Kapitel 88 – Salivary glands Flashcards

1
Q

Which are the major salivary glands in cats and dogs?

A

a. Parotid
b. Mandibular
c. Sublingual
d. Zygomatic

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2
Q

What surrounding structures makes dissection of the parotid gland challenging?

A

a. The facial nerve
b. Maxillary and temporal arteries
c. Internal maxillary vein
d. and regions surrounding the external acoustic meatus and the stylomastoid foramen

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3
Q

where does the parotid gland open?

A

It opens into the oral cavity through a small papilla at the level of the upper fourth premolar.

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4
Q

What is the main blood supply to the parotid gland?

A

parotid artery, a branch of the external carotid artery

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5
Q

Where does the major zygomatic duct drain?

A

It opens into the oral cavity at the caudolateral aspect of the last upper molar. Typically the zygomatic papilla is approximately 1 cm caudal to the parotid salivary papilla.

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6
Q

Zygomatic salivary gland:
A) Which artery supply,
B) which vein drain
C) where does its lymphatics drain?

A

a. Infraorbital artery
b. Deep facial vein
c. Medial retropharyngeal lnn

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7
Q

Where does the lymphatics of the mandibular salivary gland drain?

A

Medial retropharyngeal lnn

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8
Q

What are the two phases of salivary production?

A

Phase 1: production of saliva by the acinar cells. They absorb Na creating a sodium rich saliva

Phase 2: begins when salive have arrived in the collecting ducts with active reabsorption of Na+ and secretion of HCO3− and K+ –> The resultant saliva excreted into the oral cavity is rich in HCO3− and K+

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9
Q

How does the parasympathetic nervous system increase production of saliva?

A

a. Vasodilatation of the blood supply
b. Stimulation of cyclic guanosine monophosphate (cGMP), which directly upregulates the activity of the acinar cell.

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10
Q

What are examples of nonsurgical salivary disease?

A

a. Sialadenosis (noninflammatory, nonneoplastic, bilateral swelling of the salivary glands, most commonly affecting the mandibular salivary glands)

b. Noninfectious Sialadenitis and Necrotizing Sialometaplasia

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11
Q

What is the treatment for sialadenosis?

A

a. Phenobarbital

(Clinical signs are not alleviated by glucocorticoids, antibiotics, or surgical removal of the affected gland(s). A response to phenobarbital and detection of abnormalities on electroencephalography may support a diagnosis of limbic epilepsy. Recurrence of clinical signs after cessation of medical management is possible; therefore lifelong phenobarbital therapy may be required)

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12
Q

What additional clinical signs can be seen in sialadenitis/necrotizing sialometaplasia compared to sialodenosis?

A

a. Exophthalmos (zygomatic sialocele
b. labored breathing (pharyngeal sialocele)
c. dysphagia (sublingual sialocele or ranula)
d. Intermandibular or cranioventral cervical swelling (cervical sialocele, most common)

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13
Q

Which salivary duct is most commonly accossiated with sialoliths?

A

Parotid duct

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14
Q

What are the most common primary salivary gland tumours?

A

Adenocarcinoma and acinar cell carcinoma

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15
Q

What additional clinical signs can be seen in sialadenitis/necrotizing sialometaplasia compared to sialodenosis?

A

Pain upon palpation and vomiting

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16
Q

What are the vascular landmarks for the location of the mandibular salivary gland during surgery?

A

External jugular bifurcation; the mandibular gland sits at or just cranial to this bifurcation

17
Q

What nerve serves as a landmark during sublingual salivary gland dissection?

A

The lingual nerve

18
Q

What is a common complication of removal of the parotid salivary gland?

A

Facial nerve paresis or paralysis.