Epi Mix E 801-1000 Flashcards
1
Q
Malignant oedema can be treated with polymyxin
A
F
2
Q
Malignant oedema can be well treated with antibiotics over a long period
A
F
3
Q
gas gangrene (malignant oedema) is a regional illness
A
F
4
Q
The lesions of malignant oedema are mainly seen in the lungs
A
F
5
Q
Blackleg is caused by Clostridium septicum
A
F
6
Q
Lesions of blackleg are mainly seen on the claws
A
F
7
Q
Lameness is a clinical sign of blackleg
A
T
8
Q
Blackleg is a frequent disease in pigs
A
F
9
Q
Generally attenuated vaccines are used for the prevention of blackleg
A
F
10
Q
Anaculture or anatoxin vaccines are used for the prevention of blackleg
A
T
11
Q
Blackleg occurs only in tropical and subtropical countries
A
F
12
Q
Blackleg generally occurs in endemic
A
T
13
Q
Blackleg occurs most frequently in pigs
A
F
14
Q
Blackleg is a gas gangrene disease
A
T
15
Q
Blackleg is generally endogenous in sheep
A
F
16
Q
Blackleg is generally endogenous in cattle
A
T
17
Q
Movement disorders and lameness can be clinical signs of Blackleg
A
T
18
Q
Clostridium chauvoei can produce acids and gas from carbohydrates
A
T
19
Q
Blackleg occurs mainly in ruminants
A
T
20
Q
Oedema is a typical clinical sign of blackleg
A
T
21
Q
Live vaccines are used for the prevention of blackleg
A
F
22
Q
Blackleg infects ovine through wounds
A
T
23
Q
In Blackleg disease we use attenuated vaccine
A
F
24
Q
In the case of sheep, blackleg is generally consequence of a wound infection
A
T
25
Blackleg is caused by Clostridium chauvoei
T
26
Severe diarrhoea is the main clinical sign of blackleg
F
27
Blackleg occurs in cattle and sheep
T
28
If antibiotics are applied after appearance of the clinical signs of blackleg, treatment is generally successful
F
29
Blackleg disease occurs only in ruminants
F
30
Blackleg can usually be treated with antibiotics successfully
F
31
Blackleg in cattle is mainly endogenous between 6 months-3 years old
F
32
The disease caused by Clostridium chauvoei occurs mainly in cattle and sheep
T
33
The disease caused by Clostridium chauvoei is primarily the result of endogenous infection in cattle
T
34
Blackleg has four toxins
T
35
Blackleg can be prevented by using vaccine
T
36
We use neomycin and polymyxin to treat disease caused by Clostridium chauvoei
F
37
Blackleg in cattle is mainly endogenous between 2 months-2 years old
T
38
Blackleg in bovine is caused by wound infections
F
39
Classical swine fever is a frequent predisposing factor of bradsot
F
40
Oedema in the wall of the abomasum and duodenum are postmortem lesions of bradsot
T
41
Bradsot is caused by Clostridium chauvoei
F
42
Bradsot occurs mainly in tropical and subtropical countries
F
43
Soil contaminated frozen feed is a frequent predisposing factor of bradsot
T
44
Frozen food is a predisposing factor of bradsot
T
45
Bradsot occurs mainly late autumn and winter
T
46
Overeating can predispose the animals to bradsot
F
47
Thickening of and oedema in the stomach wall are typical lesions of bradsot
T
48
Aminoglycosides are successfully used for treatment in the case of bradsot
F
49
Bradsot is caused by Clostridium septicum
T
50
Severe pneumonia is a typical clinical sign of bradsot
F
51
Bradsot has a very fast course
T
52
Bradsot occurs only in suckling lambs
F
53
Bradsot is typically a chronic disease
F
54
Bradsot is common in the summer out on the pasture
F
55
Bradsot is an acute disease resulting in sudden death in many cases
T
56
We can use anaculture strain vaccine against Bradsot
T
57
Bradsot causes oedema of the legs and necrosis
F
58
Post mortem lesions of bradsot can be seen in the stomach (rennet).
T
59
Köves disease is an indicator disease
T
60
CSF is a predisposing factor of koves disease
T
61
Köves disease can be seen in pigs.
T
62
Köves disease is caused by Clostridium chavoei
F
63
Infectious necrotic hepatitis is mainly seen in pigs
F
64
Infectious necrotic hepatitis can be prevented by using anatoxin vaccines
T
65
Liver fluke can predispose animals to infectious necrotic hepatitis
T
66
In sheep, Clostridium septicum causes necrotic liver infection
F
67
Infectious necrotic hepatitis causes inflammation and necrotic nodules in the liver
T
68
There is no vaccine to prevent infectious necrotic hepatitis
F
69
Infectious necrotic hepatitis is caused by Clostridium septicum
F
70
Infectious necrotic hepatitis is mainly seen in suckling lambs
F
71
Parasite infection is a frequent predisposing effect of infectious necrotic hepatitis
T
72
Focal necrosis in the liver is a typical post mortem lesion of infectious necrotic hepatitis
T
73
Anatoxin vaccines can be used for the prevention of infectious necrotic hepatitis
T
74
Infectious necrotic hepatitis is caused by Clostridium novyi
T
75
Infectious necrotic hepatitis is spread by tick
F
76
Infectious necrotic hepatitis is caused by Clostridium novyi type B
T
77
Infectious necrotic hepatitis is found worldwide
T
78
Infectious necrotic hepatitis can be transmitted by liver flukes
F
79
Infectious necrotic hepatitis occurs mostly in young sheep
F
80
There is intravascular haemolysis in the case of bacillary haemoglobinuria
T
81
Bacillary haemoglobinuria is caused by Clostridium haemolyticum
T
82
There are no vaccines for the prevention of bacillary hemoglobinuria
F
83
Phospholipidase C is a virulence factor of the agent of bacillary hemoglobinuria
T
84
Bacillary haemoglobinuria is mainly seen in cattle
T
85
Bacillary haemoglobinuria is caused by Clostridium septicum
F
86
Jaundice and anaemia are important clinical signs of bacillary hemoglobinuria
T
87
Red urine is a typical clinical sign of bacillary hemoglobinuria
T
88
Bacillary hemoglobinuria is a slow, chronic disease
F
89
Bacillary hemoglobinuria can frequently be seen in horses
F
90
Clostridium novyi is the causative agent of bacillary hemoglobinuria
F
91
Bacillary hemoglobinuria causes severe haemorrhages
T
92
Bacillary hemoglobinuria are caused by infection from the soil
T
93
Lamb dysentery occurs in a week old animal
T
94
Isolation of the agent from the gut gives aetiologic diagnosis of lamb dysentery
F
95
Isolation of Cl. perfringens from the gut confirms the diagnosis of lamb dysentery
F
96
Lesions of lamb dysentery are generally seen in the large intestine
F
97
Lesions of lamb dysentery can be seen in the small intestine
T
98
Lamb dysentery is caused by Clostridium perfringens B
T
99
Lambs have to be vaccinated with anatoxin vaccine in order to prevent lamb dysentery
F
100
Lambs have to be vaccinated with attenuated vaccine in order to prevent lamb dysentery
F
101
Pregnant ewes have to be vaccinated in order to prevent lamb dysentery
T
102
Haemorrhagic diarrhoea is a clinical sign of lamb dysentery
T
103
Lamb dysentery can be seen in lambs around weaning
F
104
Lamb dysentery is found in 3-4-week-old lambs.
F
105
Pathological lesions of Lamb dysentery starts in the colon
F
106
We can culture the pathogen of Lamb dysentery from the intestines
T
107
Lamb dysentery is caused by Clostridium dysenteriae
F
108
Lamb dysentery can be seen in lambs after weaning
F
109
There is no vaccine for the prevention of lamb dysentery
F
110
Lamb dysentery occurs in 2-6 weeks old lambs
F
111
For diagnosis of lamb dysentery, the pathogen should be cultured from the intestine
T
112
Pathological symptoms of lamb dysentery can be found in the large intestines
F
113
Lamb dysentery can be prevented by vaccinating pregnant ewes
T
114
Lamb dysentery can be successfully treated with penicillin when clinical signs appear
F
115
Lamb dysentery occurs in a week-old animal
T
116
Lamb dysentery can be diagnosed by culturing the bacteria
T
117
Newborn lambs have to be vaccinated in order to prevent lamb dysentery
F
118
Toxoid vaccines can be used in the prevention of the disease
T
119
Infection of lamb by secretion in the milk
F
120
Lamb dysentery occurs in 1-2 weeks old lambs
T
121
Struck is caused by Clostridium perfringens C.
T
122
Overeating is a predisposing factor of struck
T
123
Struck can be seen mainly in lambs younger than 2 weeks
F
124
Struck is an acute disease in horses
F
125
Struck is a zoonotic disease
F
126
Struck is a slow disease of older sheep
F
127
Struck is a worldwide common disease with great economic impact
F
128
Infectious necrotic enteritis of piglets occurs in the first 1-2 weeks of life
T
129
The lesions of Infectious necrotic enteritis of piglets can be seen typically in the large intestine
F
130
Maternal protection is important in the case of Infectious necrotic enteritis of piglets
T
131
There is no vaccination for the prevention of Infectious necrotic enteritis of piglets
F
132
Pig enterotoxaemia can be prevented by vaccinating the pregnant sows
T
133
Pig enterotoxaemia is caused by Clostridium perfringens C
T
134
Pigs showing clinical signs of enterotoxaemia have to be treated with antibiotics immediately
F
135
Lesions of pig enterotoxaemia can be seen in the small intestine
T
136
Pig enterotoxaemia is more frequent in the litter of young than old sows
T
137
Clostridium Enterotoxaemia of Piglets occurs in 2-4 days old piglets
T
138
Pig enterotoxaemia can be generally seen in weaned piglets
F
139
Necrosis of gut epithelium is a postmortem lesion of pig enterotoxaemia
T
140
Clostridium enterotoxaemia of piglets is caused by C. perfringens
T
141
Clostridium enterotoxaemia of piglets is more frequent in the case of first farrowing Sows
T
142
Clostridium perfringens C causes infectious necrotic enteritis of piglets
T
143
Infectious necrotic enteritis of piglets occurs in piglets after weaning
F
144
The lesions of infectious necrotic enteritis of piglets can be seen generally in the small intestine
T
145
Necrotic enteritis of piglets can be prevented by vaccination the sow with anatoxin
T
146
Pig enterotoxaemia has to be diagnosed by detecting antibodies in the piglets
F
147
Pig enterotoxaemia causes abdominal contractions in sows
F
148
Mesenteric lymph node is congested in case of pig enterotoxaemia
F
149
Clostridium enterotoxaemia can be cultured from mesenteric lymph nodes or gut
T
150
Enteritis in piglets are caused by Clostridium perfringens D.
F
151
Enteritis in piglets can be avoided by anatoxin vaccination
T
152
Enteritis in piglets cannot be diagnosed by post-mortem, only by bacteriology
F
153
Pig enterotoxaemia is caused by β-toxin production in 1st week of life.
T
154
Pig enterotoxaemia can cause a high mortality
T
155
Necrotic enteritis of piglets cannot be diagnosed by isolating the agent from the gut
T
156
Pig enterotoxaemia is not present in Europe
F
157
Pig enterotoxaemia cannot be prevented by using vaccines
F
158
Pulpy kidney disease is caused by Clostridium perf. D
T
159
Overeating is a predisposing factor to pulpy kidney disease
T
160
The toxin of the agent of pulpy kidney disease is sensitive to trypsin
F
161
Pulpy Kidney Diseases is caused by Clostridium chauvoei
F
162
Pulpy kidney disease generally occurs in 1-2week old lambs
F
163
Pulpy kidney disease can occur at any age
F
164
Pulpy kidney disease of suckling lambs can be prevented by vaccinating pregnant ewes
F
165
Sudden change the diet is a predisposing factor to pulpy kidney disease
T
166
The toxin damages the endothelial cells in the case of pulpy kidney disease
T
167
Neurological signs are typical in the case of pulpy kidney disease
T
168
Isolation of the agent is necessary to the diagnosis of pulpy kidney disease
F
169
Pulpy kidney disease is typically seen in lambs below 2 weeks of age
F
170
Inactivated vaccines are used for the prevention of pulpy kidney disease
T
171
Pulpy kidney disease is seen in piglets in the first week of life
F
172
Pulpy kidney disease is a worldwide common disease
T
173
Enterotoxaemia of sheep is also called pulpy kidney disease
T
174
Cattle are not susceptible to this disease
T
175
Vaccination are possible against pulpy kidney disease
T
176
Coccidiosis is a predisposing factor of ulcerative enteritis in poultry
T
177
Ulcerative enteritis of chicken is caused by Clostridium colinum
T
178
Ulcerative enteritis is frequently seen in day old chicken
F
179
Ulcers sometimes covered with pseudomembranes are frequent post mortem lesions of ulcerative enteritis of chicken
T
180
Ulcerative enteritis can occur in 4-12-week-old chickens
T
181
Clostridium perfringens is the causative agent of ulcerative enteritis in poultry
F
182
Ulcerative enteritis of poultry is generally prevented with vaccination
F
183
Lesions of ulcerative enteritis are mostly seen in the small intestines
F
184
Ulcerative enteritis is a common disease in large scale farms
T
185
Prevention of coccidiosis can help lower the incidence of ulcerative enteritis
T
186
Coccidiosis is a predisposing factor of necrotic enteritis of chicken
T
187
Foamy, brownish-red faeces is a clinical sign of necrotic enteritis of chicken
T
188
Lesions of necrotic enteritis of chicken are typically occur in the large intestine
F
189
Day-old chickens are widely vaccinated in order to prevent of necrotic enteritis
F
190
Necrotic enteritis mostly occurs in chicken
T
191
Waterfowl are not susceptible to necrotic enteritis
F
192
Necrotic enteritis occurs in 1-3 weeks of age
F
193
Tyzzer’s disease is caused by Clostridium piliforme
T
194
Gangrenous dermatitis is caused by Clostridium septicum and Clostridium perfringens A
T
195
Gangrenous dermatitis is caused by obligate pathogens
F
196
Gangrenous dermatitis causes muscle oedema
T
197
Vaccines are the primary way of prevention of gangrenous dermatitis
F
198
Flaccid paralysis is a frequent clinical sign of tetanus
F
199
The agent of tetanus is strictly anaerobic
T
200
The agent of tetanus can enter the host through wounds
T
