Renal Sodium and Water Regulation Flashcards
Opiates are a (stimulatory/inhibitory) factor of antidiuretic hormone secretion
STIMULATORY
AHH is released by _______ of pituitary gland
POSTERIOR LOBE
channels in the luminal membranes allow for increased water permeability of principal cells in the late distal tubule and collecting duct, thus creating hyperosmotic urine.
Aquaporin 2
Nicotine is a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
STIMULATORY
The receptor coupled to adenylyl cyclase by a Gs protein is the receptor for antidiuretic hormone on principal cells of the late distal tubule and collecting duct.
V2 (AVPR2)
Atrial natriuretic peptide release is a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
INHIBITORY
Antineoplastic drugs are a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
STIMULATORY
Ethanol is a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
INHIBITORY
Hypoglycemia is a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
STIMULATORY
The receptor for antidiuretic hormone on vascular smooth muscles is coupled to phospholipase C by a Gq protein.
V1
Decreased extracellular fluid volume is a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
STIMULATORY
The main site of the kidney that antidiuretic hormone works on to increase water reabsorption is the principal cells of the .
Late Distal Tubule and Collecting Duct
Increased pain is a (stimulatory/inhibitory) factor of antidiuretic hormone secretion.
STIMULATORY
The target tissues of antidiuretic hormone are (two) .
KIDNEYS
ARTERIOLES
Antidiuretic hormone is synthesized in the paraventricular and nuclei of the hypothalamus.
PARAVENTRICULAR
SUPRAOPTIC
The main function of hormone is to regulate body fluid osmolarity.
ADH (Vasopressin)
When serum osmolarity is increased, the posterior pituitary secretes hormone
Antidiuretic Hormone
Step two in the countercurrent multiplication system is the flow of fluid from the proximal tubule into the of the loop of Henle.
Descending
The size of the corticopapillary gradient is dependent on the (length/width) of the loop of Henle.
LENGTH
The countercurrent multiplication system forms the corticopapillary osmotic gradient by (increasing/decreasing) sodium chloride deposits in the interstitial fluid of the deeper regions of the kidney.
INCREASING
The single effect in countercurrent multiplication refers to the (reabsorption/excretion) of sodium chloride into interstitial fluid.
Reabsorption
The osmolarity of the descending limb in the nephron is (equal/not equal) to the adjacent interstitial fluid.
EQUAL
The osmolarity of the fluid in the ascending limb of the loop of Henle continuously (increases/decreases) .
Decreases
The first step in the two-step process of building up a gradient through countercurrent multiplication is called the .
Single Effect
The descending limb of the nephron is permeable to water, so water flows (into/out of) the descending limb.
OUT OF
As tubular fluid flows down the collecting ducts, it is exposed to interstitial fluid with (higher/lower) osmolarity.
HIGHER
Countercurrent multiplication is a function of the (part of nephron) .
Loop of Henle
High/Low) osmolarity fluid is established in the descending limb of the loop of Henle during countercurrent multiplication.
HIGH