Lec 8-6 - Reproduction Flashcards
these are present until 6-7 weeks and are bipotential but atrophy without testosterone
wolffian ducts (male structures)
these grow unless they are suppressed by MIF
mullerian ducts (female structures)
these secrete MIF
sertoli cells
these secrete testosterone and are activated by LH
leydig cells
female development is due to a lack of blank
hormones
primordial germ cells do not enter meiosis in blank but they do enter meiosis in blank
females, males
during puberty, sex hormone levels are blank
low
during puberty, there is a night time pubertal blank surge
LH
in males, this causes genetalia, musculature, growth spurt, larynx
testosterone
in males, this causes sexual hair, sebaceous glands
dihydrotestosterone
in females, this causes genetalia, breasts, pubic hair, fat distribution
estrogen
sertoli cells are aka because they allow sperm development and are activated by FSH and high testosterone
nurse cells
maturation of sperm takes this long
70 days
primary follicle consists of an oocyte surrounded by blank and blank cells
granulosa, theca
granulosa cells are female equivalent to blank cells and respond to blank and produce blank
sertoli, fsh, estradiol
theca cells are female equivalent to blank cells and respond to blank and produce blank
leydig, LH, androgens
oocyte completes first meiotic division at time of blank and second division at blank
ovulation, fertilization
corpus luteum responds to blank and blank to produce blank and blank
fsh/LH, estradiol/progesterone
during implantation, a placenta is formed and secretes blank which maintains blank and blank production from corpus luteum
hCG, estrogen, progesterone
blank in placenta convert androgens to estrogens
aromatase
onset of labor is triggered by these in sheep but we dont know in humans
cortisol, increased estrogen/progesterone ratio
hormones in labor after the onset has already happened
prostaglandins, oxytocin, catecholamines
prolactin inhibits this after giving birth
menstrual cycle
estrogen is a blank antagonist
calcium
menopause has a loss of blank due to losing theca and gruanulosa cells
estrogen, progesterone
maternal anabolic phase (first half of pregnancy to start storing food for fetus) has blank sensitivity to insulin
increased
maternal catabolic phase has accelerated blank and blank sensitivity to insulin
starvation, decreased
placenta cranks out these hormones
progesterone, estrogen
blank is responsible for the let down reflex of milk
oxytocin