Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

MDCN 360: Course 2 > Workshop: Skin Infections > Flashcards

Workshop: Skin Infections Flashcards

1
Q

two broad groups of fungi:

A

yeast and mold

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Mold spores germinate to produce ____ _____=
hyphae
• ____ infections
• Tinea corporis, tinea capitis, tinea pedis,
onychomycosis

A

Mold spores germinate to produce branching filaments=
hyphae
• DERMATOPHYTE infections
• Tinea corporis, tinea capitis, tinea pedis,
onychomycosi

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

• Yeasts are solitary rounded forms that reproduce by
making more rounded forms (____/____)
• CANDIDA

A

• Yeasts are solitary rounded forms that reproduce by
making more rounded forms (budding/fission)
• CANDIDA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what is the morphology

A
  • irreuglar annular border with central clearing. its a large and riased plaque with a roled/scale border.

probably a tinea infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what is the morphology

A

hyperpifmented plaque with some raising along peripheral border.

central clearing, probably tinea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q
A

athletes foot; tinia pedis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

t/f tinea corporis and pedis treatment requries combination treatment with topical corticosteroid and anti-fungal

A

false. corticosteroids clears fungus from top surface but makes the fungal infection grow deeper into thes kin. loses its central clearing–makes it harder to diagnose.

  • Treat a fungal infection with topical antifungal
  • Treat cutaneous inflammation (eczema, psoriasis) with topical corticosteroid

• When you don’t know- do a fungal scraping, bacterial swab and/or
take a skin biopsy!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q
A

majocchi’s granuloma

Treating tinea corporis with a topical steroid
Erythematous Plaque Loses its advancing border

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q
A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q
A

tinea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

ddx

A

onychomychosis; accumulation of fungal contents underneath the nail bed and causes separation.

could be psoriasis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

morphology

A

located on skin folds

erythematous papules coalescing into patches

satellite lesions

candida intriga or diaper rash

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q
A

candida intriga

• Focus on the bright erythematous patches WITH satellite
erythematous papules • These satellite papules = candida infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

treatment with diaper dermatitis

A

its a very common to have secondary candidal infection

  • keep the area as dry as possible
  • change diaper frequently; especially afer poo
  • use barrier cream
  • use topical antifungal if candida suspected
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q
A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

morphology

A

crusting

erythematous papules coalescine together to form blistering erythematous plaques

probably impetego

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q
A

crusting

erythematous papules coalescine together to form blistering erythematous plaques

probably impetego

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q
A

crusting

erythematous papules coalescine together to form blistering erythematous plaques

probably impetego

  • kind of looks like psoriasis
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

bacteria that causes impetigo and mechanism

A

staph aureus causing local production of exfoliative toxins, separting the keratinocytes within the granular layer. same as in pemphigus (autoantibodies target demosomes)

  • it’s contagious; person to person or fomite contact
  • infection usually occurs of site of scratching (insect bites, atopic derm)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q
A

baterial folliculitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

lesion caused by bacterial folliculitis

A

furuncle= entire hair follicle and surrounding tissue involved.

  • multiple = carbuncle.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

treatment for bacterial folliculitis

A

superficial: antibacterial washes with benzoyl peroxide, topical antibiotics.
- widespread or recurrent- oral beta-lactam antibiotics, tetracyclines or macrolides.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q
A

carbuncle: larger pustules coalescing onto an inflammatory plaque.

several follicles infected.

• Tx: warm compresses, incision and DRAINAGE, systemic antibiotic
therapy if drainage not possible
• MRSA common

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

cellulitis

A
25
Q

what layers are affected by cellulitis

A

deep dermis and subcutaneous tissue causing erythema swelling warmth and tenderness with ill-defined borders

26
Q

4 key signs of inflammation/infection

A
  1. rubor
  2. calor
  3. dolor
  4. tumor
27
Q

which bacterias cause cellulitis

A

• GAS or staph aureus (diabetic often mixed gram pos, neg, anaerob)

28
Q
A

cellulitis

29
Q

t/f you should prescribe antibiotics for lower leg lredness and swelling

A

false. Cellulitis is very rarely bilateral– must rule out other causes

30
Q

primary morphology

A

grouped/clustered vesicles. indicative of viral infection

31
Q
A

groups of clustered vesicles that are now pustules. probably started as a viral infection, then became secondary impetigenized (secondary staph infection)

32
Q

HSV1, 2, 3.

A

1= oral

2= genital

3= varicella zoster

33
Q

where is latency of hsv virus “stored”

A

sensory ganglia. Virus replicates at site of infection, travels to the dorsal root ganglia
where it becomes latent (exist in non-infectious state in host = evades
detection)

34
Q

symptomatic primary infection symptoms

A

• gingivostomatitis in children or pharyngitis and mono-like syndrome in young adults-
mouth, lips, buccal mucosa, gingivae

• Painful erosive balanitis/vulvitis, often involve cervix, buttocks, perineum, dysuria, aseptic meningitis in 10% of females, urinary retention, extragenital lesions

35
Q

what is eczema herpeticum

A

occurs in someone with barrier disruption in the skin = atopic dermatitis.

  • widespread disseminated of HSV1 ( or 2) across affected skin.
36
Q
A

eczema herpeticum

37
Q
A

eczema herpeticum

38
Q

what are dermatomes

A

an area of the skin supplied by nerves from a single spinal root

39
Q
A

juicy vesicles and papules with a clear demarkation evident.

shingles

40
Q
A

shingles

41
Q
A

shingles

42
Q

treatment of Zoster

A
  • begin antiviral treatment within 72 hours optimally (but up to 7 days after is still beneficial)= earlier is better

• Acyclovir, famciclovir and valacyclovir are FDA approved
• Decrease severity and duration of skin lesions and pain • IV acyclovir for immunocompromised and those with serious complications

43
Q

most common complication of shingles

A

Postherpetic neuralgia = Damage to the sensory nerves following
zoster infection, resulting in burning, debilitating pain > 3 months
after shingles episode

44
Q

describe the morphology

A

vesicles on an erythematous base. DEW DROPS ARE CHARACTERISTIC CHICKEN POX

45
Q
A

primary varicella infection

-

46
Q

whats goin on

A

erythematous papules

  • umbilization = ciruses
  • not pustules = not yellow– not vesicles cause they are not fluid filled

MOLLUSCUM CONTAGIOSUM POXVIRUS

47
Q

treatment for molluscum

A
  • watchful waiting, most resolve on own in months to years

BUT it doesn’t look really good. topical; cantharidin, podophyllin cream, cryotherapy, curettage etc.

48
Q
A

HPV

49
Q
A

HPV; pedunculated on shaft of penis

50
Q

t/f palmar or plantar warts are oncogenic

A

false. ones that are cerivcal, anogenital warts cause squamous cell carcinoma

51
Q

how does HPV infect the host

A

it’s sneaky. HPV evolved to evade immune surveillance. There is no viremic phase during life cycle so avoids systemic immune response.

• 2/3 warts spont regress in 2 years and lesions of multifocal infections often
regress concomitantly

52
Q
A

scabies

53
Q
A

scabetic nodules of the genitals. erythematous papules

54
Q

scabies treatment

A

MAKE SURE YOU TREAT ALL FAMILY MEMBESR

55
Q
A

furuncle due to bacterial folliculitis

56
Q
A

yeast/candida = moisture and heat causing candida intertrigo

57
Q
A

scabies

58
Q
A

shingles on a specific dermatome

59
Q
A

erysipelas- superficial variant of cellulitis affecting dermis with lymphatic involvemnet vs cellulitis

GAS

  • check for recent strep infection and treat with penicillin

MDCN 360: Course 2 (70 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions