a lengthened position?
the muscle adds new sarcomeres
42
cocontraction
simultaneous contraction of antagonist muscles
43
What does cocontraction do?
- stabilizes joints - UE: enables precise movements - LE: allows individual to stand on an unstable surface