Unit 9 - Class Activities Flashcards
Which of the following sets of conditions is an indication that the client with a history of left-sided heart failure is developing pulmonary edema?
a. Distended jugular veins and wheezing
b. Dependent edema and anorexia
c. Coarse crackles and tachycardia
d. Hypotension and tachycardia
c. Coarse crackles and tachycardia
* Signs of pulmonary edema are identical to those of acute heart failure. Signs and symptoms are generally apparent in the respiratory system and include coarse crackles, severe dyspnea, and tachypnea. Severe tachycardia may occur due to sympathetic stimulation in the presence of hypoxemia. Blood pressure may be decreased or elevated depending on the severity of the edema.
Jugular vein distention, dependent edema, and anorexia are symptoms of right sided heart failure.
a way to remember symptoms L eft stands for Lungs
The major goal of therapy for a client with heart failure and pulmonary edema should be to
a. Increase cardiac output
b. Improve respiratory status
c. Decrease peripheral edema
d. Enhance comfort
a. Increase cardiac output
* Increasing cardiac output is the main goal of therapy for the client with heart failure or pulmonary edema. Pulmonary edema is an acute medical emergency requiring immediate intervention. Respiratory status and comfort will be improved when cardiac output increases to an acceptable level. Peripheral edema is not typically associated with pulmonary edema
help regulate BP/fluid -levels of BNP in the blood increases as the ventricular walls expand from increased pressure, making it a helpful diagnostic, monitoring, and prognostic tool in the setting of HF.
: also called B-type natriuretic peptide & N-terminal-pro-BNP (NT-pro-BNP)
- higher the number = more severe Heart Failure
Brain Natriuretic Peptide (BNP)( neuro- hormone )
Left sided Heart Failure Symptoms:
FORCED
F=Fatigue O=orthopnea R=Rales/Restlessness C=Cyanosis/confusion E=Extreme weakness D=Dyspnea
Right sided heart failure
BACONED
B=Bloating/ weight gain/ enlarged liver & spleen A=Anorexia/ GI distress C=Cyanosis/Cool legs O=Oliguria N=Nausea E=Edema/ dependent D=Distended neck veins
A nurse is assessing a client with heart failure. The nurse should asses the client based on which compensatory mechanisms that are activated in the presence of heart failure? Select all that apply.
a. Ventricular hypertrophy
b. Parasympathetic nervous stimulation
c. Renin-angiotensin-aldosterone system
d. Jugular venous distention
e. Sympathetic nervous stimulation
a. Ventricular hypertrophy
c. Renin-angiotensin-aldosterone system
e. Sympathetic nervous stimulation
*When the heart begins to fail, the body activates three major compensatory systems: ventricular hypertrophy, the renin-angiotensin-aldosterone system, and sympathetic nervous stimulation.
Parasympathetic stimulation and jugular venous distention are not compensatory mechanisms associated with heart failure.
The nursing instructor is teaching their clinical group how to assess a client for congestive heart failure. How would the instructor teach the students to assess a client with congestive heart failure for nocturnal dyspnea?
a. By collecting the client’s urine output
b. By observing the client’s diet during the day
c. By measuring the client’s abdominal girth
d. By questioning how many pillows the client normally uses for sleep
d. By questioning how many pillows the client normally uses for sleep
occurs at the same time each day, typically at rest, unrelated to activity , many times at night., caused by coronary spasm , cause is unknown
Atypical angina (prinzmetal angina)
most common and predictable form common manifestation of C.A.D
Stable angina
First Line Therapy:
Beta Blockers
Change positions slowly/ orthostatic BP may be an issue, check for fatigue, dizziness, light headed )
Take every day
Take own BP and chart/ demo/ return demonstrate may lower H.R.
Hold if H.R low
Can cause hypoglycemia in DM patients
Given to patients with cardiomyopathy and cardiac dysrhythmia as well as HTN
MONA
morphine
oxygen
nitrate
aspirin (ASA)
occurs when edema fluid that has accumulated during the day and is reabsorbed into the circulation at night.
This causes fluid overload and pulmonary congestion. The client awakens at night short of breath and frightened.
Paroxysmal nocturnal dyspnea
The nursing instructor is teaching their clinical group how to assess a client for congestive heart failure. How would the instructor teach the students to assess a client with congestive heart failure for nocturnal dyspnea?
a. By collecting the client’s urine output
b. By observing the client’s diet during the day
c. By measuring the client’s abdominal girth
d. By questioning how many pillows the client normally uses for sleep
d. By questioning how many pillows the client normally uses for sleep
A 69 year old female has a history of heart failure. She is admitted to the emergency department with heart failure complicated by pulmonary edema. On admission of this client, which of the following should the nurse assess first?
a. Blood pressure
b. Skin breakdown
c. Serum potassium level
d. Urine output
a. Blood pressure
* It is a priority to assess blood pressure first because people with pulmonary edema typically experience severe hypertension that requires early intervention.. Potassium levels are not the first priority. The nurse should monitor urine output after the client is stable.
Which of the following nursing diagnoses would be appropriate for a client with heart failure? Select all that apply.
a. Ineffective tissue perfusion related to decreased peripheral blood flow secondary to decreased cardiac output
b. Activity intolerance related to increased cardiac output
c. Decreased cardiac output related to structural and functional changes
d. Impaired gas exchange related to decreased sympathetic nervous system activity.
a. Ineffective tissue perfusion related to decreased peripheral blood flow secondary to decreased cardiac output
c. Decreased cardiac output related to structural and functional changes
* Heart Failure is a result of structural and functional abnormalities of the heart tissue muscle. The heart muscle becomes weak and does not adequately pump the blood out of the chambers. As a result, blood pools in the left ventricle and backs up into the left atrium and eventually into the lungs. Therefore, greater amounts of blood remain in the ventricle after contraction thereby decreasing cardiac output. In addition this pooling leads to thrombus formation and ineffective tissue perfusion because of the decrease in blood flow to the other organs and tissues of the body. Typically, these clients shave an ejection fraction of less than 50% and poorly tolerated activity. Activity intolerance is related to the decrease not increase in cardiac output. Gas exchange is impaired. However, the decrease in cardiac output triggers compensatory mechanisms, such as an increase in sympathetic nervous system activity.