14. Hardware disease (traumatic reticuloperitonitis): clinical signs, local and/or systemic consequences, diagnosis, and treatment Flashcards

1
Q

Clinical signs?

A

Clinical (acute) signs in initial penetration

• General appearance:

o Sudden onset, sharp fall in milk yield

• Sound: grunt

o Vibrations can be felt over larynx/trachea with hand/stethoscope

o Everything more visible when in the acute phase than in chronic

o Real problem starts when it stops screaming (enters chronic stage) and there are adhesions—> can’t

go back to the healthy stage anymore!

• Basic clinical values

o T: 39,5-40,0 C (acute, mild fever)

o P: 80 - 100/min

o R: >30/min, rapid, shallow, costal (won’t do the normal abdominal breathing because it’s painful)

• Digestive signs: not pathognomonic signs

o appetite, belching, rumination! (–)

o rumen motility! (–)

o rumen: small, firm, separated content, +/- mild tympany

o reticulum: No reticular sounds!

o faeces: decreased, dry, poorly digested

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2
Q

Clinical signs in chronic cases?

A

Clinical signs in chronic cases

  • If animals enter chronic stage, some of the acute stage signs disappear!
  • Feed intake and faecal output ↓
  • Milk production remains low
  • Abdominal pain become less apparent
  • T: usually returns to normal
  • Some cattle develop vagal indigestion syndrome (Hoflund syndrome) (particularly on the ventromedial

reticulum)

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3
Q

If chronic case furthermore leads to complications such as Pleuritis/pericarditis?

A

If chronic case furthermore leads to complications such as pleuritis/pericarditis

  • If the perforating object is min 8-10 cm long!
  • Depressed (very painful)
  • H: (>90 bpm) (tachycardic x2 to as much they normally have)
  • T: >40°C
  • R: fast, shallow respiration
  • Washing machine murmur: Fluid accumulate in pericardium and gas production

due to bacterial infection. = splashing/pericardial friction rubs

  • Jugular vein distention without it being raised and congestive heart failure
  • Submandibular and brisket oedema (same as in brachycephalic dogs)
  • Prognosis is poor with these complications (once heart is affected at least!)
  • Penetration of pericardium into the myocardium: sudden death due to acute cardiac arrest!
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4
Q

Prognostics for traumatic reticuloperitonitis?

A

Prognostics for traumatic reticuloperitonitis

• History and clinical findings

o Surroundings/environment (construction=screws/nails around)

o abnormal general behaviour

o spontaneous grunting and groan

o sporadic onset

o sudden digestive disorder without changes in feeding

o fever: signs of (acute) inflammation

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5
Q

Methods for diagnosis?

A

Methods for Diagnosis

• Clinical examinations

o To elicit a grunt or groan

§ Reticular pain probes

§ Temporarily prevention of breathing

o Sensitivity of the wither’s region

§ Kalchsmidt

  • Haematology
  • Rumen fluid
  • Ferroscopy
  • Abdominocentesis
  • Laparotomy
  • Ultrasonography
  • Radiography
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6
Q

Reticular pain probes: Back grip?

A

Reticular pain probes: back grip

  • = To elicit a grunt/groan
  • A fold of skin is pulled up with both hands, which forces the back to sink
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7
Q

Reticular pain probes: Pain percussion?

A

Reticular pain probes: pain percussion

• Heavy rubber hammer toward the area of the reticulum (A) for percussion of the region (B)

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8
Q

Reticular pain probes: Pole test?

A

Reticular pain probes: pole test

  • 2 assistants hold the pole in place (A) and then pull the pole upwards against the cow’s abdomen (B)
  • Repeat many times: start just in front of the udder and move a bit cranially every time. (Calibration area

needed to know the difference)

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9
Q

Knee elbow probe?

A

Knee elbow probe

• Crouching position of veterinarian, push your elbow on your knee and lift hand up towards the reticular

region

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10
Q

Temporarily prevention of breathing?

A

Temporarily prevention of breathing

• Rectal sleeve placed over the mouth and nose

o Apnoea

o Strong diaphragmatic contractions

o Pain

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11
Q

Sensitivity of the withers region?

A

Sensitivity of the withers region (Kalchschmidt)

• Touch, displace or pull the skin, or pull the hairs gently

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12
Q

Haematology?

A

Haematology

  • Haematocrit ↑
  • Leucocytosis (neutrophilia)
  • Acute cases: neutrophilia

o Differential leukocyte count is a better diagnostic indicator for peritonitis (only in the first three

days) than the total leukocyte counts commonly with a left shift.

o Chronic cases: only moderate leucocytosis, neutrophilia and monocytosis

  • Fibrinogen ↑
  • Total protein↑
  • Glutaraldehyde coagulation test (GCT) test is used to estimate the level of immunoglobulins and fibrinogen
  • Gel formation: sensitivity (87.9 - 97.8%), coagulation time of 3 min and for 6 min
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13
Q

Rumen fluid and ferroscopy?

A

Rumen fluid

• Not so informative/pathognomic for TRP

Ferroscopy

  • Metal detector
  • Scanning on ventral and lateral thoracic and abdominal wall to find ferromagnetic foreign bodies!
  • False negative result

o Dorsal penetration =too far away, aluminium/cupper (non-magnetic) material

• False positive

o Non-perforating ferromagnetic foreign bodies (bolt nuts) and magnets placed by farmers.

• Compass

o Identification of magnets that have been given previously (not nowadays)

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14
Q

Abdominocentesis?

A

Abdominocentesis

  • Use hypodermic/spinal needle with stylet under US guidance
  • Find the place first: general peritonitis has a special place and then do centesis
  • Parameters of fluid: amount, colour, transparency, odour, consistency
  • Refractometer for specific gravity
  • Cytology and bacteriological examination:
  • Exudate in 99% of the cases!
  • Cloudy, heterogenous
  • Echogenic sediment of bacteria/inflammatory cells/fibrin
  • Watery-viscous
  • Smelly
  • Fibrin flecks
  • Spec gravity >1.015
  • Protein content high; > 30 g/
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15
Q

Laparoscopy?

A

Laparoscopy

  • = opening of abdominal cavity: sometimes good enough without rumenotomy.
  • Useful for detection of inflammatory changes
  • Rumenotomy can be used but before removing foreign bodies examine the area involved, the degree and

direction of penetration.

è proper prognosis: from the previous lecture

o Simple ventral penetration: favourable

o Deep anterior penetration: unfavourable (pericarditis)

o Cranio-medial penetration: questionable (Vagus indigestion)

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16
Q

Ultrasonography?

A

Ultrasonography

  • 3.5 to 5.0 MHz linear or convex transducer
  • ventral aspect of the thorax on both sides of the sternum and both sides of the lateral thorax up to the level

of the elbow

• Reticular motility: left ventral thoracic region and for 3 minutes without moving the transducer

17
Q

Treatment Options?

A

Treatment options

  • Prevention= best option!
  • = Reticular magnets
  • They can’t collect the penetrating foreign bodies, only the loose ones.
  • Round magnet collects the FB longitudinally
  • Will be there for life
18
Q

Treatment?

A

Treatment

• Conservative therapy

o Antibiotics (penicillin, oxytetracycline, ceftiofur)

o Diet

o Reticular magnet

o Changing of housing: elevation of the cranial body/immobilization to minimize the abdominal

pressure!

• Surgery

o Laparorumenotomy: Localize and detect an abscess/foreign body; not as useful as the conservative

therapy.