Effect of Lesions Flashcards

1
Q

C1-C5…
Mental status?
Gait (LMN or UMN)? Ataxia?
Cranial nerves?
Postural reaction?
Limb tone?
Reflexes?
Cutaneous trunci?

A

Mental status = NORMAL

Gait = UMN, long and flowy steps in thoracic and pelvic limbs

Ataxia = SPINAL ATAXIA (crossing over limbs, abduction, long stride) & SPASTIC HEMI/TETRAPARESIS

Cranial nerves = Mostly normal

Postural reaction = normal to delayed in all limbs

Limb tone = normal to increased in all limbs

Reflexes = hyperreflexive in all limbs

Cutaneous trunci = normal

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2
Q

C6-T2
Mental Status?
Gait?
Cranial nerves?
Postural reaction?
Limb tone?
Reflexes?
Cutaneous trunci?
Sensation
Others

A

Mental status = NORMAL

Gait = “2-engine gait”
LMN in FRONT; short & choppy
UMN in PELVIC; long and flowy

Cranial nerves = mostly normal

Postural reaction = NORMAL to DELAYED in ALL LIMBS

Limb tone = DECREASED in front, NORMAL to INCREASED in hind

Reflexes =
DECREASED to ABSENT in front NORMAL to INCREASED in HIND

Cutaneous trunci = NORMAL or COMPLETELY ABSENT (C8-T1)

Sensation = DECREASED IPSILATERAL and caudal to lesion

Other
- Horner’s syndrome
- Issues with diaphragmatic breathing?
- URINARY INCONTINENCE/DECREASED ABILITY TO EXPRESS BLADDER

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3
Q

T3-L3
Mental Status?
Gait?
Cranial nerves?
Postural reaction?
Limb tone?
Reflexes?
Cutaneous trunci?
3 other things?

A

Mental status = NORMAL

Gait?
NORMAL in front
UMN in hind (long and flowy)

Cranial nerves? = Normal

Postural reaction?
NORMAL in front
NORMAL to DELAYED in hind

Limb tone?
NORMAL in front
NORMAL to INCREASED in hind

Reflexes?
NORMAL in front
NORMAL to INCREASED in hind

Cutaneous trunci?
- COULD be normal if lesion is NOT SEVERE ENOUGH TO CAUSE A PROBLEM
- Can localize lesions 2-4 segments CRANIAL to where CUTANEOUS TRUNCI RETURNS

3 others?
1. UMN bladder = hard to express/overcome pressure of SPHINCTER
2. SPINAL SHOCK = absence of reflexes in hind correlate with SEVERITY of spinal damage
3. SCHIFF-SHERRINGTON POSTURE = hindlimbs PARAPARETIC, forelimbs SPASTIC/STIFF

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4
Q

L4-S1
Mental status?
Gait?
Cranial nerves?
Postural reactions?
Limb tone?
Reflexes?
Cutaneous trunci?
4 others?

A

Mental status = NORMAL

Gait?
NORMAL in front
LMN (short and choppy) in HIND

Cranial nerves? = NORMAL

Postural reactions?
NORMAL in front
DELAYED to ABSENT in hind

Limb tone?
NORMAL in front
DECREASED in hind

Reflexes?
NORMAL in front
DECREASED to ABSENT in hind

Cutaneous trunci? = NORMAL

4 others?
1. LMN bladder = FLACCID, constantly LEAKS
2. PSEUDO-HYPERREFLEXIA = issue with SCIATIC NERVE causes PATELLAR HYPERREFLEXIA
3. DECREASED TAIL TONE
4. DECREASED ANAL TONE

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5
Q

Peripheral nerve lesions

A

Gait
- Stiff, short-strided gait
- Flaccid paresis/paralysis
- Generalized weakness/exercise tolerance

Postural reactions
- Normal to diminished

Reflexes
- Hyporeflexia/areflexia
- Hypotonia/atonia/flaccidity
- Muscle atrophy

Other
- Fasciculations and muscle tremors
- +/- urinary and fecal incontinence
- Muscle atrophy

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6
Q

LMN spinal ataxia clinical signs…
Stride length?
Postural reactions?
Reflexes? (2)
Muscle tone?
Muscle atrophy
often caused by…

A

Stride = SHORT-STRIDED

Postural reactions = DECREASED

Reflexes =
1. DECREASED reflexes
2. WEAK WITHDRAWAL

Muscle tone = FLACCID

RAPID MUSCLE ATROPHY +

Often caused by peripheral nerve disease

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7
Q

UMN spinal ataxia clinical signs
Stride length?
Postural reactions?
Reflexes?
Muscle tone?
Muscle atrophy?

A

stride = LONG-STRIDED

postural reactions = DECREASED

reflexes = HYPERREFLEXIA

muscle tone = SPASTIC

NO RAPID MUSCLE ATROPHY bc limb can still be used

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8
Q

what are the 3 types of ataxia?

A
  1. spinal
  2. vestibular
  3. cerebellar
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9
Q

SPINAL ataxia..
aka?
3 characteristics?

A

aka PROPRIOCEPTIVE ataxia

3 characteristics?
1. crossing over limbs
2. INCREASED stride length
3. abduction/circumduction of limbs (moving AWAY from body)

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10
Q

VESTIBULAR ataxia…
caused by disease of what 3 possible structures?

A

3 structures?
1. inner ear
2. vestibular nuclei
3. cerebellum

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11
Q

CEREBELLAR ataxia…
aka?
characterized by what 3 signs?

A

aka DYSMETRIA

characterized by…
1. abnormal RATE or RANGE of motion
2. OVER-STEPPING/GOOSE-STEPPING
3. delayed onset of voluntary motion

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12
Q

dysmetria definition

A

the inability to control the distance, speed, and range of motion necessary to perform smoothly coordinated movements

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13
Q

name the 2 neurolocalizations where we can see MENTATION change

A
  1. forebrain
  2. reticular activating system in midbrain
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14
Q

what are 2 clinical signs & 1 dz that can originate from injury at C1-C5?

A
  1. Horner’s syndrome
  2. Cervical muscle spasms
  3. Urinary INCONTINENCE/DECREASED ABILITY TO EXPRESS BLADDER
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15
Q

what neurolocalization can cause acute respiratory distress & why?

A

C1-C5

because C3-C5 innervates the PHRENIC nerve, can inhibit respiration!

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16
Q

what parameter should we monitor in C1-C5 injury & why?

A

ETCO2 because C3-C5 innervates phrenic nerve, want to make sure NOT HYPOVENTILATING