Respiratory Tract Disease in Small Animals Flashcards

1
Q

What would you expect to see clinically if a animal had lung disease?

A

•(not as obvious in SA can be seen in horses with COPD)- see the abdomen lift as they force air out of their lungs. Lung disease – EXPIRATORY dyspnoea

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2
Q

What will you see clinically in an animal with URT obstruction?

A

INSPIRATORY dyspnoea (bulldogs and pugs – will often have a noise too). Inspiratory phase will be longer

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3
Q

Is inspiratory or expiratory phase longer in pleural effusion?

A

Phases are equal

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4
Q

Should you anaesthetise a patient with pleural effusion?

A
  • Actually can be better off by anaesthetising them – but you HAVE to know what you are going to do next
  • When anaesthetised they are breathing 100% Oxygen and you have control of airway
  • Use the drugs you are used to e.g. propofol and iso. If you change to something you aren’t used to – you don’t know if something happens whether it is normal for the drugs or if there is an issue with the animal
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5
Q

Is pleural space disease or lung disease easier to manage and why?

A

•Pleural space disease in an acute situation – easier to manage than a diffuse lung disease. As you can just drain it.

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6
Q

What should you do if you find difuse lung disease on radiograph?

A

Take samples! Do this at the same time as radiographs/CT if you find lung disease as you will need to then know WHAT is causing the lung disease.

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7
Q

What pre med is good in a cardiac problem?

A

•Opiod +/- BZD

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8
Q

Why are opioids good to use in a CRS case?

A

•Opioids – Patients often have high resp rate which can cause further issues but opioids reduce resp rate and can help them relax

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9
Q

What 3 things are useful for nasal disease diagnosis?

A
  • History and clinical signs (helpful)
  • Radiography

–CT

•Diagnostic sampling – rhinoscopy or nasal flush

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10
Q

What is a common cause of nasal disease in cats?

A

Infections

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11
Q

What is important for the diagnosis of a coughing case? (5)

A
  • Acute and chronic
  • History and physical exam(!!!!)

–+/- resp difficulty? Suggests airway and lung involvement

•Radiography

–CT?

  • Endoscopy
  • Diagnostic sampling – Blind BAL or TAL is perfectly viable to help get a definitive diagnosis
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12
Q

What are the major causes of dyspnoea? (4)

A

1 airway obstruction (URT or lung disease)

2 reduced airway capacity (pleural space disease?)

3 pulmonary parenchymal disease

4 other – metabolic/physiologic causes (tend to be more rapid shallow breathing rather than difficulty)

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13
Q

What are the common causes of Airway obstruction (often +/- cough/cyanosis/noise)? (13)

A

–Trauma/haemorrhage etc

–Laryngeal paralysis/trauma/granuloma

–Brachycephalic airway obstruction- long soft palate, stenotic nares, larynx collapse etc

–Tracheal or bronchial collapse

–Extra-luminal mass lesions - thyroid, abscess, lymphoma, large heart

–Asthma/bronchospasm (cat)

–Nasopharyngeal polyp (cat)

–Nasal cavity (+/- sneezing, +/- nasal discharge) - rhinitis/F.B./neop/polyp/trauma

–F.B.

–Neoplasia

–Oslerus infestation - “lungworm”

–Brochiectasis

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14
Q

What are the common causes of Loss of thoracic capacity (+/- cyanosis)? (6)

A

–Pleural effusion – e.g. pyothorax, haemothorax

–Pneumothorax

–Neoplasia - pleural or mediastinal

  • Most tumours in the pleural space will result in pleural effusion
  • Look at the structures – if there is a change in the positions there has to be a mass

–Ruptured diaphragm

–Abdominal abnormality causing diaphragmatic compression – severe ascites, abdominal mass

–Gross cardiomegaly

•Heart gets so big that it compromises cardiac function

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15
Q

What are the common causes of Pulmonary parenchymal disease (+/- cyanosis/cough)? (10)

A

–Pulmonary oedema - L. heart failure typically

–Eosinophilic disease - “P.I.E or EBPn“

•Can be quite diffuse and cause respiratory difficulty

–Pulmonary fibrosis

–Pulmonary thromboembolism

•Acute onset, moderate difficulty and the best x ray is unremarkable. Consider this!!!! Clot in aorta preventing blood to lung. V/Q mismatch.

–Non-cardiogenic pulmonary oedema

•Following trauma or near drowning

–Neoplasia - primary or (more likely) secondary

–Pulmonary haemorrhage

–Bronchopneumonia

–Paraquat poisoning

–Angiostrongylus/Aelurostrongylus

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16
Q

What can commonly cause Others - physiologic/metabolic (often open-mouthed/panting/ shallow)? (9)

A

–Hyperthermia/heat stroke/fever

–Obesity

–Excitement/fear/stress/pain/shock

–Parturition/false pregnancy/eclampsia

–Anaemia/abnormal haemoglobin/hypoxic toxins

–Metabolic disease – acid base disturbance

•Ketoacidossis = exaggerated respiratory pattern

–CNS disease

–Endocrinopathy eg hyperadrenocorticism, GC therapy, hyperthyroidism

–Neuromuscular disease – eg myasthenia gravis

17
Q

How do we approach a dyspnoea case? (5)

A

•How critical is the patient?

–Cats often present critically – the alter their activity so you wont know they’re v sick until bad.

•Palliative procedures

–Oxygenate

–Drain effusions

•Imaging

–Based on clinical exam findings

–You will never be wrong to use an ultrasound – pick up effusions and will start to determine whether there is any lung disease

–Radiograph and CT Is the best way to determine lung disease

•Diagnostic sampling

–Based on where you think the problem is

•Locating the site of difficulty is essential!

18
Q

What do you have to have to have heart failure?

What would you typically hear in cats and dogs?

A

–To have heart failure there has to be heart disease so auscultate. Usually:

  • Dog – murmur
  • Cat – gallop or murmur
19
Q

What is this and how is it used?

A

Cats – these new drains (seldinger) (catheter through chest, then guide wire, then pleura catheter)

Much easier to use

20
Q

Plain lateral thoracic radiograph from a dog, coughing and unwell for 2 or 3 days

Which lung pattern is evident?

A

Alveolar – must have air bronchgrams. Increase soft tissue density obliterating the cardiac. Too white for interstitial.

Dog probably has bronchopneumonia (maybe aspiration pneumonia). But typical presentation – ventral and cranial. Next: BAL.

Bronchial apttern – walls are thick and we can see then. White lines against the background (see white lines and doughnuts). Alveoli around it due to ST density – Alveolar due to AIR BRONCHOGRAMS

21
Q

•You have been presented with a 10 year old West Highland White Terrier that has been seen on numerous occasions over the past year with a cough and exercise intolerance. The owners have reported that despite various treatments the condition seems to be steadily progressing. You notice mild cyanosis of the mucus membranes and diffuse crackles over both hemithoraces.

What is the most likely differential diagnosis?

A

Idiopathic pulmonary fibrosis

22
Q

You have been investigating a horse which has a 3 month history of a foul-smelling unilateral nasal discharge. You have determined that the horse has dental disease of the upper 5th cheek tooth/2nd molar (triadan no 210).

Which structure is this tooth located in?

A

Caudal maxillary sinus

23
Q

Following removal of the tooth, the horse has a continuing sinusitis and inspissated pus within the sinus.

Which regime is recommended for removal of inspissated pus from a sinus?

A

Physical removal of the pus.

24
Q

Based on the history and presentation, select the disease which would be at the top of your differential list prior to examination of a herd with acute onset respiratory disease and milk drop in a large number of Friesian/Holstein dairy cows during January. The herd is open.

A

Infectious bovine rhinotracheitis (IBR).

25
Q

What is the difference between acute, sub-acute and chronic?

A

Acute – days

Sub acute – weeks

Chronic – months

26
Q

Which clinical sign is NOT commonly associated with IBR in cattle?

  1. Conjunctivitis.
  2. Nasal discharge.
  3. Hyperpnoea.
  4. Marked pyrexia.
  5. Erosive lesions on the hard palate.
A
  1. Erosive lesions on the hard palate.
27
Q

Which sampling and testing protocol is most appropriate for a rapid confirmation of your diagnosis of IBR?

A

Conjunctival swab from six acutely affected animals. Submit for Bovine Herpesvirus 1 (BHV-1) fluorescent antibody testing (FAT).

28
Q

True true (correct explanation), true true (not linked), true false, false true, false false?

The duration and severity of an Infectious Bovine Rhinotracheitis (IBR) outbreak can be limited by vaccination in the face of infection

BECAUSE

Animals vaccinated with an Infectious Bovine Rhinotracheitis (IBR) marker vaccine can be differentiated serologically from those which have seroconverted following natural infection

A

TRUE TRUE (not correct explaination)