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Year 1/2 > Digestion & Metabolism 2: LA Common GI Parasites > Flashcards

Digestion & Metabolism 2: LA Common GI Parasites Flashcards

1
Q

STRONGYLES…
aka…
they are the ___ ___ parasites in LA
description? (2)
PRE-PATENT PERIOD in RUMINANTS?
PRE-PATENT PERIOD in HORSES
distribution?

A

STRONGYLIDS/GI NEMATODES

MOST IMPORTANT

description?
1. SMALL
2. HAIR-LIKE

PRE-PATENT PERIOD in…
RUMINANTS = 2-4 weeks
HORSES = 6-18 weeks
–> MORE VARIABEL because MANY SPECIES OF STRONGYLES CAN INFECT THEM

WIDE DISTRIBUTION THROUGHOUT THE WORLD

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2
Q

LIFE CYCLE of STRONGYLIDS…
divided into 2 phases?
5 stages
what’s special about L3 larvae? what 3 things does it give L3 larvae?

A

2 phases?
1. PRE-PARASITIC = EGG to L3
2. PARASITIC = L3 to ADULT

6 stages?
1. EGG = passed in FECES
–> EPSILLOIDAL, THIN-SHELLED, MORULATED
2. L1 develops in egg and HATCHES INTO ENVIRONMENT
3. L1 –> L3 in ENVIRONMENT
4. L3 INFECTIVE stage is INGESTED DURING GRAZING
5. L3 –> ADULT IN HOST

L3 LARVAE KEEPS 2 CUTICLES, ONE KEPT FROM PREVIOUS STAGE, causing…
1. can SURVIVE up to 6 MONTHS
2. PROTECTS WORM FROM DESSICATION
3. PREVENTS L3 from EATING, so must LIVE OFF OF ENERGY RESERVES

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3
Q

PRE-PARASITIC portion of STRONGYLE LIFE CYCLE…
range?
where are L1/L2 located & what do they feed on?
how long can development range?
HIGHLY DEPENDENT ON ____ ____

A

range? = EGG –> L3

L1/L2 located in FECES where they FEED ON BACTERIA/DEBRIS

development to L3 can range from 1 –> MULTIPLE weeks

highly dependent on ENVIRONMENTAL CONDITIONS

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4
Q

ID PARASITE

A

STRONGYLES

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5
Q

STRONGYLE life cycle stages IN HOST..
restricted to WHAT LOCATION?
how can nematodes AVOID death during SEVERE WINTERS?

A

ONLY IN GI/ALIMENTARY TRACT

avoid death in winter?
–> CAN ARREST IN ALIMENTARY MUCOSA UNTIL WARMER/ABLE TO DEVELOP & WITHSTAND ENVIRONMENT

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6
Q

HAEMONCHUS spp…
“common name?” & why? (+ WHAT SEX)

what KIND of parasite?

2 species of organism & what animals it affects?

adult worms are located WHERE?

2 clinical signs?

better adapted to WHAT kind of environments?

A

“BARBER POLE WORM” –> feeds on BLOOD and has STRIPED appearance for FEMALES

GI NEMATODE

2 species?
1. H. CONTORTUS = CERVIDS, RUMINANTS
2. H. PLACEI = CATTLE in SUBTROPICS/SOUTHERN US

ADULT worms in the ABOMASUM

CLINICAL SIGNS?
1. BLOOD LOSS/ANEMIA
2. SUBMANDIBULAR EDEMA

better adapted to WARMER CLIMATES/SEASONS

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7
Q

OSTERTAGIA/TELODORSAGIA…
“common name?”
describe animals affected in each?
present in WHAT kind of climates? how important is it?
adults are found where?
clinical signs in YOUNG CATTLE?
CHARACTERISTIC LESION? & leads to WHAT?

A

“BROWN STOMACH WORM”

animals affected?
1. OSTERTAGIA = CATTLE
2. TELODORSAGIA = SHEEP/GOATS

IMPORTANT CATTLE PARASITE in COOLER environments

adults are found in ABOMASUM

in YOUNG CATTLE…
–> CHRONIC ABOMASITIS

CHARACTERISTIC LESION?
–> “MOROCCAN LEATHER” = NODULAR APPEARANCE OF GASTRIC GLANDS due to ENCYSTED LARVAE
–> leads to ABOMASAL DYSFUNCTION

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8
Q

LARGE strongyles…
affects WHAT species?
3 examples of organisms? which one is MOST pathogenic?
prevalence?
adults are found in what 2 locations?
what dangerous disease can they cause? what SPECIFIC organism does this? (2)
larval migration locations in other 2 organisms?

A

affects HORSES

3 examples?
1. S. VULGARIS –> MOST PATHOGENIC
2. S. EDENATUS
3. S. EQUINUS

currently LOW prevalence

adults can be found in CECUM & VENTRAL COLON

dangerous dz?
= in S. VULGARIS!
1. LARVAE can MIGRATE and cause ARTERIAL DAMAGE
2. COLIC

larval migrations in…
1. S. EDENATUS = LIVER, PERITONEAL WALL
2. S. EQUINUS = LIVER, PANCREAS

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9
Q

WHY are large strongyles NOT COMMON/IMPORTANT anymore?

why do we NOT see resistant population of LARGE STRONGYLES?

A

in the past, owners used to CONSTANTLY ROTATE DIFFERENT DEWORMERS, so it’s mostly gone!

not sure why we don’t see resistance, but IF YOU GIVE IVERMECTIN, KILLS LARGE STRONGYLES WITHOUT DEVELOPMENT OF RESISTANCE

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10
Q

CYATHOSTOMINAE
aka “name?”
commonality?
2 locations adults are found in?
3 seasons the clinical signs tend to occur?
3 clinical signs?
what stage of development is MOSTLY responsible for PATHOLOGY? (2)

A

aka SMALL STRONGYLES

MOST COMMON PARASITE OF HORSES

adults found in CECUM & COLON

3 seasons?
1. LATE FALL
2. WINTER
3. EARLY SPRING

3 clinical signs?
1. WATERY D+
2. SEVERE INFLAMMATION OF INTESTINE
3. OFTEN FATAL

LARVAL MIGRATION is MOSTLY to blame for pathology!
1. ENCYSTS in MUCOSA OF INTESTINE causing ULCERS
2. produces “PEPPERED” appearance to mucosa

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11
Q

ID PARASITE & LESION

A

CYATHOSTOMINAE

“PEPPERED” ULCERS ACROSS MUCOSA

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12
Q

ID 2 POTENTIAL DZs?

A

OSTERTAGIA/TELADORSAGIA

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13
Q

3 concepts of LA parasitology…

A
  1. ALL livestock EVOLVED WITH PARASITES, they’re considered “NORMAL FLORA”
  2. can get INFECTED while GRAZING because they GRAZE WHERE THEY POOP
  3. infection is NORMAL AND EXPECTED, we can use QUANTITATIVE DIAGNOSTIC TESTS
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14
Q

ASCARIDS…
species in HORSES, RUMINANTS, SWINE?
lives in WHERE? occasionally found in WHAT OTHER 2 LOCATIONS?
females are VERY ___
what’s special about the eggs?
pre-patent period?

A

species in…
HORSES = PARASCARIS spp.
RUMINANTS = TOXOCARA VITULORUM
SWINE = ACARIS SUUM

lives in the SMALL INTESTINE, but occasionally found…
1. STOMACH
2. CECUM

females are VERY FECUND

eggs can be VERY RESISTANT

PPP = 3-8 weeks

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15
Q

ID PARASITE

A

ASCARIDS

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16
Q

ASCARID LIFE CYCLE…
what TYPE of life cycle?
what is the INFECTIVE stage?
what growth occurs in ENVIRONMENT?
what growth occurs in the LIVER?
what’s special about L3 stage? (4)

A

DIRECT life cycle!

INFECTIVE stage = EGG that’s been SHED INTO FECES WITH THIRD-STAGE LARVAE

in ENVIRONMENT = L1 –> L3 WITHIN EGG

in HOST = MIGRATION & GROWTH occurs within the LIVER

L3 stage…
1. can survive up to 6 months in ENVIRONMENT
2. protects worm from DESSICATION
3. DOUBLE CUTICLE
4. UNABLE TO EAT so NEEDS TO LIVE OFF OF ENERGY RESERVES

17
Q

ASCARIDS…
how QUICKLY can eggs become INFECTIVE? under WHAT conditions?
eggs are covered by WHAT?
can be TRANSMITTED via WHAT kind of host?
does NOT cause what 2 kinds of infections?
where do L3s inside eggs HATCH? (2 locations)

A

eggs can become infective in 2 WEEKS UNDER FAVORABLE CONDITIONS

eggs are covered with STICKY, PROTEIN COATING

CAN be transmitted by INGESTION via PARATENIC HOST

DOES NOT CAUSE VERTICAL TRANSMISSION, so doesn’t cause these 2 infections…
1. TRANSPLACENTAL
2. TRANSMAMMARY

L3s hatch in…
1. STOMACH
2. SMALL INTESTINE

18
Q

ID ORGANISM & LESION

what SPECIES of animal is this CHARACTERISTIC in? what sequelae does it cause/when?

A

“MILK SPOT LESIONS” = ASCARIS SUUM

from ASCARID L3 LARVAE penetrating the GUT WALL and MIGRATING TO THE LIVER

IN PIGS, THIS IS A HALLMARK SIGN because it CAUSES VERMINOUS PNEUMONIA ~7 days POST-INFECTION

19
Q

what’s the BIGGEST effect of ASCARIDS on LA/why?

A

ECONOMIC IMPACTS from DECREASED PRODUCTION due to LIVER DYSFUNCTION and INABILITY TO REACH APPROPRIATE WEIGHT

20
Q

ASCARIS SUUM…
most ___& ___ important disease in pigs…
diagnosis is based on what 2 things?
when do the main pathologic events occur?
CHRONIC infection (2)
ACUTE infection (2)
contagion? (2)
treatment options? (1)
PREVENTION?

A

COMMON, ECONOMICALLY IMPORTANT

diagnosis..
1. CLINICAL findings
2. NECROPSY findings

MAIN pathologic events occur DURING THE PRE-PATENT PERIOD (BEFORE 2 MONTHS POST-INFECTION)

CHRONIC infection…
1. ADULT worms
2. POOR FEED CONVERSION/ECONOMIC IMPACT

ACUTE infection…
1. LARVAL worms
2. ACUTE RESPIRATORY DZ

contagion?
1. ZOONOTIC!
2. not NECESSARILY pathologic in humans though

treatment options?
1. PYRANTEL = best for when larvae are ACTIVE

prevention?
–> TREAT/CLEAN sows with SOAP and WARM WATER ~2 WEEKS BEFORE FARROWING

21
Q

PARASCARIS…
in WHAT species?
2 organisms?
most important parasite in ___
what stage/manifestation is the MOST dangerous? 4 c/s associated?

A

in HORSES

2 organisms…
1. P. EQUORUM
2. P. UNIVALENS

most important parasite in FOALS

ADULT WORMS IN SMALL INTESTINE…
1. GUT MOTILITY
2. INTUSSUSCEPTION
3. IMPACTIONS
4. RUPTURE OF SI

22
Q

ID PARASITE in SI of FOAL

A

PARASCARIS

23
Q

FASCIOLA HEPATICA…
“common name?”
what type of parasite is this?
morphology? (2)
describe eggs (3)
LOCATION?
what are the 6 definitive hosts? (which is most common?)
INTERMEDIATE hosts?

A

“LIVER FLUKE”

this is a GI TREMATODE

morphology?
1. LARGE, LEAF-SHAPED worm
2. have “SHOULDERS” = CEPHALIC CONE

EGGS…
1. DENSE
2. GOLDEN to DARK-BROWN
3. have OPERCULUM at one end

location = BILE DUCTS

definitive hosts?
1. RUMINANTS MOST COMMON
2. camelids
3. pigs
4. horse
5. rabbit
6. HUMANS (zoonotic)

intermediate hosts? = AQUATIC SNAILS

24
Q

ID PARASITE

A

FASCIOLA HEPATICA “liver fluke”

25
Q

ID PARASITE EGG

A

FASCIOLA HEPATICA

26
Q

LIFE CYCLE for FASCIOLA HEPATICA…
what TYPE of life cycle?
where are eggs shed/where MUST they go?
what is the INFECTIVE stage?
four stages?

A

INDIRECT life cycle

eggs SHED IN FECES & MUST GO IN WATER

infective stage? = METACERCARIAE (on plants)

stages?
1. after eggs get in water, MIRACIDIUM (larvae) hatches, swims, and PENETRATES AQUATIC SNAIL
–> can take WEEKS to MONTHS depending on water temp

  1. in SNAIL, MULTIPLIES THROUGH 1 GEN of SPOROCYSTS and 2 GENS of REDIAE
  2. REDIAE produce FREE-SWIMMING CERCARIAE that LEAVE SNAIL, swim & ENCYST as METACERCARIA on vegetation
  3. animals become INFECTED WHEN THEY INGEST AQUATIC PLANTS w/ METACERCARIAE
27
Q

PATHOGENESIS of FASCIOLA HEPATICA…
SUBCLINICAL dz? (3)
CHRONIC dz? (4 + special finding in CATTLE)
ACUTE dz? (more common in what age animal, 3 c/s, + secondary infections?)
Tx? (2, drug and prevention)

A

SUBCLINICAL dz…
1. decreased production
2. weight loss
3. poor growth

CHRONIC dz…
1. GRADUAL loss of condition
2. progressive weakness
3. ANEMIA
4. HYPOPROTEINEMIA
+ in CATTLE, bile ducts become CALCIFIED –> “CLAY-PIPE” or “PIPE-STEM” LIVER

ACUTE dz = MORE COMMON IN CALVES
1. tissue damage
2. inflammatory reaction
3. death
+ CLOSTRIDIUM NOVYI can cause FATAL DZ (big head) in SHEEP

treatment?
1. CLORSULON in the US
2. SNAIL CONTROL?

28
Q

TAPEWORMS…
what type of parasite is this?
what 2 SPECIES do they present in w/ names?
commonality/pathogenicity?
morphology? (3, including egg)
TYPE of life cycle?
infective stage?
infection occurs from….
PPP?

A

GI CESTODES!

what 2 SPECIES do they present w/ names?
1. HORSES = anoplocephala
2. RUMINANTS = moniezia & thyanosoma

they are COMMON and usually NON-PATHOGENIC

morphology?
1. SCOLEX DOES NOT HAVE ROSTELLUM or HOOKS
2. GRAVID segments are WIDER than they are LONG
3. egg has PYRIFORM APPARATUS

INDIRECT life cycle!

infection occurs from INCIDENTAL INGESTION OF CYSTICEROIDS present in FORAGE

PPP = 4-6 weeks

29
Q

ANOPLOCEPHALA spp…
what type of parasite is this? (2 names)
what SPECIES does this infect?
4 gross presentations?
diagnosis?
2 reasons we don’t use fecal tests for diagnosis?

A

GI CESTODE/TAPEWORMS

infects HORSES

3 gross presentations?
1. EROSIONS at ATTACHMENT SITES (ileocecal junction)
2. INTUSSUSCEPTIONS
3. IMPACTIONS

diagnosis? = via TAPEWORM ELISA TEST (serum & saliva)

2 reasons we don’t use fecal tests for diagnosis?
1. eggs are shed IRREGULARLY
2. PROGLOTTIDS NOT SEEN IN FECES

30
Q

2 names for ORGANISM eggs?

what is CHARACTERISTIC of eggs for this organism?

A

GI CESTODE/TAPEWORM

eggs have PYRIFORM APPARATUS

31
Q

MONIEZIA spp…
what type of parasite is this? (2 names)
clinical significance? (2)
diagnosis? (2)
2 drug treatments?

A

GI CESTODES/TAPEWORM

clinical significance?
1. OVERALL, LITTLE TO NONE
2. MAYBE heavy infection can cause REDUCED GROWTH IN YOUNG ANIMALS

diagnosis?
1. detection of EGGS in FECAL FLOATS (McMaser)
2. PROGLOTTIDS PRESENT IN FECES

Tx?
1. PYRANTEL
2. PRAZIQUANTEL

32
Q

GI PROTOZOA…
what TYPE of parasites are these? (3 descriptors)
2 example organisms? what are they BOTH?
how do they cause disease?
how do they reproduce?
how is it TRANSMITTED?

A

= OBLIGATE INTRACELLULAR PARASITES

2 examples?
1. EIMERIA
2. CRYPTOSPORIDIUM
–> BOTH COCCIDIA

how do they cause disease?
= by DESTROYING HOST INTESTINAL CELLS

reproduction?
= SEXUAL & ASEXUAL PHASES of MULTIPLICATION & DEVELOPMENT

transmission?
= via FECAL-ORAL ROUTE & INGESTION of SPORULATED OOCYST (infective stage)

33
Q

ID PARASITE & the STRUCTURE on the egg on the right?

A

GI PROTOZOA/EIMERIA

right = CAP covers the MICROPYLE (upper end of oocyst)

34
Q

EIMERIA spp…
what TYPE of parasite is this?
what ORGANISMS does this infect?
SCHIZOGONY = what is it & where does it happen?
GAMETOGONY = what is it & where does it happen?
SPOROGONY = what is it & where does it happen
it’s _____-specific
MAIN clinical sign & 3 subs?

A

GI PROTOZOA

INFECTS RUMINANTS

SCHIZOGONY = ASEXUAL MULTIPLICATION in SI

GAMETOGONY = SEXUAL MULTIPLICATION in LI

SPOROGONY = shed OOCYSTS in FECES to SPORULATE IN ENVIRONMENT

it’s HOST-SPECIFIC

MAIN clinical sign = DESTRUCTION OF INTESTINAL MUCOSA
1. enteritis
2. D+ (hemorrhagic or not)
3. wt loss/dec appetite

35
Q

CRYPTOSPORIDIUM…
what type of parasite is this?
contagion?
usually ____-____
what AGE of BOVINES does it infect?
2 clinical signs?

A

GI PROTOZOA

POTENTIALLY ZOONOTIC!

usually SELF-LIMITING

CALVES NEONATAL –> 1 MONTH MOST AFFECTED

2 clinical signs?
1. PUTTY-LIKE FECES (yellow to white)
2. PROFUSE WATERY, EXPLOSIVE D+

Year 1/2 (139 decks)

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