3.14 Ch. 12.3 Flashcards

1
Q

What does MG damage?

A

ACh receptors at the NMJ

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2
Q

What happens during MG?

A

repeated use of muscle leads to increasing weakness

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3
Q

What does botulism interfere with?

A

release of ACh from motor axon

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4
Q

What happens with botulism?

A
  • produces acute, progressive weakness
  • loss of stretch reflexes
  • sensation remains intact
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5
Q

myopathy

A

disorder intrinsic to muscles

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6
Q

myopathy: sensation and autonomic function

A
  • sensation intact
  • autonomics intact

*because nervous system is not affected by myopathy

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7
Q

Later stages of myopaty

A

Coordination, muscle tone, and reflexes are unaffected until muscle atrophy becomes so severe that muscle activity cannot be elicited.

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8
Q

What are the benefits of using electrodiagnostic studies?

A

reveals pathologic location and are often diagnostic

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9
Q

nerve conduction studies can be used to differentiate among these:

A
  • primarily myelinopathy and axonopathy processes
  • UMN and LMN paresis
  • mononeuropathy and polyneuropathy
  • local conduction block and Wallerian degeneration
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10
Q

3 clinical signs of peripheral neuropathy are revealed in:

A
  • ≥ 50 yo
  • presence of 2-3 s/s

*correlate with highly electrodiagnostic evidence of peripheral neuropathy

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11
Q

3 clinical signs of peripheral neuropathy

A
  • Absence of ankle jerk reflex despite facilitation
  • Impaired vibration
  • Impaired position sense of the great toe
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12
Q

evaluation of polyneuropathy: What increases along with severity of neuropathy?

A

reports of pins and needles sensations

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13
Q

evaluation of polyneuropathy: signs of damage result from

A

hypoactivity or hyperactivity of neurons

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14
Q

Important thing when evaluating for polyneuropathy

A

distinction between PNS and CNS dysfunction is vital**

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15
Q

What guides treatment decisions for polyneuropathy?

A
  • sensory
  • MMT
  • EMG/NCV
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16
Q

Why is education so important with polyneuropathy?

A

prevent complications from the damage caused by

  • lack of sensation
  • disuse
  • overuse
17
Q

treatment for peripheral polyneuropathy (education)

A
  • visually inspect areas daily for wounds

- proper foot care

18
Q

treatment: interventions for edema

A
  • elevation of limb
  • compression bandaging
  • e-stim
19
Q

treatment of polyneuropathy: contractures prevented by

A
  • prolonged stretching

- daily activities

20
Q

endurance exercise after peripheral nerve crush injury

A

enhances sensory and motor recovery

21
Q

resistance exercise after peripheral nerve crush injury

A

may delay functional recovery (or combo of resistance and endurance)

22
Q

treatment for polyneuropathy: exercise

A

emphasize gradual strengthening and functional use of individual muscles and groups

23
Q

orthoses for the treatment of polyneuropathies are used to

A

stabilize WB joints

24
Q

orthoses prevent:

A
  • sprains and strains
  • drooping of forefoot during gait
  • deformities from paresis, paralysis, and lack of sensation
25
Q

drooping of forefoot during gait caused by

A

paresis or paralysis of tibialis anterior