Exam 2 week 8 ppt 8 Intro to Somatic Motor System Flashcards

1
Q

What are motor systems defined as?

A

–All structures of the central and peripheral nervous systems (CNS and PNS) that contribute to motor activity

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2
Q

Define Upper motor neuron

A

–Upper motor neuron

  • Projects to lower motor neuron
  • Located in motor cortex or brainstem
    • Indirectly in Cerebellum & basal nuclei

As discussed previously Upper motor neuron are those motor system neurons that Project to lower motor neurons. These are located in motor cortex, Also as previously discussed upper and lower motor neurons are defined by their connections not their position in the nervous system. For instance upper motor neurons of the pontomedullary junction (vestibular nuclei) can innerate lower motor neurons of the midbrain (oculomotor nucleus neurons)

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3
Q

Define lower motor neuron

A

–Lower motor neuron

  • §Projects directly to skeletal muscle

As discussed previously Upper motor neuron are those motor system neurons that Project to lower motor neurons. These are located in motor cortex, Also as previously discussed upper and lower motor neurons are defined by their connections not their position in the nervous system. For instance upper motor neurons of the pontomedullary junction (vestibular nuclei) can innerate lower motor neurons of the midbrain (oculomotor nucleus neurons)

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4
Q

Components/paths (general) of motor systems & pathways: (4)

A
  1. Origin from the upper motor neurons
  2. Descending pathway – with decussation in some pathways
  3. Synapse on a lower motor neuron
  4. The efferent fibers of the lower motor neuron are the only connection between the CNS and PNS
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5
Q

Two things that Motor systems include;

A

Include

  1. Neuromuscular junction
  2. Circuitry to plan, program, coordinate, and modulate the descending motor pathways
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6
Q

More about UMNs (2)

A
  1. •Projections that descend to converge on alpha lower motor neurons
  2. •Originate from many higher motor centers
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7
Q

What are the higher motor centers that UMNs originate from?

A
  1. Cerebral Motor Cortex
  2. Brainstem nuclei
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8
Q

As a higher motor center where UMNs orgiginate, what is the cerebral motor cortex responsible for?

A

§Responsible for voluntary movement

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9
Q

As a higher motor center where UMNs orgiginate, what are the brainstem nuclei responsible for? (4)

A
  1. Influencing voluntary movement,
  2. Posture
  3. Muscle tone
  4. Regulation of reflexes
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10
Q

Name the motor tracts that are included in the prominent motor descending pathways? (5)

A
  1. Corticospinal
  2. Reticulospinal
  3. Vestibulospinal
  4. Tectospinal
  5. Rubrospinal
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11
Q

another name for corticospinal tract

A

pyramidal tract

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12
Q

do motor tracts go ipsilaterally or contralaterally?

A

–Some decussate,

some ipsilateral &

some bilateral

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13
Q

In descending Decussated pathways: are s/s contralateral or ipsilateral if the damage is rostral to the decussation?

A

Contralateral

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14
Q

In descending Decussated pathways: are s/s contralateral or ipsilateral if the damage is caudal to the decussation?

A

ipsilateral s/s

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15
Q

What are two distinct types of LMNs?

A
  1. –Alpha (a) motor neurons – cause voluntary muscle contraction
  2. –Gamma (g) motor neurons – specialized to control gain of muscle receptor (muscle spindle)
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16
Q

what type of LMN is considered the final common pathway?

A

•alpha-motor neurons - the final common pathway
–ONLY connection to skeletal muscle

17
Q

Where are cell bodies of LMNs?

A

•Cell bodies in CNS nuclei

  • –Anterior horn of spinal cord
  • –Cranial nerve nuclei of brainstem
18
Q

Where do LMN axons go?

A

•Axons leave CNS to innervate striated skeletal muscle cells

19
Q

can a single motor neuron innervate more than one muscle cell?

A

yes

Single motor neuron can innervate multiple muscle cells

20
Q

what is the term used for a single lower motor neuron and all of the muscle cells it innervates?

A

Motor Unit

21
Q

What is the pattern of LMN activity and muscle contraction dependent on?

A

the pattern of excitatory & inhibitory inputs to LMNs

22
Q

What is a neuromuscular junction?

A

A speciallized synaptic ending where LMN communicates with the muscle cell
also called hte motor end plate

23
Q

what happens at a Neuromusclular junction? (two more specific steps)

A
  1. –Cholinergic receptors produce end-plate potentials
  2. –Creates action potentials in skeletal muscle cells to produce contraction
24
Q

Somatic Motor Systems: Complexity (3)

A
  1. •Modulation of UMN systems by thalamus, basal ganglia, and cerebellum
  2. •Topographical organization of cortical motor areas, descending fibers, and nuclei
  3. •Parallel operations – UMNs exert simultaneous influences on the final common pathway

Modulation of UMN systems can occur by the action of thalamus, basal ganglia, and cerebellum. Just as we saw in sensory systems, there is a Topographical organization of cortical motor areas, descending fibers, and lower motor nuclei. There are Parallel operations of upper motor neurons exert simultaneous influences on the final common pathway of the lower motor neurons

25
Q

Explain the feedforward control mechanism

A
  • –movements that require anticipatory relationships between the motor system and environment
  • –Quality of the outcome depends upon quality of the sensory information available & analysis

As described previous there are two levels of control – Feedforward and feedback control. Feedforward control are involved in movements that require anticipatory relationships between the motor system and environment

Quality of the outcome depends upon quality of the sensory information available & analysis prior to the movement

26
Q

Explain the feedback control mechanism

A
  • –Movements that require ongoing sensory feedback from the body and environment
  • –Adjustments for unexpected perturbations

Feedback control is involved in Movements that require ongoing sensory feedback from the body and environment. Feedback involves Adjustments for unexpected perturbations.

27
Q

Explain Reciprocal innervation of agonist and antagoinst muscles

A
  • •Coordination of movement utilizes reciprocal innervation of agonist and antagonist muscles
    • –Excitation of one muscle & simultaneous inhibition of antagonist
    • –Difficulty of co-contraction of antagonists

Coordination of movement utilizes reciprocal innervation of agonist and antagonist muscles. When there is Excitation of one muscle, there is simultaneous inhibition of antagonist muscles. This is done by the excitation of the agonist motor neurons and inhibition of antagonist motor neurons. This pattern of innervation explains why there is a Difficulty of co-contraction of antagonist.

28
Q

what does purposeful movements involve?

A

•Purposeful movements

  • –Involve multiple muscles working together in a variety of synergistic ways
  • –Simultaneous & sequential activation
29
Q

Hown can the complexity of siglenas be reduced when purposeful movement is produced?

A
  • –Central pattern generators (CPGs) reduce the complexity of signals required for routine purposeful movement
    • This provides a Pre-organization of activation of diverse muscles & sequence.
30
Q

List three types of motor activity

A
  1. Tonus
  2. Reflexes
  3. Voluntary Movement
31
Q

What is tonus?

A

•background muscular activity

32
Q

what are reflexes?

A

•Reflexes – simple, stereotyped overt movements set into motion by a peripheral stimulus

33
Q

what is voluntary movment?

A

•Voluntary movement – decision to act, learned, exploits pre-programmed subroutines

34
Q

what two terms should never be used to describe neural activity?

A
  1. “hard-wired”
  2. “brain is like a computer”

But there are two terms that should not be used to terms of neural activity including “hard wired” or the” brain is like a computer”. Motor activity is so plastic that these terms are not responsible to use

35
Q

What are two types of motor signs when there is neural damage?

A
  1. Positive Signs
  2. Negative signs
36
Q

what is another term for positive signs?

A

release phenomena

37
Q

•Positive signs (release phenomena)

A

–An excess of neural activity
–Hyperreflexia, hypertonicity, Babinski sign

38
Q

negative signs

A

–Motor deficits due to the loss of function of damaged neural structures
–Weakness, loss of speed of movement, loss of dexterity