2.21 Motor System 4 Flashcards

1
Q

tools used to measure hypotonia

A
  • EMG recordings

- Ashworth scale

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2
Q

benefit to using EMG to measure hypotonia

A

to determine which factors are contributing to movement impairment

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3
Q

factors that may contribute to movement impairment

A
  • contracture
  • hyperreflexia
  • cocontraction
  • inappropriate timing of muscle activity
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4
Q

What does the Ashworth scale provide?

A

a subjective clinical assessment of resistance to passive stretch

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5
Q

UMN lesion types

A
  • spastic CP
  • stroke, middle cerebral artery
  • SCI
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6
Q

common characteristics of UMN lesions

A
  • paresis
  • abn timing of muscle activity
  • Babinski’s sign
  • myoplasticity
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7
Q

In spasticity, what factors lead to movement dysfunction?

A
  • abnormal supraspinal influences
  • failure of normal neuronal selection
  • consequent aberrant muscle development
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8
Q

motor disorders in spastic CP include:

A
  • problems with coordination
  • abnormal tonic stretch reflexes both at rest and during movement
  • reflex irradition
  • lack of postural preparation before movement
  • abnormal cocontraction of muscles
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9
Q

What does stroke most commonly affect?

A

middle cerebral artery

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10
Q

two important things affected after stroke

A
  • myoplasticity

- voluntary movement

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11
Q

movement disorders after middle cerebral artery stroke are consequences of

A
  • paresis
  • decreased fractionation of movement
  • myoplasticity
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12
Q

hyperreflexia and stroke

A

rarely does hyperreflexia contribute significantly to movement limitations

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13
Q

What tract provides voluntary movement of the paretic limbs following stroke?

A

reticulospinal tract

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14
Q

complete SCI

A

all descending neuronal control is lost below the level of the lesion

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15
Q

incomplete SCI

A

function of some ascending and/or descending fibers is preserved within the spinal cord

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16
Q

After SCI, what produces excessive resistance to muscle stretch?

A
  • excessive stretch reflexes
  • muscle contracture
  • increased cross-bridge binding
17
Q

What limits movement in people with SCI?

A
  • hyperreflexia

- contracture

18
Q

With CP, what is the primary impairment interfering with balance recovery?

A

paresis of agonist postural muscles

19
Q

effect on spasticity of LE strengthening in children with spastic CP

A

has no effect on spasticity

20
Q

spasticity and LE dysfunction in children with spastic CP

A

spasticity is not a significant contributor to LE dysfunction in children with spastic CP

21
Q

improved movement in people after stroke has been demonstrated with:

A
  • hand and finger movements against resistance
  • robotic therapy for UE
  • constraint-induced movement
  • botox injections as an adjunct to therapy
  • cycling
  • task-oriented gait training
  • gait training using treadmill
22
Q

activity based therapy

A

rehab that activates the NM system below the level of the lesion in SCI pts

23
Q

treadmill training with BW support in pts with SCI vs overground walking

NM activation

A

somatosensory input to spinal cord during treadmill training with BW support elicits neuromuscular activation that does not occur during overground walking

24
Q

FES

A

functional electrical stimulation

25
Q

FES for SCI pts

A
  • provided via implanted or skin surface electrodes
  • If FES promotes recovery of NM function, FES can be withdrawn and walking will continue to be possible
  • If continued, FES is required for walking and acts as a neuroprosthesis
26
Q

causes of spasticity

A
  • hyperreflexia

- brainstem UMN overactivity

27
Q

What do medications do for spasticity?

A

interfere with UMN mechanisms

28
Q

What are the common meds used for spasticity?

A
  • baclofen
  • tizanadine
  • dantrolene