PERDENT (intro to dental caries) Flashcards

1
Q

is a multifactorial, transmissible,
infectious oral disease caused primarily
by the complex interaction of cariogenic
oral flora (biofilm) with fermentable
dietary carbohydrates on the tooth
surface over time.

A

DENTAL CARIES

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2
Q

characterized by
localized demineralization and loss of
tooth structure

A

dental caries

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3
Q

During an acid attack
PH level will go down
to critical level:

A

5.5 for enamel

6.2 for dentin

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4
Q

The low pH level will
trigger Phosphate and
Calcium minerals from
the tooth to the Biofilm
in attempt to balance or
to reach equilibrium.

A

DEMINERALIZATION

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5
Q

When pH neutralizes, the
concentration of soluble
calcium and phosphate is
supersaturated relative to that
in the tooth, mineral can then
be added back to partially
demineralized enamel

A

ReMINERALIZATION

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6
Q

Repeated demineralization events may
result from a predominantly pathologic environment causing the localized dissolution and destruction of the calcified dental tissues,

A

caries lesion or a “cavity.”

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7
Q

factors that can affect the process of

demineralization and remineralization

A

Number and type of microbial flora in the biofilm

diet
oral hygiene
genetics
dental anatomy
use of fluorides and other chemotherapeutic agents

salivary flow and buffering capacity

inherent resistance of the tooth structure and composition.

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8
Q

is a term historically
used to describe the soft,
tenacious film
accumulating on the
surface of teeth.

A

Dental plaque

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9
Q

has been more recently
referred to as a plaque
biofilm, or simply biofilm

A

Dental plaque

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10
Q

is composed mostly of bacteria,
their by-products, extracellular
matrix, and water.

A

biofilm

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11
Q

Teeth normally have a biofilm
community dominated by

A

Streptococcus sanguis and S.
mitis

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12
Q

The population size of mutans
streptococci (MS) or S.

A

mutans on
teeth varies.

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13
Q

tooth habitats for cariogenic biofilms

A

Pits and fissures

Smooth enamel surfaces

Root surfaces (cervical areas)

Subgingival areas

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14
Q

provide excellent mechanical shelter for organisms
and harbor a community dominated by S. sanguis and

other streptococci

A

pits and fissures

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15
Q

immediately gingival to
the contact area are the second most susceptible areas to caries

A

Smooth Enamel Surfaces

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16
Q

particularly near the cementoenamel junction (CEJ), often is unaffected by the action of hygiene procedures such as flossing because it may have concave anatomic surface contours (fluting) and occasional roughness at the termination of the enamel.

A

root surfaces

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17
Q

Caries originating on the root is
alarming because:

A

1.it has a comparatively rapid
progression.
2.it is often asymptomatic.
3.it is closer to the pulp.
4. it is more difficult to restore.

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18
Q

Accomplished primarily by
proper tooth brushing and
flossing, is another ecologic
determinant of caries onset
and activity.

A

oral hygiene

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19
Q

Careful mechanical cleaning
of teeth disrupts the biofilm
and leaves a clean enamel
surface.

A

oral hygiene

20
Q

The cleaning process does
not destroy most of the oral
bacteria but merely removes
them from the surfaces of
teeth.

A

oral hygiene

21
Q

is an extremely important
substance for the proper digestion
of foods, and it also plays a key
role as a natural anticaries agent

22
Q

protective mechanisms
that maintain the normal oral flora
and tooth surface integrity include
bacterial clearance, direct
antibacterial activity, buffers, and
remineralization

23
Q

anticaries properties of saliva

A

Bacterial Clearance

Direct Antibacterial Activity

Buffer Capacity
Remineralization

24
Q

The amount of saliva secreted
varies greatly over time. When
secreted, saliva remains in the
mouth for a short time before
being swallowed.

A

bacterial clearance

25
Q

Adults produce ___ of saliva a
day, very little of which occurs
during __.

A

1-1.5 L, sleep

26
Q

most effective
during mastication or oral
stimulation, both of which
produce large volumes of saliva.

27
Q

play an important role in the
protection of soft tissue in the
oral cavity from infection by
pathogens, they have little effect
on caries because similar levels of
antibacterial proteins can be
found in caries-active and caries-
free individuals.

A

direct bacterial activity

28
Q

also can
dilute and buffer biofilm acids.

A

Large volumes of saliva

29
Q

may have significantly higher
caries susceptibility.

A

Individuals with decreased
salivary production (Dry mouth)

30
Q

medical term for Dry mouth.

A

Xerostomia

31
Q

`xeros =
stomia =

A

= dry
= conditions of the mouth

32
Q

is to
reduce the potential for acid
formation.

A

buffer capacity

33
Q

Without a means to control
precipitation of these ions, the
teeth literally would become
encrusted with mineral deposits.

A

remineralization

34
Q

provides a constant opportunity
for remineralizing enamel and can
help protect teeth in times of
cariogenic challenges.

A

remineralization

35
Q

Acidogenic
Acido =

genic =

A

Acid

forming/producing

36
Q

The eventual metabolic product of
cariogenic diet is

37
Q

leads to the development of caries,
the exposure to acidity from other
sources also may result in caries.

38
Q

Cariogenic
Cario =

genic =

A

Caries

forming/producing

39
Q

clinical characteristics

A

Pits and Fissures

Smooth enamel surfaces

Root surfaces

40
Q

the earliest
evidence of caries on the smooth
enamel surface of a crown is a

A

white spot.

41
Q

are chalky white, opaque areas
that are revealed only when the
tooth surface is desiccated and
are termed noncavitated enamel
caries lesions.

A

white spots

42
Q

is affected
less by drying and wetting

A

hypocalcified enamel

43
Q

caries
partially or totally disappears
visually when the enamel is
hydrated (wet),

A

Noncavitated (white spot)

44
Q

(international caries dectection assessment system) ICDAS

1
2
3
4
5
6
7

A

1 - Sound surface
2 - First visual change in enamel (seen only after prolonged air‐drying or
restricted to within the confines of a pit or fissure)
3 - Distinct visual change in enamel
4 - Localized enamel breakdown (without clinical visual signs of dentin
involvement)
5 - Underlying dark shadow from dentin
6 - Distinct cavity with visible dentin
7 - Extensive distinct cavity with visible dentin

45
Q

buccal and linggual

grade 1 (B1)
grade 2 (B2)
grade 3 (B3)
grade 4 (B4)
grade 5 (B5)

A

GRADE 1- white or discolored enamel, no cavitation clinically.

GRADE 2- small cavitation in enamel.

GRADE 3- moderate sized cavity in enamel with exposed dentin

GRADE 4- large cavity in enamel and moderate cavity in dentin

GRADE 5- extensive cavity in enamel and substantial loss of denitn

46
Q

occlusal

grade 1(o1)
grade 2(O2)
grade 3(o3)
grade 4(o4)
grade 5(05)

A

GRADE 1- white or brown discoloration in enamel, no clincial cavitation, no radiographic

GRADE 2- small cavity formation

GRADE 3- moderate sized cavity and radiolucency in the outer third

GRADE 4- Big cavitation in the middle third

GRADE5- very big in the iner third