6/5/13 Flashcards

1
Q

What is the mechanism of α-blockers in benign prostatic hyperplasia?

A

relax urethral sphincter

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2
Q

What are the best hypertensive choices for pts w/ chronic kidney failure?

A

ACE-Is and ARBs

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3
Q

What causes black pigment stones in the gallbladder?

A

hemolysis

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4
Q

What causes brown pigment stones in the gallbladder?

A

chronic infection, esp. parasitic

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5
Q

In any acute spinal injury or compression of any process, the first intervention should be ______.

A

hi-dose IV steroids

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6
Q

What is the next best step for suspected cord compression after steroids?

A

MRI

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7
Q

How long is the max. delay allowed for choosing PCI over thrombolytics for AMI?

A

90 mins

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8
Q

What is the inheritance pattern of hereditary spherocytosis?

A

AD

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9
Q

What vitamin should be supplemented in pts w/ hereditary spherocytosis?

A

folate

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10
Q

What is the best medication for slowing the progression of diabetic nephropathy in all pts, whether or not they have htn?

A

ACE-Is

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11
Q

What is the major cause of mortality in TCA overdose?

A

hypotension

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12
Q

What should be administered to pts of TCA overdose to combat hypotension, prolonged QT interval, and arrhythmia?

A

sodium bicarb

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13
Q

What should be infused into pts suffering from torsade de pointes?

A

Mg sulfate

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14
Q

What is the initial treatment of anal fissure?

A

dietary modification, stool softeners, and local anesthetic

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15
Q

Define “salvage therapy”.

A

A form of therapy used after standard therapy as failed.

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16
Q

How is induction therapy different from neoadjuvant?

A

induction may or may not be followed by another tx (e.g. surgery)

17
Q

What is the other name of herceptin, the monoclonal Ab that targets the HER2 receptor?

A

trastuzumab

18
Q

What is resistance, a psychological defense mechanism?

A

refusal to bring an unpleasant unconscious thought to awareness

19
Q

What electrolyte abnormality is a common complication of subarachnoid hemorrhage?

A

hyponatremia - “cerebral salt-wasting syndrome”

20
Q

What are the two main mediators of cerebral salt-wasting syndrome following SAH?

A

vasopressin and Atrial/brain natriuretic peptide