GU Book Flashcards

1
Q

do we have all the nephrons that we will ever have at birth

A

yes

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2
Q

when does most renal growth occur

A

during 5 yearsi

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3
Q

increase in renal size is due to

A

enlargement of nephrons

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4
Q

under 2 years old what can the kidneys not do

A

regulate electrolyes/fluids

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5
Q

infant UO

A

2mL/kg/hr

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6
Q

child UO

A

1mL/kg/hr

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7
Q

adol UO

A

40-80mL/hr

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8
Q

bladder capacity formula

A

age + 2 = reserve in oz
(ex: 4 yr + 2 = 6oz)

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9
Q

oz to mL

A

30mL

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10
Q

when can they maintain bladder control

A

2 years

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11
Q

UTI upper vs lower types

A

lower: cystitis and urethritis
upper: ureteritis and pyelonephritis

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12
Q

common cause of UTI

A

e coli

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13
Q

UTI s/s newborn

A

nonspecific
unexplained fever, failure to thrive, poor feeding, vomiting, strong smelling urine

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14
Q

UTI s/s lower infant

A

fever, diarrhea, irritability, foul smelling diaper, poor feeding, failure to gain wt

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15
Q

UTI s/s lower preschooler

A

hematuria, urgency, dysuria, frequency, cloudy urine, foul smelling urine, dehydration, abdominal pain

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16
Q

UTI s/s lower school

A

hematuria, urgency, dysuria, frequency, cloudy urine, foul smelling urine, dehydration, abdominal pain

suprapubic or flank pain

17
Q

UTI s/s upper

A

high fever, chills, flank pain, CVA tenderness, moderate to severe dehydration

18
Q

UTI tx

A

antibiotics

19
Q

hydrospadias

A

congenital anomalies involving abdonral location of the urethral meatus
- ventral surface
- bottom

20
Q

hydrospadias with kids who are having a circumsizion what happens

A

its delayed

21
Q

hydrospadias when is it corrected

A

1 year
- 6-12 mo

22
Q

hydrospadias stent or catheter, why?

A

maintain patency of the new urethram canal opening

23
Q

hydrospadias what technique do we use post op

A

double diaper
- outer one for pee
- inner one for poop

24
Q

nephrotic syndrome

A

alteration in kidney function 2ndary to increase glomerular basement membrane
permeability to plasma protein

25
Q

nephrotic syndrome is this a specific disease

A

no, its a clinical state

26
Q

nephrotic syndrome
- primary vs secondary

A

primary: no idenfeiable cause
2ndary: 2nd to something

27
Q

nephrotic syndrome s/s

A

massive proteinuria
hypoalbumenia
hypoproteinemia
hyperlipidemia
altered immunity
hypercoag
periorbital edema when waking that resolves during the day as fluid shifts to abdominal and lower extremities
hypertension
hematuria
foamy and frothy urine

28
Q

nephrotic syndrome tx

A

steroids
- prednisone
lasix

29
Q

acute post infectious glomerulonephritis cause

A

GABS

30
Q

acute post infectious glomerulonephritis what is it

A

become ill after contracting an infection of the URT or skin

glomerular damage occurs as a result of an immune complex reaction that localizes on the glomerular capillary wall

antibody and antigen complexes become lodged in the glomeruli leading to inflammation and obstruction

resulting in decrease glomerular filtration rate and vascular permeability increases and allows RBC and casts to be extorted

31
Q

acute post infectious glomerulonephritis s/s

A

midabominal pain
irritability
fever
microscopic or gross hematuria
tea colors urine
mild periorbital edema
dependent edema
pulmonary congestion
hypertnesion
oliguria

32
Q

acute post infectious glomerulonephritis BUN

A

increase

33
Q

acute post infectious glomerulonephritis protein

A

decrease

34
Q

acute post infectious glomerulonephritis tx

A

bed rest
sodium restrictions and lasix
- HTN and edema
antibiotics for original infection

35
Q
A