Epi Mix P 3001-3200 Flashcards
per os antibiotic treatment must be used in the case of fowl cholera
T
nasal discharge and conjunctivitis are clinical signs of fowl cholera
T
the morbidity of fowl cholera is high
T
the exotoxin of the agent is responsible for the clinical signs of fowl cholera
F
inflammation of the wattle is a clinical sign of acute fowl cholera
F
the endotoxin of the agent is responsible for the clinical signs of fowl cholera
T
Europe is free from fowl cholera
F
Haemorrhagic diarrhoea is a clinical sign of fowl cholera
T
Fowl Cholera can occur in ducks and geese
T
Fowl cholera is always an acute disease
F
Feather pecking or force feeding can predispose animals to fowl cholera
T
Parent birds have to be vaccinated just before laying in order to prevent fowl cholera
F
Germinative infection is a frequent way of transmission of the agent of fowl cholera
F
Geese is resistant to fowl cholera
F
Fowl cholera cannot be prevented by vaccination
F
Focal inflammation in the liver is a typical post mortem lesion of fowl cholera
T
Hemorrhages generally cannot be seen as post mortem signs of fowl cholera
F
After recovering from fowl cholera the animals do not carry the agent any more
F
There are vaccines on the market to prevent fowl cholera
T
Fowl cholera occurs only in hens
F
Fowl cholera is caused by certain serotypes of Pasteurella multocida
T
The agent of fowl cholera is an obligate pathogen
F
Germinative infection is common in the case of fowl cholera
F
Turkey are highly susceptible to fowl cholera
T
Fowl cholera is caused by Pasteurella multocida strains
T
Fowl cholera occurs only in chicken
F
Fowl cholera is typically seen in day-old birds
F
Fowl cholera is a septicemic disease
T
The agent of fowl cholera is a facultative pathogenic bacterium
T
The endotoxin of the agent is responsible for the lesions of fowl cholera
T
Fowl cholera is caused by Riemerella anatipestifer
F
Endotoxins of Pasteurella multocida cause fowl cholera
T
Fowl cholera has always an acute course
F
Fowl cholera could cause focal inflammation-necrosis in the liver
T
Fowl cholera is caused by introducing highly virulent Pasteurella multocida strains
T
Plucking and fattening of geese is a predisposing factor for fowl cholera
T
Fowl cholera causes acute septicaemia
T
The chronic form of fowl cholera is caused by less virulent strains
T
Day-old chickens are resistant to fowl cholera
T
Vaccines against fowl cholera are not available
F
Fowl cholera may cause high mortality in water birds
T
Turkeys are not sensitive to fowl cholera
F
Fowl cholera occurs mainly at the end of the winter
F
Fowl cholera occurs mostly during the summer and autumn
T
Germinative infection is the primary way of spreading fowl cholera
F
Mortality of fowl cholera can reach 100% in a susceptible flock
T
In development of fowl cholera, plucking the animal has an important role
T
Fowl cholera can cause high losses among day-old chicken
F
Fowl cholera is caused by leukotoxin-producing Pasteurella (Mannheimia) haemolytica
F
Fowl cholera has higher mortality in young broiler stocks
F
Fowl cholera has high mortality in water poultry
F
Fowl cholera is caused by Pasteurella haemolytica
F
Fowl choler occurs in the tropics and subtropics mostly
T
Wild birds are very susceptible to fowl cholera
F
Susceptibility in hens decrease with age in case of fowl cholera
F
The clinical signs of anatipestifer disease are more severe in hens than in ducks
F
Fibrinous perihepatitis is a postmortem lesion of anatipestifer disease
T
Ataxia and spasms are common clinical signs of anatipestifer disease
T
Anatipestifer disease is a zoonosis
F
Sinusitis is a frequent clinical sign of ornithobacteriosis
T
Nervous signs are frequently seen in the case of ornithobacteriosis
F
The agent of anatipestifer disease causes septicaemia
T
The agent of anatipestifer disease is an obligate pathogenic bacterium
F
Clinical signs of anatipestifer disease are limited to the respiratory tract
F
Anatipestifer disease can be prevented by inactivated and attenuated vaccines
T
Ornithobacterium rhinotracheale can cause disease in chicken and turkey
T
Production of large amount of mucous in the upper respiratory tract is common in ornithobacteriosis
T
The agent of ornithobacteriosis colonises the upper respiratory tract
T
Nasal discharge and conjunctivitis are clinical signs of anatipestifer disease
T
The endothelium is damaged in anatipestifer disease
T
germinative transmission is important in the case of anatipestifer disease
F
Generally laying flocks are vaccinated in order to prevent ornithobacteriosis
F
Overcrowding can predispose animals to ornithobacteriosis
T
Deformation of the eggshell can be seen in the case of ornithobacteriosis
T
Arthritis is a frequent clinical sign of ornithobacteriosis
F
Pasteurella antaipestifer is the aetioligcal agent of Anatipestifer disease
F
Anatipestifer disease is caused by Riemerella anatipestifer
T
Endotoxin release is important in the pathogenesis of the anatipestifer disease
T
Anatipestifer disease mostly occurs in small backyard flocks
F
We can use agglutination to diagnose the anatipestifer disease
T