Epi Mix L 2201-2400 Flashcards

1
Q

E. coli can cause clinical signs only in chicken below of 2 weeks of age

A

F

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2
Q

E. coli disease of poultry is zoonotic

A

F

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3
Q

Isolation of Escherichia coli from the faeces confirms diagnosis of coli septicaemia of poultry

A

F

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4
Q

Mycoplasma can predispose chicken to Escherichia coli infection

A

T

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5
Q

Escherichia coli can cause only local lessons in chicken, it cannot be generalized

A

F

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6
Q

E. coli in chicken found worldwide

A

T

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7
Q

E. coli is caused by enteroinvasive E. coli

A

T

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8
Q

Germinative infection occurs in case of E. coli disease in poultry

A

T

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9
Q

Enterotoxigenic E. coli strains can cause septicaemia in day old chicken

A

F

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10
Q

Enterotoxigenic E. coli causes death in young chicks

A

F

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11
Q

E. coli kills the embryo in the egg

A

T

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12
Q

PO antibiotics via drinking water is a good way of treating E. coli in poultry.

A

T

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13
Q

E. coli disease in poultry mostly occurs in layers at beginning of egg-laying period

A

F

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14
Q

E. coli disease in poultry causes severe encephalitis in septicaemic form

A

F

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15
Q

Vaccines can be used for the prevention of E. coli disease in poultry

A

T

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16
Q

Retarded absorption of the yolk sack is a post mortem lesion in coli-septicaemia in chickens

A

T

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17
Q

Coli-diarrhoea of rabbits is typically seen in suckling animals

A

F

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18
Q

Verotoxigenic Escherichia coli strains cause coli-diarrhoea of rabbits

A

F

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19
Q

High fibre content of the feed can prevent coli diarrhoea of rabbits

A

T

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20
Q

Mucoid and watery diarrhoea are typical in the case of Coli Diarrhoea of Rabbits

A

T

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21
Q

Enterotoxigenic E. coli strains are the causative agents of E. coli diarrhoea in rabbits

A

F

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22
Q

Decreased fiber content of the food can predispose rabbits to E. coli diarrhoea

A

T

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23
Q

There is widespread vaccination in order to prevent E. coli diarrhea in rabbits

A

F

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24
Q

Coli diarrhoea of rabbits is caused by enteropathogenic E. coli strains

A

T

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25
Coli diarrhoea of rabbits is mainly seen in the first week of life
F
26
Insufficient fiber content is a predisposing factor of coli diarrhoea of rabbits
T
27
Coli diarrhoea is prevented by widespread vaccination of the mothers
F
28
Insufficient amount of milk predisposes rabbits to coli-diarrhoea
F
29
Mucoid diarrhoea is a typical sign of coli-diarrhoea of rabbits
T
30
Isolation of Escherichia coli from the faces of rabbits with diarrhoea confirms coli- diarrhoea
T
31
E. coli is not a normal inhabitant of the gut flora in rabbits
F
32
Foot rot is more severe in cattle than in sheep
F
33
The hoof can be detached in the case of foot rot
T
34
Virulent foot rot can be treated with antibiotics
T
35
Foot rot occurs only in sheep
F
36
The agent of foot rot can produce proteases; they are virulence factors
T
37
the causative agent of foot rot is dichelobacter nodosus
T
38
the main virulence factors of dichelobacter nodosus are toxins
F
39
the warm and wet environment can predispose to foot rot
T
40
Fusobacterium necrophorum subsp. Necrophorum can help the agent of foot rot in causing disease
T
41
Keratinsase production is a virulence factor of the agent of Foot Rot
T
42
Foot Rot cannot be prevented by vaccination
F
43
Production of keratinase is a virulence factor of Dichelobacter nodosus
T
44
Morbidity of foot rot is very low, 1-5%.
F
45
Foot bath in zinc sulphate can be used for the treatment of foot rot
T
46
Dichelobacter nodosus causes foot rot in sheep
T
47
Dichelobacter nodosus is helped by other bacteria in the pathogenesis of foot rot
T
48
Dichelobacter nodosus can produce keratinase
T
49
Foot rot has more severe clinical signs in goats than sheep
F
50
Exotoxins of the causative agent are responsible for the lesions of foot rot
F
51
Extracellular enzymes of the causative agent are responsible for the lesions of foot rot
T
52
Footbath with formalin can be used for prevention or for treatment of milder cases of foot rot
T
53
Toxins are the main virulence factors of Dichelobacter nodosus
F
54
Warm and wet climate predispose animals to foot rot
T
55
Animals with softened hoof wall are more susceptible for panaritium (foot rot).
T
56
Contaminated, dirty bedding is a predisposing factor for foot rot
T
57
In the development of foot rot, fimbriae of the pathogen have an important role
T
58
Treatment of foot rot is using foot bath containing formalin and sterogenol
T
59
For treatment of panaritium, formalin foot baths are recommended
T
60
Foot rot is a very rare disease in Europe
F
61
Fimbria and extracellular enzymes are the virulence factors of the agent of foot rot
T
62
Dichelobacter causes Necrobacillosis
F
63
The causative agent of foot root can produce exotoxins
F
64
The causative agent of foot root can produce enzymes
T
65
Foot rot can be prevented by foot bath
T
66
Animals with soft horn are more predisposed to panaritium
T
67
Strongly contaminated bedding is a predisposing factor to panaritium
T
68
The fimbriae of the pathogens play an important role in the development of panaritium
T
69
To treat severe foot rot we use parenteral antibiotics
T
70
Fusobacterium necrophorum subsp. necrophorum causes necrobacillosis
T
71
Aerosol infection is a common form of transmission of the agent of necrobacillosis
F
72
Necrobacillosis is mainly seen in young animals
T
73
Rumen parakeratotosis can predispose to necrobacillosis
T
74
Insufficient disinfection of the navel can predispose to necrobacillosis
T
75
Abortion is a frequent clinical sign of necrobacillosis
F
76
Laryngeal erosion serves as place of entry for the agent of calf diphtheria
T
77
calf diphtheria is caused by Dichelobacter nodosus
F
78
Calf diphtheria is caused by Fusobacterium necrophorum subsp. necrophorum
T
79
Respiratory diseases can predispose to calf diphtheria
T
80
Fusobacterium necrophorum subsp. funduliforme is the agent Necrobacillosis in lambs
F
81
Necrobacillosis can occur as a consequence of navel infection in lambs
T
82
Pulmonary necrosis is a frequent lesion of necrobacillosis of lambs
F
83
Liver necrosis is a frequent lesion of necrobacillosis of lambs
T
84
Fusobacterium funduliforme is the causative agent of Necrobacillosis
F
85
Necrosis of the mucous membranes in the oral cavity is a clinical sign of Necrobacillosis
T
86
Focal necrosis in the kidney is a frequent post mortem lesion of Necrobacillosis
F
87
Necrobacillosis is prevented with wide vaccination
F
88
Wounds can predispose to Necrobacillosis
T
89
Navel infection can occur in the case of Necrobacillosis
T
90
Lesions of Necrobacillosis can be seen frequently in the oral cavity
T
91
Rough, sticky feed can predispose to Necrobacillosis
T
92
Necrosis in the liver can be a post mortem lesion of Necrobacillosis
T
93
Necrobacillosis can only be seen in lambs
F
94
In the case of Necrobacillosis infection can happen through wounds
T
95
In the case of Necrobacillosis, aerogenic infection is common
F
96
Lesions of Necrobacillosis are frequently found in or around the oral cavity
T
97
Fusobacteria are highly resistant, they remain viable in the environment for several weeks
F
98
Wounds and abrasions can predispose to Necrobacillosis
T
99
Incorrect disinfection of the naval area can lead to Necrobacillosis
T
100
Wounds on the oral mucosa can predispose animals to Necrobacillosis
T
101
The clinical signs of Necrobacillosis can frequently be seen in young animals
T
102
Necrobacillosis is a generalized disease with high fever and depression
F
103
Vaccines containing attenuated agents are widely used to prevent Necrobacillosis
F
104
Necrobacillosis is caused by Dichelobacter nodosus
F
105
Foot rot is the main sign of Necrobacillosis
F
106
The lesions in Necrobacillosis are restricted to the mouth
F
107
Necrobacillosis occurs mainly in young animals
T
108
There is no treatment for Necrobacillosis
F
109
Umbilical disinfection has an important role in the prevention of Necrobacillosis
T
110
Necrobacillosis in lambs can be seen as a result of an umbilical infection
T
111
Fusobacterium necrophorum subsp. necrophorum strains can cause Necrobacillosis of calves
T
112
You can find necrosis in the mouth in case of Necrobacillosis in lambs
T
113
You can find liver abscesses in case of Necrobacillosis
F
114
There are good immune reactions against salmonellae in the case of generalised salmonella diseases
T
115
Salmonella enterica has 6 subspecies
T
116
The salmonella serotypes that cause salmonellosis in animals mainly belongs to Salmonella enterica. Subs. Enterica
T
117
Feeding dogs with slaughterhouse waste can predispose them to Salmonellosis
T
118
Salmonella typhi is a zoonotic agent
F
119
Salmonella Typhysuis is a causative agent of swine typhoid
T
120
Salmonella typhisuis is an obligate pathogen
T
121
The agent of swine typhoid mainly replicates in the small intestine
F
122
The agent of swine typhoid can cause lesions only in the gut
F
123
Swine typhoid can be transmitted by contaminated utensils
T
124
Swine typhoid occurs in endemic herds above 3 months of age
T
125
Swine typhoid is a chronic disease
T
126
Cough is a frequent clinical sign of swine typhoid
T
127
The agent of swine typhoid can cause focal inflammation and necrosis in the parenchymal organs
T
128
Typhocolitis of swine mainly occurs in growers and adults
T
129
Swine typhoid mainly occurs in suckling piglets
F
130
salmonella choleraesuis is the causative agent of swine typhoid
F
131
the agent of swine typhoid replicates only in the gut, it cannot get into the blood
F
132
vaccination is not recommended for the prevention of swine typhoid
T
133
swine typhoid is a very frequent disease in Europe nowadays
F
134
Swine typhoid is an acute disease
F
135
The agent of swine typhoid can infect pigs, dogs, cats and humans
F
136
Lesions of swine typhoid are always limited to the intestin
F
137
Lesions of swine typhoid are in the large intestine
T
138
Swine typhoid is an acute disease; it is spreading fast in the herd
F
139
S. Typhisuis replicates in the lymphoid patches of the large intestine
T
140
Infection caused by S. Typhisuis is limited to the intestine
F
141
S. Typhisuis can cause a severe acute disease in swine
F
142
Swine typhoid is caused by Salmonella Choleraesuis
F
143
Salmonella Choleraesuis is an obligate pathogen
F
144
Ulcers in the large intestine are frequent post mortem lesions of swine typhoid
T
145
Swine typhoid is treated with penicillin
F
146
Per os infection is frequent in the case of swine typhoid
T
147
Swine typhoid is limited to the intestine, it cannot cause bacteremia
F
148
Lesions of swine typhoid can be seen in the small intestine
F
149
Swine typhoid is an acute disease of swin
F
150
Swine typhoid is caused by Salmonella typhisuis
T
151
Salmonella typhisuis can infect ruminants and swine
F
152
Swine typhoid occurs only in piglets between 2-5 months
F
153
Certain lesions of swine typhoid can be seen in the large intestine
T
154
Swine typhoid is a frequent disease on large scale farms
F
155
Swine typhoid is spreading slowly in the herd
T
156
Swine typhoid can be transmitted with faecal contamination
T
157
The agent of swine typhoid can only replicate in the gut
F
158
Salmonella typhisuis and S. gallinarum are obligate pathogens
T
159
Salmonella typhisuis is widespread in different animal species
F
160
Salmonella typhisuis can be introduced with infected animals
T
161
Salmonella typhisuis can cause a fast spreading infection in the herd
F
162
Swine typhus causes significant economic losses in larger farms
F
163
Salmonella enteritidis causes swine typhus
F
164
Swine typhus is usually an acute disease
F
165
Swine typhus only affects the intestines.
F
166
Salmonella typhisuis causes paratyphoid in swine
F
167
Swine typhoid is a frequent disease in Europe causing high losses
F
168
The agent of swine typhoid replicates in the large intestine
T
169
Killed vaccines are used to prevent swine typhoid
F
170
Swine typhoid is spread by rodents
F
171
In swine typhoid transport is an important predisposing factor
F
172
Swine typhoid are mostly seen in 2-3-week-old piglets
F
173
In swine typhoid, pathological changes are seen in the large intestine
T
174
In swine typhoid, pathological changes are seen in the whole intestines
F
175
Swine typhoid is caused by S. typhi
F
176
Yellow, watery diarrhoea is a clinical sign of swine paratyphoid
T
177
Salmonella Typhimurium can cause swine paratyphoid
T
178
Swine paratyphoid is mainly seen in suckling piglets
F
179
Fever is a common clinical sign of swine paratyphoid
T
180
Focal inflammation and necrosis are a common postmortem lesions of fowl paratyphoid
T
181
Swine paratyphoid is an acute disease
T
182
Classical swine fever can predispose animals to swine paratyphoid
T
183
Antibiotic treatment isn’t allowed in the case of swine paratyphoid
F
184
inflammation of the small intestine is a postmortem lesion of swine paratyphoid
T
185
swine paratyphoid is caused by obligate pathogenic bacteria
F
186
swine paratyphoid has been eradicated from Europe
F
187
swine paratyphoid is most frequently seen in pigs between 2 and 5 months of age
T
188
There is septicaemia in the case of swine parathyphoid
T
189
Cyanosis is a clinical sign of swine paratyphoid
T
190
Salmonella Choleraesuis can cause swine paratyphoid
T
191
Swine paratyphoid generally occurs in all age groups
F
192
High fever is a typical sign of swine paratyphoid
T
193
There are vaccines against swine paratyphoid on the market
T
194
Only Salmonella Typhimurium can cause swine paratyphoid
F
195
Swine paratyphoid is an acute generalized disease
T
196
Antibiotics are not used for the treatment of swine paratyphoid
T
197
Salmonella choleraesuis and S. typhimurium can cause swine paratyphoid
T
198
Swine paratyphoid is most frequent in suckling piglets
T
199
There is no use of antibiotic treatment in the case of swine paratyphoid
F
200
Salmonella Choleraesuis is the only agent of swine paratyphoid
F