Nutrition and GI: Rumen Acidosis Flashcards

1
Q

What causes rumen acidosis?

A
  • Excessive concentrates- rapid digestion
  • Insufficient saliva- reduced saliva flow

Decreased pH rumen
D lactic acid builds up- cannot be metabolised

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2
Q
  1. What should the rumen pH be?
  2. What is the problem with low pH?
A
  1. 6-7
  2. Problems with Low pH
    * Kills fermentation bacteria
    * Encourages- lactobacilli
    * Efficiency of digestion falls
    * Undigested particles pass through- hind gut fermentation, osmotic diarrhoea
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3
Q

What factors affect rumen pH?

A
  • VFAs produced
  • Type of acid- lactic
  • Rate of fermentation
  • Rate of acid remova- rumen papillae
  • Buffering by saliva
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4
Q

Why is saliva so important?

A

Produced when chewing long fibre- cudding
Contains sodium bicarbonate
70% cows should be cudding at any time

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5
Q

What does long fibre do?

A

Encourages cuffing- bicarb buffer
Forms a rumen mat
* Keeps food particles in rumen to be digested
* Home to bugs- biofilms

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6
Q

What are risk factors for SARA?

A
  • Insufficient SARA
  • Inaccurate fodder DM estimation- insufficient fodder provided
  • Overmixing of TMR
  • Excessive feeding of sugars and starches
  • Poor dry cow managment
  • Slug feeding of concentrates in the parlour
  • Food deprivation and irregular feeding
  • Poor cow comfort
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7
Q

What is SARA?

A

Sub-acute rumen acidosis
Effect of large concentrate feeds

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8
Q

What are the signs of SARA?

A
  • Herd problem
  • pH < 5.5
  • Most common nutritional disorder
  • Energy deficit
  • Overall poor health
  • Faces- loose and soft, swishing tails, undigested grains, long fibre present
  • Lower cleanliness score

Score faeces 1-5- 3 ideal

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9
Q

What are the effects of SARA?

A
  • Reduced DMI
  • Reduced digestability- reduced energy intake, NEB
  • Immunosuppression- disease susceptibility
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10
Q

What are the production effects of SARA?

A

Poor yields
* poor peak yields
* Decline in yields

Milk quality- sometimes

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11
Q

What are the health effects of SARA?

A
  • Displaced abomasum- VFAs enter abomasum- atony
  • Digestive upsets
  • Ketosis- NEB
  • Lameness- sub clinical laminities (ulcers, WLL)
  • Mastitis- dirty cows
  • Poor resistance and health
  • Infections- endocarditis
  • Fertility- not seen bulling
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12
Q

How is SARA diagnosed?

A

Clinical signs
* Fertility
* Lameness
* Ketosis
* LDA
* Faeces
* ‘Odd sick cows’

Observe group
* Cudding
* rumen fill
* tail swishing
* dirt score

Faeces- score 1-5, sieve

History- nutritional managment

Measure rumen pH

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13
Q

What is the maximum length of fibre in faeces?

A

No longer then 1/2 inch long

No or little undigested grain

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14
Q

What causes mucus casts with SARA?

A
  • When undigested particles pass through rumen causes hindgut fermentation
  • Leads to colonic acidosis- damage to colon wall
  • Fibrin casts in faeces
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15
Q

How should cows be selected for sampling of rumen pH?

When should the cows be sampled?

A

Cows calved 14-21 days-
* still adapting to the tation
* DMI is not yet maximal
* Assess transition and early managment

Cows calved 60-80 days
* Should have adapted to ration
* Maximal DMI
* Assess overall diet quality

Sample both groups 2-4 hours after feeding
Record parlour cake fed

DIAGNOSIS- confirmted when 2 cows from either group below threshold
pH < 5.7

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16
Q

How is cows rumen pH sampled?

A
  • Retrain cow- nose and tail
  • Site- level of stiffle and 6-8 inches behind last rib
  • Clip and scrub
  • Local anaesthetic
  • 3-5” needle- 16-18G
  • Read sample immediately
17
Q

What forage actually acts more like a concentrate?

A

Maize

18
Q

What should the ratio of cocentrate to fodder be?

A

Max 60:40

Concentrate to fodder

19
Q

What is acute ruminal acidosis?

A
  • Excessive acid production
  • pH falls below 5.0
  • Lactic acid production predominates
  • ‘lactic acid eating bugs’ killed off
  • Cannot metabolise ‘D’ lactate
20
Q

When does acute ruminal acidosis occur?

A
  • Over eating grain
  • Sudden nitroduction of high levels of grain- barley beef
21
Q

What are the clinical signs of acute ruminal acidosis?

A
  • Distended rumen- bloat
  • Ataxia
  • Diarrhoea- profuse and smelly
  • Depression
  • Recumbency and shock
22
Q

How do cows present when:
1. Mild
2. Sub-acute
3. Acute
4. Peracute

A
  1. Almost normal
  2. Fairly bright, eats, no ataxia
  3. Ataxia, anorexia, dilated pupils
  4. Severe ataxia, recumbency, apparently blind, severe dehydration
23
Q

How is mild, subacute, and peracute acute acidosis treated?

A
  • Mild- give hay and observe
  • Subacute- oral antacids- magnesium hydroxide or carbonate, feed hay
  • Peracute- rumenotomy (empty contents)
    5l 5% sodium bicarb
    Continue with balanced fluids for 12-24 hours

General
* NSAIDs
* ABs
* Calcium borogluconate in peracute
* Restrict water intake for 12-24 hours

24
Q

How is a rumenotomy done?

A
  • Recumbent animal
  • 18 inch wound
  • Pack towels around
  • Open rumen and empty of all contents