72. Small ruminant medicine: prolapse of the uterus and vagina Flashcards

1
Q

Vaginal prolapse incidence?

A

Vaginal prolapse

Incidence

• 1% (0–15%) 8 cm to 20 cm vaginal wall urinary bladder, and/or uterine horn(s) or both of these

during the last month of gestation

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2
Q

Vaginal Prolapse aetiology?

A

Aetiology

  • excess body condition
  • multigravida uterus
  • high-fibre diets, particularly root crops
  • lameness
  • steep fields
  • subclinical hypocalcaemia
  • short-docked tails
  • abdominal straining
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3
Q

Consequences of vaginal prolapse?

A

Consequences
• prolonged straining →
o premature onset of labour or abortion
o pregnancy toxaemia
o Rupture of the dorsal vaginal wall and evisceration

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4
Q

Diagnosis of vaginal prolapse?

A

Diagnosis

  • Difficult (extra care is needed)
  • Undocked tails
  • Long fleeces
  • Extensively managed systems
  • Good lighting is essential to examine housed sheep
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5
Q

Clinical signs of vaginal prolapse?

A

Clinical signs (first stage of labour):

  • isolation from the remainder of the flock
  • Failure to come forward for concentrate feeding
  • Lateral recumbency with repeated
  • Forceful abdominal contractions and associated vocalization
  • increased respiratory rate
  • frequent urination without urine
  • neither cervical mucus plug nor foetal membranes are visible
  • contamination with faeces, bedding material and soil
  • vaginal wall quickly becomes oedematous and turgid
  • rupture during manual replacement
  • effective caudal analgesia
  • increase toxin uptake
  • fibrin exudation on the surface
  • particular attention should be paid to the cervix
  • haemorrhage

o externally from tears in the vaginal wall

o pale mucous membranes

o significant internal bleeding

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6
Q

Viability of foetuses?

A

Viability of the foetus(es)

  • Transabdominal real-time B-mode with a 5 MHz sector transducer
  • Foetal heartbeat, difficult to detect in near-term foetuses even after searching for 5 minutes
  • US findings only indicate the presence of one or more live foetuses at the time of examination
  • If no foetal movement is detected after 5 minutes → the foetuses are dead
  • foetal membranes in the cervix → indicates impending abortion/parturition →immediate action
  • caesarean operation

o foetal membranes appear normal

o hopeless when there is a foetid vaginal discharge.

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7
Q

US urinary bladder and vaginal prolapse?

A

The vaginal prolapse

• Urinary bladder, uterine horn(s) or both

Urinary bladder

  • An anechoic (black) area
  • Usually greater than 10 cm in diameter
  • Compressed dorsoventrally
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8
Q

Treatment of vaginal prolapse?

A

Treatment

• Standing ewe + caudal block (hindlimb paresis) + prolapse is raised relative to the vulva→ emptying

of the bladder

  • Puncture ↔ allanto-chorion and/or vessels
  • Closing the vaginal gap:

o Buhner suture

o Single interrupted and mattress sutures ?

o Hindlimb paresis → well-bedded pens for 36 hours

o Permanent prolapses → culling

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9
Q

Closing the vaginal prolapse, complications?

A

Closing the vaginal gap

• Harnesses

o Difficult to fit

o Faecal contamination

o First stage of labour!

• Plastic retention devices

o Tied to the fleece of the flanks

o In early cases but can cause irritation

To

  • No more tenesmus → Buhner suture be untied 3–4 days later
  • First stage of labour

Complications

  • Abortus within 24-48 hours, foeto-placental unit
  • Incomplete cervical dilation
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10
Q

Uterine prolapse incidence and cause?

A

Uterine prolapse

Incidence:• Immediately after lambing or within 12–48 hours

Cause:

  • Large singleton foetus + prolonged 2. stage of parturition
  • Unskilled assistance → pain, abdominal straining → dystocia
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11
Q

Treatment and management of uterine prolapse?

A

Treatment:

  • Epidural anaesthesia
  • Reposition
  • Buhner method
  • After reposition restraining → invagination?
  • AB, NSAID
  • Probability of re-prolapse → breeding

Management:

• Timely correction of the dystocia by a veterinary surgeon

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