Epi Mix D 601-800 Flashcards
Maedi/visna virus is shed only in the tracheal discharge
F
Clinical signs of maedi are generally seen above 3-4 years of age
T
Caprine arthritis-encephalitis virus can cause persistent infection
T
Sheep are resistant against caprine arthritis encephalitis virus
F
Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs
Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs
Encephalitis caused by caprine arthritis encephalitis virus is generally seen in 2-4 month old kids
T
Caprine arthritis encephalitis virus can be transmitted by milk
T
There is intensive vaccination against Caprine arthritis encephalitis in endemic countries
F
Caprine arthritis encephalitis virus is more frequent in dairy goats than in rural breeds
T
Caprine arthritis encephalitis virus is shed in the milk
T
In the case of caprine arthritis encephalitis the signs of encephalitis can be seen in 2-4 months old kids
T
In the case of caprine arthritis encephalitis, arthritis is less frequent than encephalitis
F
Kids are recommended to be isolated in a herd where caprine arthritis encephalitis is present
T
Kids can be infected with caprine arthritis encephalitis virus through the milk
T
Arthritis caused by caprine arthritis-encephalitis virus is typically seen in kids below half a year of age
F
Caprine arthritis encephalitis is characterized by CNS signs in young goats
T
Caprine arthritis encephalitis virus replicates in the intestinal tract
F
Equine infectious anaemia virus is transmitted with blood of the infected animals.
T
Blood sucking arthropods can transmit Equine infectious anaemia virus
T
Mosquitoes are the main vectors of equine infectious anaemia virus; the virus can replicate in them
F
Agar gel diffusion test is used to detect antibodies against equine infectious anaemia
T
Equine infectious anaemia is an acute disease; it does not have a chronic form
F
Iatrogenic transmission of Equine infectious anaemia can happen
T
Equine infectious anaemia virus can damage the bone marrow
T
There are no vaccines for the prevention of equine infectious anaemia
T
Equine infectious anaemia virus is more resistant than other retroviruses
T
Equine infectious anaemia virus disappears from animals after the viraemia
F
Fever is a major clinical sign of equine infectious anaemia
T
Equine infectious anaemia virus is resistant, it can survive several months in the environment
T
Only Equidae are susceptible to equine infectious anaemia virus
T
Ticks are vectors of equine infectious anaemia virus
F
Febrile waves can be seen in equine infectious anaemias
T
Equine infectious anaemia is a vector borne disease
T
Equine infectious anaemia virus causes persistent infection
T
There is no immune reaction in the case of equine infectious anaemia
F
Equine infectious anaemia is zoonotic
F
Equine infectious anaemia is caused by a lentivirus
T
Blood sucking arthropods are mechanical vectors of equine infectious anaemia virus.
T
Agar gel precipitation test can be used to the detection of antibodies against equine infectious anaemia virus.
T
Equine infectious anaemia can damage the medulla of the bone.
T
Equine infectious anaemia has a weak resistance
F
You cannot diagnose Equine infectious anaemia with serology
F
The resistance of equine infectious anaemia is very low
F
Horses and cattle are susceptible to equine infectious anaemia virus
F
In the case of equine infectious anaemia, haemorrhages cannot be seen
F
Animals infected with equine infectious anaemia virus are lifelong carriers.
T
Equine infectious anaemia virus is mainly transmitted with tracheal discharge
F
Attenuated vaccines are used for the prevention of equine infectious anaemia
F
Equine infectious anaemia can be asymptomatic
T
Equine infectious anaemia may cause recurrent fever in horses
T
Horseflies are mechanical vectors equine anaemia virus
T
Equine anaemia is a notifiable disease
T
Animals showing clinical signs of anthrax have to be killed, treatment is not allowed
F
The agent of anthrax is spreading in the herd very fast from animal to animal
F
Anthrax is frequently a peracute disease in cattle
T
Anthrax is caused by Clostridium anthracis
F
Enlargement of the spleen is a frequent postmortem lesion of anthrax
T
Anthrax can occur only in ruminants
F
Anthrax can be diagnosed by staining blood smear
T
Anthrax is zoonosis
T
Incomplete blood clotting is a typical postmortem finding in the case of anthrax
T
Animals are infected with the agent of anthrax mainly per os
T
There are no vaccines for the prevention of Anthrax
F
Anthrax can cause clinical signs in pigs
T
Horses are resistant to Anthrax
F
Generally live vaccines are used for the prevention of anthrax
T
There is a metachromatic staining in the case of Bacillus Anthracis
T
Capsule and oedema factor are virulence factors of Bacillus anthracis
T
Carbon dioxide is needed to the spore production of Bacillus Anthracis
F
Capsule is a virulence factor of the agent of anthrax
T
Pigs are more susceptible to anthrax than sheep
F
Oedema factor is a virulence factor of the agent of anthrax
T
Human anthrax cannot be treated with antibiotics
F
Inactivated vaccines are used for the prevention of Anthrax
F
Only herbivorous animals can show clinical signs of Anthrax
F
The spore of Bacillus anthracis can survive several decades in the soil
T
Bacillus Anthracis cannot produce spores in the infected animals
T
Dogs are more susceptible to Bacillus Anthracis than sheep
F
Europe is already free from anthrax
F
Cattle are infected with B. anthracis mainly from the soil
T