Epi Mix E 801-1000 Flashcards

1
Q

Malignant oedema can be treated with polymyxin

A

F

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2
Q

Malignant oedema can be well treated with antibiotics over a long period

A

F

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3
Q

gas gangrene (malignant oedema) is a regional illness

A

F

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4
Q

The lesions of malignant oedema are mainly seen in the lungs

A

F

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5
Q

Blackleg is caused by Clostridium septicum

A

F

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6
Q

Lesions of blackleg are mainly seen on the claws

A

F

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7
Q

Lameness is a clinical sign of blackleg

A

T

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8
Q

Blackleg is a frequent disease in pigs

A

F

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9
Q

Generally attenuated vaccines are used for the prevention of blackleg

A

F

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10
Q

Anaculture or anatoxin vaccines are used for the prevention of blackleg

A

T

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11
Q

Blackleg occurs only in tropical and subtropical countries

A

F

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12
Q

Blackleg generally occurs in endemic

A

T

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13
Q

Blackleg occurs most frequently in pigs

A

F

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14
Q

Blackleg is a gas gangrene disease

A

T

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15
Q

Blackleg is generally endogenous in sheep

A

F

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16
Q

Blackleg is generally endogenous in cattle

A

T

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17
Q

Movement disorders and lameness can be clinical signs of Blackleg

A

T

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18
Q

Clostridium chauvoei can produce acids and gas from carbohydrates

A

T

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19
Q

Blackleg occurs mainly in ruminants

A

T

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20
Q

Oedema is a typical clinical sign of blackleg

A

T

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21
Q

Live vaccines are used for the prevention of blackleg

A

F

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22
Q

Blackleg infects ovine through wounds

A

T

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23
Q

In Blackleg disease we use attenuated vaccine

A

F

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24
Q

In the case of sheep, blackleg is generally consequence of a wound infection

A

T

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25
Q

Blackleg is caused by Clostridium chauvoei

A

T

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26
Q

Severe diarrhoea is the main clinical sign of blackleg

A

F

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27
Q

Blackleg occurs in cattle and sheep

A

T

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28
Q

If antibiotics are applied after appearance of the clinical signs of blackleg, treatment is generally successful

A

F

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29
Q

Blackleg disease occurs only in ruminants

A

F

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30
Q

Blackleg can usually be treated with antibiotics successfully

A

F

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31
Q

Blackleg in cattle is mainly endogenous between 6 months-3 years old

A

F

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32
Q

The disease caused by Clostridium chauvoei occurs mainly in cattle and sheep

A

T

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33
Q

The disease caused by Clostridium chauvoei is primarily the result of endogenous infection in cattle

A

T

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34
Q

Blackleg has four toxins

A

T

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35
Q

Blackleg can be prevented by using vaccine

A

T

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36
Q

We use neomycin and polymyxin to treat disease caused by Clostridium chauvoei

A

F

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37
Q

Blackleg in cattle is mainly endogenous between 2 months-2 years old

A

T

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38
Q

Blackleg in bovine is caused by wound infections

A

F

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39
Q

Classical swine fever is a frequent predisposing factor of bradsot

A

F

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40
Q

Oedema in the wall of the abomasum and duodenum are postmortem lesions of bradsot

A

T

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41
Q

Bradsot is caused by Clostridium chauvoei

A

F

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42
Q

Bradsot occurs mainly in tropical and subtropical countries

A

F

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43
Q

Soil contaminated frozen feed is a frequent predisposing factor of bradsot

A

T

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44
Q

Frozen food is a predisposing factor of bradsot

A

T

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45
Q

Bradsot occurs mainly late autumn and winter

A

T

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46
Q

Overeating can predispose the animals to bradsot

A

F

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47
Q

Thickening of and oedema in the stomach wall are typical lesions of bradsot

A

T

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48
Q

Aminoglycosides are successfully used for treatment in the case of bradsot

A

F

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49
Q

Bradsot is caused by Clostridium septicum

A

T

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50
Q

Severe pneumonia is a typical clinical sign of bradsot

A

F

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51
Q

Bradsot has a very fast course

A

T

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52
Q

Bradsot occurs only in suckling lambs

A

F

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53
Q

Bradsot is typically a chronic disease

A

F

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54
Q

Bradsot is common in the summer out on the pasture

A

F

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55
Q

Bradsot is an acute disease resulting in sudden death in many cases

A

T

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56
Q

We can use anaculture strain vaccine against Bradsot

A

T

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57
Q

Bradsot causes oedema of the legs and necrosis

A

F

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58
Q

Post mortem lesions of bradsot can be seen in the stomach (rennet).

A

T

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59
Q

Köves disease is an indicator disease

A

T

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60
Q

CSF is a predisposing factor of koves disease

A

T

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61
Q

Köves disease can be seen in pigs.

A

T

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62
Q

Köves disease is caused by Clostridium chavoei

A

F

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63
Q

Infectious necrotic hepatitis is mainly seen in pigs

A

F

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64
Q

Infectious necrotic hepatitis can be prevented by using anatoxin vaccines

A

T

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65
Q

Liver fluke can predispose animals to infectious necrotic hepatitis

A

T

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66
Q

In sheep, Clostridium septicum causes necrotic liver infection

A

F

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67
Q

Infectious necrotic hepatitis causes inflammation and necrotic nodules in the liver

A

T

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68
Q

There is no vaccine to prevent infectious necrotic hepatitis

A

F

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69
Q

Infectious necrotic hepatitis is caused by Clostridium septicum

A

F

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70
Q

Infectious necrotic hepatitis is mainly seen in suckling lambs

A

F

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71
Q

Parasite infection is a frequent predisposing effect of infectious necrotic hepatitis

A

T

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72
Q

Focal necrosis in the liver is a typical post mortem lesion of infectious necrotic hepatitis

A

T

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73
Q

Anatoxin vaccines can be used for the prevention of infectious necrotic hepatitis

A

T

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74
Q

Infectious necrotic hepatitis is caused by Clostridium novyi

A

T

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75
Q

Infectious necrotic hepatitis is spread by tick

A

F

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76
Q

Infectious necrotic hepatitis is caused by Clostridium novyi type B

A

T

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77
Q

Infectious necrotic hepatitis is found worldwide

A

T

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78
Q

Infectious necrotic hepatitis can be transmitted by liver flukes

A

F

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79
Q

Infectious necrotic hepatitis occurs mostly in young sheep

A

F

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80
Q

There is intravascular haemolysis in the case of bacillary haemoglobinuria

A

T

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81
Q

Bacillary haemoglobinuria is caused by Clostridium haemolyticum

A

T

82
Q

There are no vaccines for the prevention of bacillary hemoglobinuria

A

F

83
Q

Phospholipidase C is a virulence factor of the agent of bacillary hemoglobinuria

A

T

84
Q

Bacillary haemoglobinuria is mainly seen in cattle

A

T

85
Q

Bacillary haemoglobinuria is caused by Clostridium septicum

A

F

86
Q

Jaundice and anaemia are important clinical signs of bacillary hemoglobinuria

A

T

87
Q

Red urine is a typical clinical sign of bacillary hemoglobinuria

A

T

88
Q

Bacillary hemoglobinuria is a slow, chronic disease

A

F

89
Q

Bacillary hemoglobinuria can frequently be seen in horses

A

F

90
Q

Clostridium novyi is the causative agent of bacillary hemoglobinuria

A

F

91
Q

Bacillary hemoglobinuria causes severe haemorrhages

A

T

92
Q

Bacillary hemoglobinuria are caused by infection from the soil

A

T

93
Q

Lamb dysentery occurs in a week old animal

A

T

94
Q

Isolation of the agent from the gut gives aetiologic diagnosis of lamb dysentery

A

F

95
Q

Isolation of Cl. perfringens from the gut confirms the diagnosis of lamb dysentery

A

F

96
Q

Lesions of lamb dysentery are generally seen in the large intestine

A

F

97
Q

Lesions of lamb dysentery can be seen in the small intestine

A

T

98
Q

Lamb dysentery is caused by Clostridium perfringens B

A

T

99
Q

Lambs have to be vaccinated with anatoxin vaccine in order to prevent lamb dysentery

A

F

100
Q

Lambs have to be vaccinated with attenuated vaccine in order to prevent lamb dysentery

A

F

101
Q

Pregnant ewes have to be vaccinated in order to prevent lamb dysentery

A

T

102
Q

Haemorrhagic diarrhoea is a clinical sign of lamb dysentery

A

T

103
Q

Lamb dysentery can be seen in lambs around weaning

A

F

104
Q

Lamb dysentery is found in 3-4-week-old lambs.

A

F

105
Q

Pathological lesions of Lamb dysentery starts in the colon

A

F

106
Q

We can culture the pathogen of Lamb dysentery from the intestines

A

T

107
Q

Lamb dysentery is caused by Clostridium dysenteriae

A

F

108
Q

Lamb dysentery can be seen in lambs after weaning

A

F

109
Q

There is no vaccine for the prevention of lamb dysentery

A

F

110
Q

Lamb dysentery occurs in 2-6 weeks old lambs

A

F

111
Q

For diagnosis of lamb dysentery, the pathogen should be cultured from the intestine

A

T

112
Q

Pathological symptoms of lamb dysentery can be found in the large intestines

A

F

113
Q

Lamb dysentery can be prevented by vaccinating pregnant ewes

A

T

114
Q

Lamb dysentery can be successfully treated with penicillin when clinical signs appear

A

F

115
Q

Lamb dysentery occurs in a week-old animal

A

T

116
Q

Lamb dysentery can be diagnosed by culturing the bacteria

A

T

117
Q

Newborn lambs have to be vaccinated in order to prevent lamb dysentery

A

F

118
Q

Toxoid vaccines can be used in the prevention of the disease

A

T

119
Q

Infection of lamb by secretion in the milk

A

F

120
Q

Lamb dysentery occurs in 1-2 weeks old lambs

A

T

121
Q

Struck is caused by Clostridium perfringens C.

A

T

122
Q

Overeating is a predisposing factor of struck

A

T

123
Q

Struck can be seen mainly in lambs younger than 2 weeks

A

F

124
Q

Struck is an acute disease in horses

A

F

125
Q

Struck is a zoonotic disease

A

F

126
Q

Struck is a slow disease of older sheep

A

F

127
Q

Struck is a worldwide common disease with great economic impact

A

F

128
Q

Infectious necrotic enteritis of piglets occurs in the first 1-2 weeks of life

A

T

129
Q

The lesions of Infectious necrotic enteritis of piglets can be seen typically in the large intestine

A

F

130
Q

Maternal protection is important in the case of Infectious necrotic enteritis of piglets

A

T

131
Q

There is no vaccination for the prevention of Infectious necrotic enteritis of piglets

A

F

132
Q

Pig enterotoxaemia can be prevented by vaccinating the pregnant sows

A

T

133
Q

Pig enterotoxaemia is caused by Clostridium perfringens C

A

T

134
Q

Pigs showing clinical signs of enterotoxaemia have to be treated with antibiotics immediately

A

F

135
Q

Lesions of pig enterotoxaemia can be seen in the small intestine

A

T

136
Q

Pig enterotoxaemia is more frequent in the litter of young than old sows

A

T

137
Q

Clostridium Enterotoxaemia of Piglets occurs in 2-4 days old piglets

A

T

138
Q

Pig enterotoxaemia can be generally seen in weaned piglets

A

F

139
Q

Necrosis of gut epithelium is a postmortem lesion of pig enterotoxaemia

A

T

140
Q

Clostridium enterotoxaemia of piglets is caused by C. perfringens

A

T

141
Q

Clostridium enterotoxaemia of piglets is more frequent in the case of first farrowing Sows

A

T

142
Q

Clostridium perfringens C causes infectious necrotic enteritis of piglets

A

T

143
Q

Infectious necrotic enteritis of piglets occurs in piglets after weaning

A

F

144
Q

The lesions of infectious necrotic enteritis of piglets can be seen generally in the small intestine

A

T

145
Q

Necrotic enteritis of piglets can be prevented by vaccination the sow with anatoxin

A

T

146
Q

Pig enterotoxaemia has to be diagnosed by detecting antibodies in the piglets

A

F

147
Q

Pig enterotoxaemia causes abdominal contractions in sows

A

F

148
Q

Mesenteric lymph node is congested in case of pig enterotoxaemia

A

F

149
Q

Clostridium enterotoxaemia can be cultured from mesenteric lymph nodes or gut

A

T

150
Q

Enteritis in piglets are caused by Clostridium perfringens D.

A

F

151
Q

Enteritis in piglets can be avoided by anatoxin vaccination

A

T

152
Q

Enteritis in piglets cannot be diagnosed by post-mortem, only by bacteriology

A

F

153
Q

Pig enterotoxaemia is caused by β-toxin production in 1st week of life.

A

T

154
Q

Pig enterotoxaemia can cause a high mortality

A

T

155
Q

Necrotic enteritis of piglets cannot be diagnosed by isolating the agent from the gut

A

T

156
Q

Pig enterotoxaemia is not present in Europe

A

F

157
Q

Pig enterotoxaemia cannot be prevented by using vaccines

A

F

158
Q

Pulpy kidney disease is caused by Clostridium perf. D

A

T

159
Q

Overeating is a predisposing factor to pulpy kidney disease

A

T

160
Q

The toxin of the agent of pulpy kidney disease is sensitive to trypsin

A

F

161
Q

Pulpy Kidney Diseases is caused by Clostridium chauvoei

A

F

162
Q

Pulpy kidney disease generally occurs in 1-2week old lambs

A

F

163
Q

Pulpy kidney disease can occur at any age

A

F

164
Q

Pulpy kidney disease of suckling lambs can be prevented by vaccinating pregnant ewes

A

F

165
Q

Sudden change the diet is a predisposing factor to pulpy kidney disease

A

T

166
Q

The toxin damages the endothelial cells in the case of pulpy kidney disease

A

T

167
Q

Neurological signs are typical in the case of pulpy kidney disease

A

T

168
Q

Isolation of the agent is necessary to the diagnosis of pulpy kidney disease

A

F

169
Q

Pulpy kidney disease is typically seen in lambs below 2 weeks of age

A

F

170
Q

Inactivated vaccines are used for the prevention of pulpy kidney disease

A

T

171
Q

Pulpy kidney disease is seen in piglets in the first week of life

A

F

172
Q

Pulpy kidney disease is a worldwide common disease

A

T

173
Q

Enterotoxaemia of sheep is also called pulpy kidney disease

A

T

174
Q

Cattle are not susceptible to this disease

A

T

175
Q

Vaccination are possible against pulpy kidney disease

A

T

176
Q

Coccidiosis is a predisposing factor of ulcerative enteritis in poultry

A

T

177
Q

Ulcerative enteritis of chicken is caused by Clostridium colinum

A

T

178
Q

Ulcerative enteritis is frequently seen in day old chicken

A

F

179
Q

Ulcers sometimes covered with pseudomembranes are frequent post mortem lesions of ulcerative enteritis of chicken

A

T

180
Q

Ulcerative enteritis can occur in 4-12-week-old chickens

A

T

181
Q

Clostridium perfringens is the causative agent of ulcerative enteritis in poultry

A

F

182
Q

Ulcerative enteritis of poultry is generally prevented with vaccination

A

F

183
Q

Lesions of ulcerative enteritis are mostly seen in the small intestines

A

F

184
Q

Ulcerative enteritis is a common disease in large scale farms

A

T

185
Q

Prevention of coccidiosis can help lower the incidence of ulcerative enteritis

A

T

186
Q

Coccidiosis is a predisposing factor of necrotic enteritis of chicken

A

T

187
Q

Foamy, brownish-red faeces is a clinical sign of necrotic enteritis of chicken

A

T

188
Q

Lesions of necrotic enteritis of chicken are typically occur in the large intestine

A

F

189
Q

Day-old chickens are widely vaccinated in order to prevent of necrotic enteritis

A

F

190
Q

Necrotic enteritis mostly occurs in chicken

A

T

191
Q

Waterfowl are not susceptible to necrotic enteritis

A

F

192
Q

Necrotic enteritis occurs in 1-3 weeks of age

A

F

193
Q

Tyzzer’s disease is caused by Clostridium piliforme

A

T

194
Q

Gangrenous dermatitis is caused by Clostridium septicum and Clostridium perfringens A

A

T

195
Q

Gangrenous dermatitis is caused by obligate pathogens

A

F

196
Q

Gangrenous dermatitis causes muscle oedema

A

T

197
Q

Vaccines are the primary way of prevention of gangrenous dermatitis

A

F

198
Q

Flaccid paralysis is a frequent clinical sign of tetanus

A

F

199
Q

The agent of tetanus is strictly anaerobic

A

T

200
Q

The agent of tetanus can enter the host through wounds

A

T