Clinical Skills - Spinal Disorders Flashcards
Spinal hx
Pain hx incl stiffness Red flags for bone pain Long tract sytptoms Nerve root irritation Cauda equina symptoms
Long tract symptoms atoms
Is pathology from within spine itself
Nerve root irritation
Compression of peripheral nerve outside spinal cord
Characteristics of nerve root irritation
Referred pain
Worse on sneezing and coughing (increases pressure around nerve root)
Positional
How do we divide spine examinations
Cervical
Thoracic
Lumbar
Cervical spine examination outline
Look - standing
Feel
Move
Neurological exam
Cervical spine - look
Deformity - kyphosis/ scoliosis Scars Swelling Muscle wasting - trapezius, sternocleidomastoid Gait
Kyphosis
Question mark sign
Scoliosis
S-shape
Cervical spine - feel
Spinous process of 7th cervical vertebra
Spinous process of other vertebrae - alignment,m irrregulatitesd, tenderness
Paraspinal muscles - tenderness, spasm
Trapezius - Tenderness, spasm
Cervical spine - move
Flexion
Extension
Lateral flexion
Rotation
Testing flexion of cervical spine
‘Look down at toes’
Testing extension of cervical spine
‘Look up at ceiling’
Testing lateral flexion of cervical spine
“Take your left ear to your left shoulder …”
Testing rotation of rotation
‘Look over left shoulder, look over right shoulder”
Neurological exam for cervical spine
Upper Limb neurology
- Tone
- Power
- Reflexes (triceps, biceps, supinator)
- Sensation
What does power test for
Motor supply
What does sensation test for
Sensory supply
Outline of thoracic spine exam
Look
Feel
Move
Thoracic spine - look
Deformity
Scars
Wasting of paraspinal muscles
Thoracic spine - feel
Palpate spine processes (T1-12) - alignment, irregularities, tenderness
Paraspinal muscles - tenderness, spasm
Thoracic spine - move
Rotation examined from behind, with patient seated
Examining rotation - thoracic spine
“Cross your arms over your chest, now turn your body as far to the right as you can”
Outline of lumbar spine - lumbar spine
Look
Feel
Move
Neurological exam
Lumbar spine - look
Look from anteriorly, laterally and posteriorly at skin – colours, scars
- Anteriorly: ASIS, muscle bulk, pelvis-leg angle
- Laterally: lumbar lordosis, kyphosis, muscle bulk, pelvic tilt
- Posteriorly – spinal stenosis, posterior iliac spine, dimples of Venus
Ask pt to walk - gait
Lumbar spine - feel
Vertebral spine - alignment, tenderness, tenderness
Paravertebral muscles - tenderness, spasm
SI joint
ASIS
PSIS
Height of iliac crest
Lumbar spine - move
Flexion
Extension
Lateral flexion
Testing flexion at thoracic spine
“Please bend over to touch your toes”
Testing extension at thoracic spine
“Lean backwards as far as you are able”
Testing lateral flexion at thoracic spine
“With your right hand reach down your right leg as far as possible”
Neurological exam - lumbar spine
Schober’s test
Sciatic stretch test
Femoral stretch test
Neurology in lower limb - tone, power, reflex, sensation
Spine - feel
Spinour processes - alignment, tenderness, irregularities Paraspinal muscles Trapezius SI joint ASIS and PSIS Height of iliac crest
Spine - move
Cervical flexion, extension, lateral flexion, rotation
Thoracic rotation
Lumbar flexion, extension, lateral flexion
What is the Schober’s test for
Tests that flexion is not coming from hips with rigid spine
Performing Schober’s test
Identify dimples of Venud
Mark skin 5cm above and 10cm below this
Place tape measure with 0cm on the bottom mark
Note change between 2 higher marks (Should be >5cm)
Performing sciatic stretch test
Lie pt. supine and examine hip movement Straight leg raise (good side first) Note angle at which referred pain occurs Lower leg by 5-10 degrees Extend/ dorsiflex ankle Confirm the distribution of the pain
Performing femoral stretch test
Exclude fixed flexion of the hip
Lie the pt. prone
Slowly flex knee (can extend the hip with the knee in flexion)
Confirm the distribution of the pain, usually pain down front of thigh
Testing dermatomes
Using cotton wool for light touch – drag cotton ball all along limb to make sure you don’t miss a patch of numbness at least 15-20x
Use neurotip injection pen
Upper limb reflexes
Biceps (C5-6)
Brachioradialis (C5-6)
Triceps (C7)
Lower limb reflexes
Knee (L3-4)
Ankle (S1)
Non-infl back pain
Mechanical/ low back pain +/- sciatica OA Spinal stenosis Spondylolisthesis Scoliosis Vertebral fracture
Infl back pain
Infection e.g. disciitis, osteomyelitis, abscess
AxSpA
Malignancy
Other terms for back pain
Discogenic pain Degenerative disc disease
Lumbar disc herniation
Secondary to lumbar degenerative disease
Facet joint pain
Other terms for sciatica
Sciatica/ lumbago Radicular pain/ radiculopathy Pain radiating to the leg Nerve root compression/ irritation Neurogenic claudication Spinal stenosis
Epidemiology of MBP
Prevalence and burden increases with age until around 6th decade
Prevalence of back pain is more common in women and increases w/ age, peaking around 7th decade
Principles of back pain assessment
Symptoms Assess if nerve root irritation is present Nerve root irritation tests Document neurological signs Excl cauda equina syndrome
Clinical features of MBP
Exact cause rarely identifiable: ligaments, muscles, fascia, bursae, facet joints, vertebral discs, SI joints Onset 20-55 yrs. Lumbosacral, buttocks and thighs Pain worse towards end of day Pt is well
What % of back pain is caused by MBP
90
Prognosis for MBP
Good
50% of pts better within a week
90% better within 6 weeks
Recurrence of mechanical pain
60% will have a recurrence within 1 year
Recurrent attacks tend to settle within 3-5 yrs.
Peaks in middle decades and becomes less frequent in later life
Features of nerve root pain
Unilateral leg pain > back pain
Radiation below knee
Numbness and paraesthesia
Nerve irritation signs
Nerve root pain in only one nerve root
Motor, sensory or reflex change
Nerve roots usually affected
83% of prolapsed intervertebral discs will involve L5 (51%) or S1 (22%) roots
L5 and S1; 10%
L3 or L4: 17% (usually elderly)
+ve sciatic stretch test
Pain behind knee down to heel between 30-70+ degrees
Motor signs for L5 pathology
Weak dorsiflexion big toe
Weak dorsiflexion lateral 4 toes
Weak eversion
Motor signs for S1 pathology
Absent ankle jerk
Weak gluteal contraction *
Weak knee flexion Weakness toe plantar flexion *
* do not occur without absent ankle jerk
Movement affected by L2 pathology
Hip flexion/ abduction
Movement affected by L3 pathology
Hip adduction
Knee extension
Tendon reflex affected by L3 pathology
Knee jerk
Movement affected by L4 pathology
Knee extension
Foot inversion/ dorsiflexion
Tendon reflex affected by L4 pathology
Knee jerk
Movement affected by L5 pathology
Hip extension/ abduction
Knee flexion
Foot/ toe dorsiflexion
Movement affected by S1 pathology
Knee flexion
Foot/toe plantar flexion
Foot eversion
Tendon reflex affected by S1 pathology
Ankle jerk
Epidemiology of sciatica
Sciatica has a lifetime incidence ranging from 13 to 40%
The incidence is related to age – rarely seen before the age of 20, incidence peaks in 5th decade and then declines
Modifiable factors associated w/ a 1st onset of sciatica
Smoking
Obesity
Occupational factors
General health status
Prognosis for nerve root pain
50% of pts with root pain are better within 6 weeks – self-limiting
NICE recommendations around back pain
Examine pt.
Do not refer for investigations unless high risk of poor outcome
Imaging in specialist setting of care only if result Is likely to change management
NICE recommendations about treatment for back pain
Educate to self-manage pain and encourage normal activities
Consider a group exercise programme
Consider manual therapy
Consider psychological approaches
Give analgesia
Promote and facilitate return to work or normal activities of daily living
Manual therapy for back pain
Spinal manipulation
Mobilisation
Soft tissue technologies such as massage
Analgesia for back pain
Oral NSAIDS and weak opioids (w/or w/out paracetamol)
What does NICE say not to recommend
Belts or corsets Foot orthotics Traction Acupuncture USS, TENS, interferential therapy Paracetamol alone, opioids, antidepressants or anticonvulsants
NICE approved interventions
Radio-frequency denervation
Epidural/ nerve root injection
Spinal fusion
Sacral epidural
Needle goes through one of the holes in the sacrum and is injected with combination of local anaesthetic and steroid
Blind injection
Nerve root block
Radiologist does CT scan and injects around inflamed nerve root
Why recommend physical activity for back pain?
Rest perpetuates disability
May relieve venous congestion and oedema Muscular afferent activity may interfere with pain signal processing
Spinal movement may have a similar effect
Precise form of exercise seems unimportant
What do we do for the 10% of pts with back pain that aren’t better at 6 weeks
Biological assessment
Physchcological assess,mt
Social assessmnet
Biological assessment for back pain
Nerve root problems
Red flags
Check CRP/ L spine x-ray if relevant
Psychological assessment for back pain
Unjustified fears?
Depressed?
Social assessment for back pain
Family relationships
Work problems
Risk factors for chronic
Previous hx of back pain Previous time off work/radicular pain Unfit Poor general health Smoking Depression/ anxiety Disproportionate pain behaviour Personal problems Medicolegal proceedings
Red flags for back pain
Malignancy Corticosteroids Pt systematically unwell Wt. loss Widespread neurology Age <20 yrs. or >55yrs Violent trauma Constant, progressive, non-mechanical back pain Thoracic pain IV drug abuse/ HIV infection Persisting severe restriction of lumbar flexion Structural deformity
Cauda equina syndrome
Large central disc herniation compressing cauda equina (also tumours/ abscesses)
Symptoms of cauda equina
Bilateral sciatica
Urinary/ faecal incontinence
Saddle anaesthesia
Widespread (>one nerve root) or weakness in legs
O/E for cauda equina
Rectal examination reveals reduced tone
OA of the spine
Incl facet OA
Typically pain is most pronounced in morning and then recurs as joint has been stressed w/ exercise/weight-bearing
Who does spinal stenosis affect mostly
Elderly pt
Spinals stenosis characteristics
LBP radiation to the legs w/ exercise
Worst after exertion and standing
Relieved by rest over 10 mins or so
Relieved by bending forward
Spondylosis
Defect in pars intra-articular, usually 5th neural arch
Usually asymptomatic
Can be associated with LBP
What is spondylosis related to
Sport in teenage years
Dx of spondylosis
Oblique plain radiographs (MRI if neurological symptoms)
Treatment for spondylosis
Conservative
Spondylolisthesis
Spontaneous displacement of a vertebral body in relation to the vertebral body directly beneath it
Usually displaced in an anterior direction
Neurological involvement can occur (less likely w/ OA
Causes of spondylolisthesis
Spondylolysis
Congenital malformation
Facet joint OA
Treatment for spondylolisthesis
Conservative, rarely spinal fusion
Features of AxSpA as a cause of back pain
Synovitis
Enthesitis
Ossification of enthesis esp. the spine
HLA B27 association
Infections causing back pain
Disciitis
Osteomyelitis
Epidural abscess
Spread of vertebral osteomyelitis and disciitis
Haematogenous
Orig infection may not be identified
Clinical features of vertebral osteomyelitis and disciitis
Insidious onset of pain
Spinal tenderness
15% have symptoms and signs of nerve root compression
Fever in less than 50%
Ix of spinal infection
Infl markers Blood cultures MR Do whole spine MRI Other levels involved in up to 10%
Why do we not regularly culture spine biopsy
Difficult to get into disc
Low yield of 50%
Commonest bugs in spinal infection
Staph aureus and coagulase -ve staph
Referred pain
Pain arising or occurring in a region of the body innervated by nerves other than those innervating source of pain (allows us to distinguish two locations)
IASP definition of referred pain
Pain located in an area with cutaneous innervation that differs from that overlying the site of pathology
Based on dermatomes
Main regions activated in response to acute nociceptive stimulation
Spinal cord Thalamus S1 and S2 Insula Anterior cingulate cortex Prefrontal cortex
Pain pathways
Noiciceptive info enter spinal cords and follows contralateral ascending pathways – e.g. spinothalamic tract
How does spatial info on pain reach S1 cortex
Thalamus
Where does emotional interpretation about pain occur
Via limbic system
Where is sensory info about pain processed
S1 sensory cortex
Which division of the body is related to radicular referred pain
Dermatomes
Which division of the body is related to myofascial referred pain
Myotomes
Which division of the body is related to bone/joint referred pain
Sclerotomes
Which division of the body is related to visceral referred pain
Viscerotomes
Neuropathic pain
Pathological activation in neurones in dorsal root ganglion
Nociceptor terminals not activated
May involve all fibres: A-alpha, A-beta, A-delta, C
Features of radicular referred pain
Neuropathic pain
Nerve root tension signs
Sensory, motor, reflex abnormality
Dermatomal representation of pan from nerve root infl
How does the brain interpret radicular referred pain
S1 cortex interprets pain is segmental nerve root input Pain ‘projected’ and referred to dermatome
Somatic referred pain
Activation of those nerves distal to dorsal root ganglion and nerve rot may refer to dermatormal segment
Convergent input to dorsal horn at segmental level
Somatic muscular referred pain - gluteus minimus
Innervate by Sup. Gluteal n.
Pain in L5, S1, S2
Somatic muscular referred pain - tibialis anterior
Innervated by deep peroneal n
Pain in L4, L5
Somatic muscular referred pain - supraspinatus
Pain in C5, C6
Why do pt’s w/ hip OA get knee x-rays
T2, L1 (cutaneous)
L2,3,4 lumbar plexus (hip flexors/ capsule)
L5,S1 sciatic (acetabular)
Hilton’s Law
Hilton’s law
A nerve supplying a muscle controlling a joint also innervates the joint
Why do pts w/ sacroilitis get a lumbar MRI scan
Innervation – L4/5, S1/2/3
Dorsal Horn convergence
Any afferent input to that spinal segment may refer to the dermatome
- Visceral input via visceral ‘SNS’ autonomic fibres
- Muscle via nociceptive afferents that run with motor nerve
- Joint via nociceptive afferents from joint structures
How is pain localised in the brain
According to a somatotopic map on S1 sensory cortex
What do spinal segmental levels map to
Body surface dermatomes which also corresponds to S1 cortex representation of body surface
Red flags for back pain- spinal fracture
Sudden onset of severe central spinal pain which is relived by lying down
People with osteoporosis or those using corticosteroids
Structural deformity of the spine (e.g. a step from one vertebra to an adjacent one) may be present
There may be point tenderness over a vertebral body
Red flags for back pain - malignancy
50+ yrs. or <20yrs
Gradual onset of symptoms
Severe unremitting pain that remains when pt. is supine, aching night pain that prevents or disturbs sleep, pain aggravated by straining e.g. at stool or coughing and thoracic pain
Localised spine tenderness
No symptomatic improvement after 4-6 weeks of conservative LBP therapy
Unexplained wt. loss
Past hx of cancer
Cancers most likely to metastasise to spine
Breast Lung GI Prostate Renal Thyroid
Red flags for back pain - infection
Fever TB or recent UTI Diabetes Hx of iv drug use HIV infection, use of immunosuppressants or the pt. is immunocompromised
Pain management jigsaw
Interventions Medication Relaxation Complimentary therapies Psychology Neuromodulation exercise Lifestyle changes/ coping strategies
Pt assessment for back pain - subjective
HPC, pain patterns, descriptors of pain PMH Previous treatment Medication use Activity patterns A real clear picture of what pain means to the pt.
Pt assessments for back pain - objective
Physical assessment/ spp to individual pt.
Exercise/activity and persistent pain
Pt education Goals and education Look at function/ meaningful activities Parameters of exercise – stretching, strength, aerobic Start with an achievable level Gradual increments – (0-20%)
Why do psychological factors affect recovery?
Affect everyday behaviour and actions
Chronic pain and mental health
Pain can affect mental health issues, but mental health can exacerbate pain and associated with poorer outcomes
Higher risk of suicide
More likely to have lower wellbeing scores
Psychosocial assessment for back pain
Attitudes – towards the current position
Beliefs – bout the pain
Compensation – is the pt. awaiting payment for an accident/ injury
Diagnosis – iatrogenesis and miscommunication
Behavioural assessment for back pain
Impact on quality of life – daily functioning – changes in their social, occupational and physical activities/ sleep
Interactions w/ health service
Coping strategies – exacerbating and relieving factors
Use of passive coping e.g. alcohol, inactivity
What is pain management about
Improve function QoL
Helping people to learn how to manage their experience of pain and associates distress in helpful ways
NOT about ‘curing pain’
Pts taking more ACTIVE approach and decreasing reliance on healthcare and analgesic medications
Medication and back pain
In the absence of red flag pathology, very little evidence for benefit of medication for back pain
High risk of long-term dependence w/. short acting opiates
Anti- infl may benefit for short term use
PMP
Patients offered 10 x 3.5hrs sessions
Timetable includes a weekly practical exercise session, a review of personal value-based goals and a psychology session.
Qualitative outcomes focused on patient’s own values and goals rather than reduction in measured / VAS pain scores
What do we operate on the spine for
Trauma Degenerative condns Infections Deformities Tumours
Triad of epidural access
Fever
Back pain
Neurological deficit
Degenerative condns of spine
Claudication Spinal stenosis Disc prolapse Spondylolisthesis Sciatica
Reasons for spine re-operation
Sagittal balance problem Metal work Impingement on nerve roots Pseudoarthrosis and failure Infection
Non-surgical treatment for spinal issues
Injections
Ablation
Cord stimulation
Materials used in spinal surgery
Pedicle screws and rods
Disc replacements
Evolution of spinal fixation techniques
Minimally invasive spine surgery
Endoscopic spine surgery
Computer navigated spine surgery
Robotic spine surgery
Co-morbidities for spinal degenerative condns
HTN
OS
RhA
DM
Convergence- projection theory
Referred pain is due to parts of the brain being unable to differentiate noxious stimuli from visceral nociceptors vs. somatic nociceptors
This is because the somatic and visceral inputs converge at the same spinal level
Somatic vs visceral
Somatic = skin, muscles + soft tissue, visceral = internal organs
Criteria for infl back pain
Chronic back pain > 3 months with onset before 45
Morning stiffness for > 30 mins
Back pain awakens patient in second half of the night
Alternating buttock pain
2/4 needed for dx
Conservative treatment for sciatica
Anti - infl
Muscle relaxants
Physiotherapy
Analgesia
Majority of pt’s settle within 6 weeks
Investigation if symptoms of sciatica fail to settle after 6 weeks
MRI of spine - identify if there is a disc bulge and any possible impingement
What can cause vertebral crush fractures
Osteoporosis
Metastasis
Contraindications of MRI
Pacemakers
Aneurysm clips
Ocular metallic foreign body
Claustrophobia