Fetal Heart Monitoring Lecture Powerpoint Flashcards

1
Q

2 methods to monitor the fetal heart rate

A
  • auscultation
  • electronically (nowadays this is the most common obstetrical procedure reviewed frequently thru the first and 2nd stages of delivery)
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2
Q

Methods of external fetal heart rate monitoring electronically (2)

A
  • ultrasound doppler with gel held in position by belt (not as strong)
  • internal FHR monitoring, a bipolar electrode attached to fetal scalp with a wire penetrating the scalp (membranes are ruptured) where a 2nd pole is the metal base of the electrode and vaginal fluid creates saline electric bridge (requires intact maternal physiology, functional placenta, and fetus)
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3
Q

Insufficient o2 or nutrition (chronic, long term malnutrition) in the mother can cause these things (5)

A
  • intrauterine growth restriction
  • hypoxia
  • metabolic acidosis
  • acute aspyxia
  • fetal death
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4
Q

Placenta function can be chronically impaired by these things (5)

A
  • hypertension
  • pre-eclampsia
  • type 1 diabetes
  • Rh sensitization
  • intrauterine growth restriction
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5
Q

Placenta function can be acutely impaired by these things (3)

A
  • placental separation
  • placental infarct
  • maternal or fetal hypotension
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6
Q

Tachysystole (uterine activity)

A

More than 5 contractions in 10 min averaged over 30 minutes, should always be qualified as to the presence or absence of fetal heart rate abnormality

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7
Q

Basic warning signs in labor (4)

A
  • contractions lasting longer than 90 seconds (starve fetus of oxygen and nutrients)
  • relaxation between contractions of less than 60 seconds (if not at rest for long enough, not enough time to wash out co2 and gain o2 and nutrients)
  • resting uterine tone above 20mmhg (normal is 5-10 mmHg, during labor may be 10-15)
  • peak pressure of contractions above 90 mmHg
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8
Q

Normal fetal heart rate during development and at term

A

110-160bpm, until week 26 heart rate controlled by sympathetic NS, as matures parasympathetic increases in control slowing the heart to 110-120bpm

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9
Q

Tachycardia of fetal heart rate

A

Greater than 160bpm

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10
Q

Factors associated with fetal tachycardia (6)

A
  • fetal/maternal temp elevation
  • medication side effects
  • acidosis
  • arrhythmia
  • hypovolemia
  • hyperthyroidism
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11
Q

Factors associated with fetal bradycardia (4)

A
  • post term fetus (mature parasympathetic system)
  • hypoxia
  • B blockers
  • damage to fetal heart
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12
Q

Short term healthy variability in fetal heart rate

A

Should have amplitude ranging from 5-15 beats per minute indicates good autonomic interplay

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13
Q

Short term variability in the fetal heart rate below 5bpm may indicate (4)

A
  • fetal sleep period
  • CNS depression
  • hypoxia
  • drugs or maternal smoking
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14
Q

Response to decreased short term or long term variability in fetal heart rate (5)

A
  • stimulate fetus
  • attach scalp electrode for internal monitor
  • check fetal pH
  • change maternal position
  • just deliver the damn thing
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15
Q

Long term variability in fetal heart rate

A

Variability in the baseline rate over a more extended period of time (1 min) usually amplitude of 3-25 bpm, decreased during sleep, drugs, hypoxia, and tachycardia and increased during hypoxia

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16
Q

Head compression

A

Predictable event as uterus compresses engaged head causing vagal slowing causing early decelaration of fetal heart rate and ends with same time as uterine contraction, NOT considered a sign of distress

17
Q

Uteroplacental insufficiency

A

Predictable event as uterus compresses engaged head causing vagal slowing causing LATE decelaration of fetal heart rate in comparison to uterine contraction,

18
Q

Response to uteroplacental insufficiency (late decelerations)

A
  • change maternal position to side (prevent positional compression)
  • stop oxytocin
  • increase IV fluids
  • o2 for mom up to 100%
19
Q

Cord compression

A

Predictable event as uterus comresses the umbilical cord causing VARIABLE decelerations of fetal heart rate, most common, if prolonged causes hypoxia

20
Q

Response to cord compression (variable decelerations) (4)

A
  • change maternal position to right or left side
  • 100% o2
  • stop oxytocin
  • deliver via forceps or c-section
21
Q

Category 1-3 FHR tracings

A

Category 1 - normal, strongly predictive of normal fetal acid base status at delivery
Category 2 - Tracings are indeterminate, not predictive of abnormal fetal acid base status, require eval and continued surveillance and re-eval
category 3 - tracins are abnormal, associated with abnormal fetal acid base balance, requires prompt eval, maternal o2, change in position, etc

22
Q

Category 1 FHR tracings characteristics

A
Baseline rate 110-160
Baseline variability: moderate
Late or variable decelerations: absent
Early decelerations: present or absent
Accelerations: present or absent
23
Q

Category 2 FHR tracings characteristics

A

Baseline rate: bradycardia or tachycardia without absent baseline variability
Baseline Variability: absent with no recurring decelerations, minimal, marked
Accelerations: absent or present after fetal stimulation

24
Q

Category 3 FHR tracings characteristics

A
Category 3 tracings include absent baseline FHR variability and any of the following:
Recurring late decelerations
Recurring variable decelerations
Bradycardia
Sinusoidal pattern
25
Q

Brain damage in labor determining factors (did it happen during labor and not beforehand?) (4)

A
  • profound umbilical artery acidemia (pH <7)
  • apgar score 0-3 for longer than 5 min
  • neonatal neurological sequelae (seizures, coma, hypotonia)
  • multiorgan system dysfunction
26
Q

Fetal monitoring overreliance

A

Out of 1000 fetuses with appearing fetal distress on FHR tracing only 1-2 developed cerebral palsy, in other words we are over c-sectioning due to overreliance however it is better than auscultation technique in most and makes it easier on nurses so it is here to stay