Integumentary Pathologies Flashcards

1
Q

Cellulitis

A
  • fast spreading inflammation that occurs as result of bacterial infection of the skin and connective tissues
  • can develop anywhere under the skin but typically affects the extremities
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2
Q

cellulitis etiology

A
  • caused by particular bacterial infections including streptococci or staphylococci
  • predisposing factors include increased age, immunosuppression, trauma, the presence of wounds or venous insufficiency
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3
Q

cellulitis s/s

A
  • localized redness that may spread quickly
  • skin that is warm or hot to touch
  • local abscess or ulceration
  • tender to palpation
  • chills
  • fever
  • malaise
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4
Q

cellulitis treatment

A
  • should be immediately referred to a physician
  • requires pharmacological intervention usin systemic antibiotics
  • differential diagnosis should attempt to rule out deep vein thrombosis and contact dermatitis
  • can lead to sepsis or gangrene
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5
Q

contact dermatitis

A
  • superficial irritation of the skin resulting from the irritation (poison ivy, latex, soap, jewelry sensitivity)
  • can be acute or chronic based on exposure
  • very common that can occur at any age
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6
Q

contact dermatitis etiology

A
  • occurs with exposure to mechanical, chemical, environmental or biological agents
  • nickel, rubber, latex, and topical antibiotics are common precipitating agents
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7
Q

contact dermatitis s/s

A
  • intense itching
  • burning
  • red skin in areas of irritation
  • edema
  • symptoms can expand beyond the initial point of topical irritation
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8
Q

contact dermatitis treatment

A
  • should focus on identifying and removing the source of irritation
  • topical steroid application is commonly employed
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9
Q

eczema

A
  • used to describe a group of disorders that cause chronic skin inflammation typically due to an immune system abnormality, allergic reaction or external irritant
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10
Q

eczema etiology

A
  • based on the particular form of the disorder
  • infants and children are at higher risk for eczema but outgrow the condition with age
  • geriatric population is at an increased risk
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11
Q

eczema s/s

A
  • red or brown-gray, itchy, lichenified skin plaques that may be exacerbated by some topical agents such as soaps and lotions
  • younger population with experience oozing and crushing of the patchy areas of irritation
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12
Q

eczema treatment

A
  • pharmacological vary from topical to oral corticosteroids to oral antibiotics and antihistamines
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13
Q

gangrene dry

A
  • loss of vascular supply resulting in local tissue death
  • fingers, toes, and limbs are often most affected
  • hardened tissue is not painful
  • may be significant pain at the line of demarcation
  • typically develops slowly and in some cases results in auto amputation
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14
Q

gangrene dry etiology

A
  • most commonly in blood vessel disease such as diabetes or atherosclerosis
  • develops when blood flow to an affected area is impaired
  • infection is not typically present however it can progress to wet gangrene if infection occurs
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15
Q

gangrene dry s/s

A
  • presents with dark brown or black nonviable tissue that eventfully becomes a hardened mass
  • patient may complain of cold or numb skin and they may present with pain
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16
Q

gangrene dry treatment

A
  • may be treated with pharmacological intervention, surgery, and hyperbaric oxygen therapy
17
Q

gangrene wet

A
  • referred as wet if there is an associated bacterial infection in the affected tissue
  • may develop as a complication of an infected untreated wound
  • swelling resulting from te bacterial infection causes a sudden stoppage of blood flow
18
Q

gangrene wet etiology

A
  • can develop after a severe burn, frostbite, or injury and requires immediate treatment since it can spread quickly and be fatal
  • there is a cessation of blood flow that starts a chain of events including invasion by bacteria at the affected site
  • as a result of the occluded blood supply, the white blood cells are unable to fight the infection
19
Q

gangrene wet s/s

A
  • swelling and pain at site of infection
  • change in skin color from red to brown to black
  • blisters that produce pus
  • fever
  • general malaise
20
Q

gangrene wet treatment

A
  • requires immediate medial intervention
  • surgical debridement of the gangrene and intravenous antibiotic treatment are typical
  • can be treated via pharmacological intervention, surgery, and hyperbaric oxygen therapy
21
Q

onychomycosis

A
  • fungal infection that primarily affects the toenails and nailbeds
  • divided into subtypes but treated fairly the same
22
Q

onychomycosis etiology

A
  • acquiring a fungal infection can be a fairly common
  • risk factors include manicures and pedicures with unsteril utensils, possessing nail injuries or deformities, excess skin moisture, wearing closed toe shoes, and an impaired immune response
23
Q

onychomycosis s/s

A
  • yellow or brown nail discoloration

- hyperkeratosis and hypertrophy of the nail causing to partially detach from the nailbed

24
Q

onychomycosis treatment

A
  • manual debridement of nail and topical antifungal medications
  • fungal infections may return
  • nailbeds may have permanent damage
25
Q

plaque psoriasis

A
  • most common of five types of psoriasis
  • chronic autoimmune disease of the skin
  • T cells trigger inflammation of within the skin and produce an accelerated rate of skin cell growth
  • skin cells accumulate in raised red patches on the surface of the skin
26
Q

plaque psoriasis etiology

A
  • some have genetic predisposition
  • other factors may trigger psoriasis such as injury to skin, insufficient or excess sunlight, stress, excessive alcohol, HIV infection, smoking, or certain medications
27
Q

plaque psoriasis s/s

A
  • red raised blotches that present in bilateral fashion
  • appear anywhere on the body and will itch and flake
  • complications can include arthritis, pain, severe itching, secondary skin infections, and side effects secondary to pharmacological interventions
28
Q

plaque psoriasis treatment

A
  • primary goal for treatment is to control the symptoms and prevent secondary infection
  • varies widely from topical application to systemic medications and phototherapy
  • is a life long condition that can be effectively managed
29
Q

tinea pedis

A
  • referred to as athletes foot
  • superficial fungal infection which causes epidermal thickening and a scaly skin appearance
  • will rapidly multiply in a warm and moist environment
30
Q

tinea pedis etiology

A
  • risk factors include wearing closed toe shoes that dont allow airflow, prolonged periods of moisture or wetness, excessive sweating, and possessing small nail or skin abrasions
  • infection is contagious through direct contact or when making contact with a surface containing the tinea pedis
31
Q

tinea pedis s/s

A
  • itching
  • redness
  • peeling skin between the toes
  • pain
  • odor
  • and in more severe cases breaks in skin continuity
32
Q

tinea pedis treatment

A
  • pharmacological intervention includes topical or oral antibiotics
  • may persist or recur
  • prevention includes thorough drying of feet when bathing or swimming, wearing sandals around public pools or showers, changing socks frequently, proper hygiene, and avoided shoe wear that creates a moist environment