Cardiovascular Book Flashcards

1
Q

** what is preload
afterload
how do these two things work with contractility in terms of stroke volume

A

preload: end diastolic volume
afterload: amount of resistant the heart must overcome
the volume of blood ejected by the ventricle with each contraction

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2
Q

** how do we do a cardiac assessment in chikldren? what are we looking for? what should we be hearing?what shouldnt we see or hear? how do we evaulate overall work status in infants? what would you notice in a patient with cardiac failure if the were an infant? what if they were an older child

A

skin color, cap refill, heart rate/rhythm, BP, working of breathing
hearing S1 S2 S3
- S3 is normal in children related to rapid filling
should not see thril or S4 abnormal, heard late in diastole or early systole
CCHD: 95-98% with less than 3% change, R hand is preductal, either foot is postudctal
poor feeding, failure to thrive, tachpnea, tachycardia, poor wt gain, activity intolerance, developmental delayed

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3
Q

** what causes congetintal heart defects

A

drugs, age, chromosomal

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4
Q

**ASD

A

left to right atria,soft ejection murmur with fixed wide splitting of O2

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5
Q

** VSD

A

left to right ventricle, systolic murmur (3rd-4th intercostal) at sternal border, thrill may be present

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6
Q

** PDA

A

aorta to pulmonary artery, mcahnery murmr during systole and diastole, thrill over pulmonic artery

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7
Q

**TOF

A

pulmonary stenosis, right ventriclar hypertrophy, VSD, aortic overriding, tet spells (knee to chest morphine), boot shaped heart

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8
Q

**CoA

A

narrowing of descending aorta increase in BP in arms/neck decreasd BP in legs, PGE to keep PDA open

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9
Q

**HLHS

A

mitral and aortic valves are absent or stenosed with abnormally small left ventricle and small aorta, no murmur, PGE to keep PDA open

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10
Q

** TGA

A

aorta and pulmonary swtiched, egg shaped heart, cyanosis dosent improved with O2 PGE to keep PDA open

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11
Q

** are there specific nursing consdeirations for the disorder

A

PDA may give PGE and may not give O2 as itcan close
tet spell wants knees to chest

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12
Q

** understand what Kawasaki disease is, hoe does it present, what are the complications, what do you anticipate for nursing considerations and how is it treated?

A

acute stage: fever (5 days) >39, conjuctival hyperermia, cervivallymphode enlargement, strawberry tongue, cracker lips, rash on trunk
subacute: cracking skin (lips, fingers, toes) cornary artery anerysm
convalsecnt: 6-8 weeks after onset of s/s

monitor fever and CV status
IVIG 2g/kg for 18hrs

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13
Q

** how does hypovolemic shock occur? What are the stages of hypovolemic shock? is BP a good indicator of the Childs status? How to assess the patient for adequate tissue perfusion? what is the nursing management of shock? what would you anticipate for fluid replacement in shock?

A

hemorrhage, plasma loss from burns, nephrotic syndrome, sepsis, decrease fluids

no BP not good indicator, early comp is a normal BP, moderate decomposition is lowered systolic

urine output

IV fluid bolus, keep child warm

isotonic fluid

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14
Q

**review how you would care for a patient who is returning from cardiac Cath

A

vital signs, bleeding, infection, pressure dressing

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15
Q

why do infants have greater risk of heart failure than older children

A

due to sensitivity to volume or pressure overload

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16
Q

less compliance of the heart muscle means

A

stroke volume cannot increase substantially

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17
Q

how do kids increase CO

A

increase HR

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18
Q

polycythemia

A

increase RBC response to chronic hypoxemia

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19
Q

children respond to severe hypoxemia with

A

bradycardia

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20
Q

PDA

A

aorta to pulmonary artery

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21
Q

s/s of PDA

A

dyspnea
tachycardia
CHF
machinery murmur
thrill in pulmonic area

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22
Q

ASD

A

left to right atrium

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23
Q

ASD s/s

A

CHF
easy tiring
soft systolic ejection murmur

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24
Q

VSD

A

left to right ventricle

25
VSD s/s
CHF systolic murmur thrill
26
TOF
pul stenosis, RV hyperthropy, VSD, overriding of aorta
27
TOF s/s
polycythemia m acid knee to chest systolic murmur thrill
28
TOF radiology
boot shaped
29
TGA
pulmonary artery and aorta switch
30
s/s of TGA
cyanosis soon after birth and doesn't improve with O2 rapid resp rate systolic murmur - S2 is louder
31
TGA radiology
egg on a string
32
CoA
narrowing of the descending aorta which obstructs the systemic flow
33
s/s of CoA
decrease BP in legs bounding pulse in UE weak pulse in LE poor feeding CHF loud S2 systolic ejection murmur
34
HLHS
mitral or aortic valves are missing or stenosised with a abnormally small left ventricle and small aorta
35
S/S of HLHS
progressive cyanosis and CHF - reiterations, and decrease perierpal pulses pulmonary edema no murmur acidosis
36
HLHS murmur
none
37
most common cardiomegaly
dilated
38
what is occurring in dilated cardiomegaly
4 chambers dialate and systolic contraction is weakened
39
dilated cardiomeg s/s
weakness external dyspnea
40
dilated cardiomeg treatment
diuretics, dig, ACEI
41
what cardiomegaly is most common cause of sudden unexpected cardiac death in young athletes
hypertrophic
42
what is occurring in hypertrophic cardiomeg
enlargement of left ventricle and ventricle septum diastolic filling is affected
43
s/s of hypertrophic cardiomeg
dizziness chest pain
44
hypertrophic cardiomeg meds
beta blockers ca blockers
45
what is Kawasaki disease
acute febrile systematic vascular inflammation disorders
46
why is Kawasaki disease thought to occur
caused by infectious agent
47
3 stages of Kawasaki
acute subacute convalescent
48
Kawasaki acute stage time frame
1-2 weeks
49
Kawasaki acute stage s/s
high fever for 5 days hyperemic conjuct red throat swollen hands and feet maculopapular erythema multiform like rash on trunk and peri area
50
Kawasaki sub acte time frame
2+ weeks
51
Kawasaki subacute s/s
no fever cracking lips joint pain desquamation of skin on tips of fingers and toes
52
Kawasaki convalescent time frame
6-8 weeks after disease onset
53
Kawasaki convalescent s/s
lingering s/s of inflammation deep transverse lines may appear across nails on hands and feet
54
Kawasaki treatment
single high dose IVIG (2g/kg) over 10-12 hours
55
hypovolemic shock
inadequate tissue and organ perfusion resulting from inadequate blood or plasma volume in vascular space
56
s/s of hypovolemic shock
nonspecific for early sustained tachycardia increased resp effort weak peripheral pulses pallor cold extremities decrease urine output
57
hypovolemic shock treatment
20mL/kg over 5 mins
58