Monitoring and Responding Flashcards

1
Q

What are possible causes of bradycardia?

A

Drugs (e.g., alpha 2 agonist, induction agent, opioid)
Toxaemia (endogenous/exogenous)
Vagal stimulation
Heart problem (e.g. sick sinus)
Hypoxia
Hypothermia

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2
Q

What should you check when assessing bradycardia or tachycardia?

A

Determine animal’s normal heart rate & check if it compromises perfusion & oxygen delivery using SpO2% & ABP

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3
Q

2 yr old Frenchie ASA 1, HR 110 bpm prior to surgery for cherry eye

Premed: acepromazine and methadone

Induction: alfaxalone to effect

Maintenance: isoflurane in oxygen, during surgery the HR suddenly drops from 98bpm to 55 bpm

What do you think? What do you do?

A

Cause of bradycardia:
- Could be due to vagal stimulation during surgery, anaesthetic depth, hypothermia, or drug effects (e.g., methadone or acepromazine)

Response:
- Check depth of anaesthesia & reduce if too deep
- Investigate potential vagal stimulation (e.g., surgical manipulation)
- Administer anticholinergic (e.g., atropine or glycopyrrolate) if bradycardia compromises perfusion
- Ensure there is no hypoxia or toxaemia contributing to bradycardia

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4
Q

Cockerpoo, castrate, ASA 1, HR 108 bpm prior to surgery

Premed: dexmedetomidine and methadone

Induction: alfaxalone to effect

Maintenance: isoflurane in oxygen, 5 mins after induction you see this.

What do you think? What do you do?

A

Bradycardia (HR: 60 bpm):
- Likely due to dexmedetomidine (alpha-2 agonist), which reduces sympathetic tone

Perfusion & oxygenation:
- Normal MAP (91 mmHg) & SpO2 (100%) indicate no immediate compromise in perfusion or oxygen delivery

Response:
- Monitor closely: Since perfusion (MAP) & oxygenation (SpO2) are adequate, immediate intervention may not be required
- Reverse alpha-2 effects if needed: If bradycardia worsens or perfusion is compromised, administer atipamezole to reverse dexmedetomidine
- Check anaesthetic depth: Ensure anaesthesia is not excessively deep
- Administer anticholinergics if needed: Consider atropine or glycopyrrolate if perfusion deteriorates

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5
Q

GSD X, TPLO, ASA 2, HR 96bpm prior to surgery

Premed: acepromazine and buprenorphine

Induction: propofol to effect

Maintenance: isoflurane in oxygen, during anaesthesia a gradual decline in HR & ABP

What do you think? What do you do?

A

Bradycardia:
- Likely due to acepromazine (vasodilation) & buprenorphine (opioid effects), combined with propofol & isoflurane, which depress cardiovascular system

Hypotension (MAP 57 mmHg):
- Suggests compromised tissue perfusion & requires intervention to prevent further complications

Response:

  1. Address bradycardia:
    - If perfusion is compromised, consider atropine or glycopyrrolate to increase HR
  2. Manage hypotension:
    - Reduce isoflurane vaporiser settings if anaesthetic depth is excessive
    - Administer IV fluids to improve circulating volume.
    - If needed, use inotropes like dobutamine or vasopressors like norepinephrine to support blood pressure
  3. Monitor closely:
    - Continuously assess ABP, HR, & SpO2 to ensure interventions are effective
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6
Q

TBx, ASA 1, HR 40 bpm prior to surgery, bilat hock arthroscopy

Premed: acepromazine, morphine

Induction: romifidine, ketamine, midazolam

Maintenance: sevoflurane in oxygen 3.1% (ET), 30 minutes later during prepping of the leg, this is the situation

What do you think? What do you do?

A

What’s happening:
- Severe bradycardia likely caused by romifidine (alpha-2 agonist) & morphine effects.
- Hypercapnia (EtCO2 67 mmHg) due to hypoventilation caused by respiratory depression from sevoflurane & sedatives.
- MAP (76 mmHg) acceptable, indicating perfusion is not yet critically compromised

Response:
- Reverse romifidine with atipamezole if needed or use atropine/glycopyrrolate for bradycardia if perfusion worsens
- Ventilate manually or mechanically to address hypercapnia.
- Reduce sevoflurane vaporiser setting to decrease respiratory depression
- Monitor HR, EtCO2 & MAP to ensure stability

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7
Q

Male 6y DSH, HR 90bpm prior to sedation, v large bladder

Premed: methadone

Induction: ketamine, midazolam

Maintenance: isoflurane in oxygen, 0.8% (ET)

What do you think? What do you do?

A

What is happening:
- Bradycardia (HR 75 bpm): Not critical but may be influenced by methadone (opioid) & midazolam, with no immediate compromise to perfusion or oxygen delivery
- Hypocapnia (EtCO2 30 mmHg): Likely caused by hyperventilation (RR 24) & shallow ventilation
- MAP (77 mmHg): Within acceptable range for perfusion.
- SpO2 (92%): Indicates borderline hypoxaemia; possible ventilation-perfusion mismatch

Response:
- Address hypocapnia: Reduce RR if manually ventilating & evaluate anaesthetic depth.
- Improve oxygenation: Adjust fresh gas flow & ensure proper ETT placement.
- Monitor closely: Observe for any further changes in HR, MAP, SpO2 & EtCO2 to maintain stability
- Support perfusion: Administer fluids if indicated to sustain MAP & oxygen delivery

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8
Q

What drug should be used to treat alpha 2 induced bradycardia?

A

atipamezole (alpha 2 antagonist)

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9
Q

What anticholinergics can be used to treat bradycardia?

A

atropine or glycopyrrolate

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10
Q

What are possible causes of tachycardia?

A

Sympathetic stimulation, too ’light’
PaCO2, PaO2, pH abnormalities
CNS disturbances
Low ABP, or cardiac disease
Drugs e.g. anticholinergics

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11
Q

2 yr old Bassett ASA 1, HR 96 bpm prior to surgery for lumpectomy

Premed: dexmedetomidine, methadone

Induction: propofol, local block

Maintenance: isoflurane
2 mins after incision, HR increased

What do you think? What do you do?

A

What’s happening:
- Tachycardia: due to pain or insufficient anaesthetic depth
- Hypotension: Compromised perfusion from vasodilation or sympathetic overdrive.
- SpO2 92%: Borderline hypoxaemia

Response:
- Increase anaesthetic depth or provide additional analgesia (e.g., fentanyl).
- Administer IV fluids & consider vasopressors (e.g. norepinephrine) if hypotension persists
- Verify oxygen delivery & adjust fresh gas flow or ETT placement
- Monitor HR, MAP & SpO2 to ensure stabilisation

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12
Q

14yr old DSH cat chronic renal insufficiency, ASA 3, dental

Premed: midazolam and methadone

Induction: alfaxalone to effect

Maintenance: isoflurane in oxygen

What do you think? What do you do?

A

What’s happening:
- Tachycardia (HR 231 bpm): due to pain, stress, or hypoxia, exacerbated by renal insufficiency
- Hypotension (MAP 56 mmHg): Compromised perfusion from vasodilation or poor cardiac output
- Hypocapnia (EtCO2 24 mmHg): Indicates hyperventilation or poor perfusion
- SpO2 90%: Borderline hypoxaemia, needs addressing.

Response:
- Administer analgesia (e.g., fentanyl) & reassess depth of anaesthesia
- Provide IV fluids & consider vasopressors (e.g. norepinephrine) to improve BP.
- Ensure proper oxygen delivery by adjusting ETT placement or increasing oxygen flow.
- Monitor HR, MAP, SpO2 & EtCO2 for stabilization.

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13
Q

1 year old 2.2kg Dutch rabbit for castration

Premed: buprenorphine SC

Induction: Medetomidine, ketamine SC

What do you think? What do you do?

A

What’s happening:
- Tachycardia (HR 209 bpm): stress or pain response from insufficient analgesia or light anaesthesia.
- Hypocapnia (EtCO2 24 mmHg): due to hyperventilation or poor cardiac output.
- MAP 94 mmHg & SpO2 97%: Perfusion & oxygenation adequate

Response:
- Administer additional analgesia (e.g., fentanyl) to manage pain
- Assess & adjust anaesthetic depth to address potential stress or sympathetic stimulation
- Monitor ventilation to ensure hyperventilation resolves & EtCO2 normalises.
- close monitoring of HR, EtCO2, & MAP for stability.

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14
Q

What’s happening and what is your response?

A

What’s happening:
- Initial tachycardia progressing to bradycardia: due to prolonged anaesthesia, inadequate ventilation, or cardiac strain
- Hypotension (MAP 56 mmHg): decreased perfusion from vasodilation or inadequate cardiac output
- Hypercapnia (EtCO2 54 mmHg): Due to hypoventilation or insufficient respiratory support

Response:
- Address hypotension: Increase IV fluids, consider inotropes (e.g., dobutamine) to support cardiac output.
- Improve ventilation: Increase intermittent positive pressure ventilation (IPPV) to reduce EtCO2 & support oxygenation.
- Adjust anaesthetic depth: Reduce isoflurane concentration to minimize cardiovascular depression.
- Monitor HR, MAP & EtCO2 continuously to ensure stabilisation

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15
Q

What are the key steps in approaching the treatment of tachycardia?

A

Assess if output is being compromised (ABP and SPO2%)
Identify the cause (check anaesthesia depth, CO2 levels etc)
Administer IV opioid, lidocaine or propanolol/esmolol (beta-blockers)
Adjust vaporiser setting to deepen anaesthesia if needed

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16
Q

What are common causes of ventricular ectopic beats

A

Circulating catecholamines (stress)
Hypoxia or hypercapnia
Hypovolaemia or hypotension
Anaesthetic drugs
Myocardial inflammation or stimulation
Trauma
Major organ disease

17
Q

what are ventricular ectopic beats?

A

extra or early heartbeats that occur when the heart’s electrical signals start in the wrong place

18
Q

How are ventricular ectopic beats managed?

A

Assess if output is being compromised (ABP and SPO2%) - danger of ventricular fibrillation?
Identify the cause (check anaesthesia depth, CO2 levels etc)
Administer IV opioid, lidocaine or propanolol/esmolol (beta-blockers)
Adjust vaporiser setting to deepen anaesthesia if needed

19
Q

What are common causes of hypotension in anaesthesia?

A

Anaesthetic drugs
Hypovolaemia and haemorrhage
Cardiac insufficiency

20
Q

How is hypotension in anaesthesia managed?

A

Check patient and attempt to determine cause
Reduce volatile/inhalant agent (TIVA/PIVA)
Increase IVFT (intravenous fluid therapy)
Check urine output if possible
Re-check ABP
Could give crystalloid bolus
Consider use of inotrope (dobutamine)
Consider use of vasopressor (norepinephrine)

21
Q

What are the common causes of hypercapnia in anaesthesia? (increased EtCO2)

A

depth of anaesthesia
hypoventilation
pyrexia
rebreathing
fresh gas flow rates too low
exhaustion of soda lime
changes in dead space

22
Q

Why do cats on non-rebreathing systems often have lower EtCO2 readings?

A

high fresh gas flow (FGF) rate dilutes sampled exhaled CO2, leading to artificially lower ETCO2 readings

23
Q

How can hypercapnia in anaesthesia be treated?

A
  1. Increase ventilation (manual or mechanical)
  2. Check breathing system for rebreathing or soda lime exhaustion
  3. Adjust anaesthetic depth to prevent respiratory depression.
24
Q

What are common causes for hypocapnia in anaesthesia? (decreased EtCO2)

A

Disconnection (check breathing system)
Mis-intubation
Obstruction
Hyperventilation
Circulatory failure
Hypotension
Cardiac arrest

25
Q

How is hypocapnia treated in anaesthesia?

A
  1. Decrease ventilatory rate if hyperventilation is occurring
  2. Check for & resolve airway obstructions or mis-intubation

3.Support perfusion if circulatory failure is identified.

26
Q
A

No, they are typically benign finding & don’t indicate clinical issue

27
Q

How can hypoxaemia be identified in anaesthesia?

A

Palpate a pulse
Check oxygen is connected/ animal is not disconnected
Check the pulse oximeter (move it?, moist gauze) to verify the SPO2%

28
Q

What are common causes of hypoxaemia in anaesthesia?

A

Disconnection
hypoventilation
anaesthetic depth
ventilation-perfusion mismatch

29
Q

How is hypoxaemia treated?

A

Reconnect breathing system if disconnected
Re-intubate if extubated or tube is blocked
Replace the pulse oximeter probe

Reduce the volatile agent
Improve perfusion (IVFT) & inotropes (e.g. dobutamine in equine)

Ventilate the animal if there is hypoventilation/obesity

30
Q

What are common recovery issues after anaesthesia?

A

Dysphoria (unhappiness/distress) - provide analgesia, oxygen or microdose sedation
prolonged recovery - often due to hypothermia