Epi Mix D 601-800 Flashcards
1
Q
Maedi/visna virus is shed only in the tracheal discharge
A
F
2
Q
Clinical signs of maedi are generally seen above 3-4 years of age
A
T
3
Q
Caprine arthritis-encephalitis virus can cause persistent infection
A
T
4
Q
Sheep are resistant against caprine arthritis encephalitis virus
A
F
5
Q
Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs
A
Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs
6
Q
Encephalitis caused by caprine arthritis encephalitis virus is generally seen in 2-4 month old kids
A
T
7
Q
Caprine arthritis encephalitis virus can be transmitted by milk
A
T
8
Q
There is intensive vaccination against Caprine arthritis encephalitis in endemic countries
A
F
9
Q
Caprine arthritis encephalitis virus is more frequent in dairy goats than in rural breeds
A
T
10
Q
Caprine arthritis encephalitis virus is shed in the milk
A
T
11
Q
In the case of caprine arthritis encephalitis the signs of encephalitis can be seen in 2-4 months old kids
A
T
12
Q
In the case of caprine arthritis encephalitis, arthritis is less frequent than encephalitis
A
F
13
Q
Kids are recommended to be isolated in a herd where caprine arthritis encephalitis is present
A
T
14
Q
Kids can be infected with caprine arthritis encephalitis virus through the milk
A
T
15
Q
Arthritis caused by caprine arthritis-encephalitis virus is typically seen in kids below half a year of age
A
F
16
Q
Caprine arthritis encephalitis is characterized by CNS signs in young goats
A
T
17
Q
Caprine arthritis encephalitis virus replicates in the intestinal tract
A
F
18
Q
Equine infectious anaemia virus is transmitted with blood of the infected animals.
A
T
19
Q
Blood sucking arthropods can transmit Equine infectious anaemia virus
A
T
20
Q
Mosquitoes are the main vectors of equine infectious anaemia virus; the virus can replicate in them
A
F
21
Q
Agar gel diffusion test is used to detect antibodies against equine infectious anaemia
A
T
22
Q
Equine infectious anaemia is an acute disease; it does not have a chronic form
A
F
23
Q
Iatrogenic transmission of Equine infectious anaemia can happen
A
T
24
Q
Equine infectious anaemia virus can damage the bone marrow
A
T
25
There are no vaccines for the prevention of equine infectious anaemia
T
26
Equine infectious anaemia virus is more resistant than other retroviruses
T
27
Equine infectious anaemia virus disappears from animals after the viraemia
F
28
Fever is a major clinical sign of equine infectious anaemia
T
29
Equine infectious anaemia virus is resistant, it can survive several months in the environment
T
30
Only Equidae are susceptible to equine infectious anaemia virus
T
31
Ticks are vectors of equine infectious anaemia virus
F
32
Febrile waves can be seen in equine infectious anaemias
T
33
Equine infectious anaemia is a vector borne disease
T
34
Equine infectious anaemia virus causes persistent infection
T
35
There is no immune reaction in the case of equine infectious anaemia
F
36
Equine infectious anaemia is zoonotic
F
37
Equine infectious anaemia is caused by a lentivirus
T
38
Blood sucking arthropods are mechanical vectors of equine infectious anaemia virus.
T
39
Agar gel precipitation test can be used to the detection of antibodies against equine infectious anaemia virus.
T
40
Equine infectious anaemia can damage the medulla of the bone.
T
41
Equine infectious anaemia has a weak resistance
F
42
You cannot diagnose Equine infectious anaemia with serology
F
43
The resistance of equine infectious anaemia is very low
F
44
Horses and cattle are susceptible to equine infectious anaemia virus
F
45
In the case of equine infectious anaemia, haemorrhages cannot be seen
F
46
Animals infected with equine infectious anaemia virus are lifelong carriers.
T
47
Equine infectious anaemia virus is mainly transmitted with tracheal discharge
F
48
Attenuated vaccines are used for the prevention of equine infectious anaemia
F
49
Equine infectious anaemia can be asymptomatic
T
50
Equine infectious anaemia may cause recurrent fever in horses
T
51
Horseflies are mechanical vectors equine anaemia virus
T
52
Equine anaemia is a notifiable disease
T
53
Animals showing clinical signs of anthrax have to be killed, treatment is not allowed
F
54
The agent of anthrax is spreading in the herd very fast from animal to animal
F
55
Anthrax is frequently a peracute disease in cattle
T
56
Anthrax is caused by Clostridium anthracis
F
57
Enlargement of the spleen is a frequent postmortem lesion of anthrax
T
58
Anthrax can occur only in ruminants
F
59
Anthrax can be diagnosed by staining blood smear
T
60
Anthrax is zoonosis
T
61
Incomplete blood clotting is a typical postmortem finding in the case of anthrax
T
62
Animals are infected with the agent of anthrax mainly per os
T
63
There are no vaccines for the prevention of Anthrax
F
64
Anthrax can cause clinical signs in pigs
T
65
Horses are resistant to Anthrax
F
66
Generally live vaccines are used for the prevention of anthrax
T
67
There is a metachromatic staining in the case of Bacillus Anthracis
T
68
Capsule and oedema factor are virulence factors of Bacillus anthracis
T
69
Carbon dioxide is needed to the spore production of Bacillus Anthracis
F
70
Capsule is a virulence factor of the agent of anthrax
T
71
Pigs are more susceptible to anthrax than sheep
F
72
Oedema factor is a virulence factor of the agent of anthrax
T
73
Human anthrax cannot be treated with antibiotics
F
74
Inactivated vaccines are used for the prevention of Anthrax
F
75
Only herbivorous animals can show clinical signs of Anthrax
F
76
The spore of Bacillus anthracis can survive several decades in the soil
T
77
Bacillus Anthracis cannot produce spores in the infected animals
T
78
Dogs are more susceptible to Bacillus Anthracis than sheep
F
79
Europe is already free from anthrax
F
80
Cattle are infected with B. anthracis mainly from the soil
T
81
Anthrax is a per-acute or acute diseases in cattle
T
82
Colic is a typical clinical sign of anthrax in horses
T
83
Anthrax can be diagnosed with microscopic examination of blood
T
84
Bacillus anthracis main virulence factor is in the capsule
T
85
Bacillus anthracis spores: after 1 hour of boiling they are still alive
T
86
Bacillus anthracis makes spores only without oxygen
F
87
Anthrax important symptom is high fever
T
88
If the animals have Anthrax and they have a fever, you have to vaccinate them immediately
F
89
For anthrax we use inactive vaccine
F
90
Humans infected with Anthrax, primarily per os
F
91
Bacillus anthracis, herbivores are especially susceptible
T
92
Bacillus anthracis is not in pig
F
93
Anthrax spreads rapidly in a herd
F
94
Bacillus anthracis is in the soil
T
95
In anthrax, tracheitis common in carnivores
F
96
Anthrax causes necrotic foci in liver
F
97
Anthrax diagnosis with blood/staining
T
98
Anthrax cannot occur in dogs and cats
F
99
The agent of anthrax can infect only herbivorous animals
F
100
The agent of anthrax is not spreading from animal to animal
T
101
There is a septicaemia in cattle in the case of anthrax
T
102
Anthrax is caused by Bacillus bovin
F
103
The capsule of the agent of anthrax is polypeptide
T
104
Anaerobic conditions are needed to the spore formation of the agent of anthrax
F
105
Pigs are the most susceptible animals to the agent of anthrax.
F
106
Animals showing clinical signs of anthrax are not allowed to be treated with antibiotics
F
107
Only capsulated strains of Bacillus anthracis can cause anthrax
T
108
Oedema factor and lethal factor are important virulence factors of Bacillus anthracis
T
109
The clinical signs of anthrax in pigs are more severe than in cattle
F
110
Dogs and cats are resistant against the agent of anthrax
F
111
Only capsulated strain of B. anthracis is virulent
T
112
Toxin is a virulence factor of B. anthracis
T
113
Lethal factor is a virulence factor of B. anthracis
T
114
Cell wall antigen is a virulence factor of B. anthracis
F
115
Oxygen is needed to the spore production of B. anthracis
T
116
Spore is a virulence factor of B. anthracis
F
117
B. anthracis can cause blackleg
F
118
Anthrax is generally seen as a chronic disease in cattle
F
119
In case of anthrax, febrile animals have to be separated and vaccinated
F
120
Animals with anthrax can be treated with penicillin
T
121
B. anthracis can only be diagnosed by bacterial culture
F
122
B. anthracis can only be diagnosed by Ascoli test.
F
123
Animals suspected of being infected with anthrax should be vaccinated
T
124
Animals infected with anthrax should be treated with antibiotics
T
125
Virulence factors of anthrax: capsule, toxin, protective antigen
T
126
Virulence factors of anthrax: capsule, toxin, cilia
F
127
Virulence factors of anthrax: capsule, toxin, oedema factor
T
128
Virulence factors of anthrax: capsule, toxin, cell wall antigen
F
129
Anthrax is an epidemic disease that rapidly develops
F
130
Anthrax is a quickly spreading, contagious infectious disease
F
131
For lab examination of Anthrax you always have to send a spleen sample
F
132
Animals can only be infected by anthrax on the pasture
F
133
Sheep, cattle, and goats are the most sensitive animals to anthrax infection.
T
134
Flagella is a virulence factor of B. anthracis
F
135
The source of anthrax infection on animals is generally the soil
T
136
Anthrax appears generally in the form of a local infection in pigs
T
137
Fever is a typical sign of acute anthrax
T
138
Anthrax can be prevented by using a live vaccine.
T
139
Anthrax is caused by Clostridium chauvoei
F
140
Anthrax is not spreading from animal to animal
T
141
Ruminants are the most sensitive to anthrax
T
142
Animals suffering from anthrax should be treated with antibiotics and hyperimmune sera, they should not be slaughtered
T
143
Causative agent of anthrax is spore-forming bacterium in air
F
144
Anthrax spreads in a herd by direct contact
F
145
In order to diagnose anthrax all carcasses have to be dissected
F
146
Anthrax is an acute disease in cattle with high fever
T
147
Swine is highly susceptible in anthrax
F
148
Splenic fever causes suffocation
T
149
Splenic fever in cattle is a per-acute/acute disease
T
150
Splenic fever is similar in every species
F
151
Carnivorous animals are resistant to Bacillus anthracis
F
152
Incomplete clotting of the blood is a post mortem lesion of anthrax
T
153
Fibrinous pneumonia is a common post mortem lesion of anthrax
F
154
Animals showing clinical signs of anthrax have to be treated with antibiotics immediately
T
155
Horses are more susceptible to Bacillus anthracis than pigs
T
156
Only vaccinated animals are allowed to graze on pastures infected with Bacillus anthracis
T
157
Gastric juice can kill Bacillus anthracis in the meat, so per os infection does not occur in humans
F
158
Most clostridia have low invasive capacity
T
159
Spores of clostridia are generally very resistant against heat
T
160
The habitat of clostridia is the gut and the soil
T
161
Clostridia are obligate aerobic bacteria
F
162
Clostridium perfringens is an obligate pathogenic bacterium
F
163
Clostridium perfringens can produce main and auxillary toxins
T
164
Extracellular enzymes and toxins are virulence factors of clostridia
T
165
There are no vaccines for the prevention of diseases caused by clostridia
F
166
Clostridium is anaerobe spore forming bacteria
T
167
Clostridium bacteria is not in the environment, because it cannot tolerate oxygen
F
168
Clostridium spreads usually rapid in a herd
F
169
Clostridium spread mostly with insecticides
F
170
Clostridium difficile can be treated with metronidazole
T
171
Clostridium difficile is seen in foal and piglets
T
172
Many Clostridium species have flagella
F
173
Clostridium species are only found in the subtropics
F
174
Clostridium can cause severe contagious diseases
F
175
Clostridium are obligate pathogens
F
176
Anaculture or anatoxin vaccines are used for the prevention of malignant oedema
T
177
Cl. chauvoei is the agent of malignant oedema
F
178
Lesions of malignant oedema are mainly seen in the large muscles
T
179
Malignant oedema is generally endogenous in cattle
F
180
Malignant oedema is generally a consequence of wound infection
T
181
Movement difficulties are frequently seen in the case of malignant oedema
T
182
Clostridium novyi can cause malignant oedema
T
183
Malignant oedema can be diagnosed based on clinical signs
F
184
Malignant oedema is caused due to wound infection
T
185
Malignant oedema is only in ruminants
F
186
Malignant oedema, one of the clinical signs is lameness/movement problems
T
187
Malignant oedema, attenuated vaccine for prevention
F
188
Clostridium channel is the agent of malignant oedema
F
189
Clostridium septicum is an agent of malignant oedema
T
190
Clostridium histolyticum can cause malignant oedema
T
191
Agents of malignant oedema can be detected by bacterium culture
T
192
There are no vaccines for the prevention of malignant oedema
F
193
Malignant oedema occurs in ruminants and pigs
T
194
Malignant oedema is an acute fatal disease
T
195
Malignant oedema can be treated with antibiotics
T
196
Malignant oedema can occur in any warm-blooded animal
T
197
Once an area is infected with gas gangrene re-occurrence is common
T
198
Malignant oedema cannot occur in swine
F
199
Malignant oedema usually develop following an endogenous infection
F
200
Malignant oedema is well treated with long-term antibiotics therapy
F
