Epi Mix N 2601-2800 Flashcards
Germinative infection can happen in the case of fowl typhoid
T
Animals infected with the agent of fowl typhoid can be recognised by using plate agglutination test
T
Lesions of fowl typhoid is limited to the gut
F
asymptomatic carriage of salmonella by birds is generally limited to the gut
T
Focal inflammation and necrosis can be seen in the liver in birds with fowl typhoid
T
fowl typhoid has a double peak death curve
T
fowl typhoid can be seen sporadically in zoo birds in Europe
F
omphalitis is a clinical sign of fowl typhoid
T
fowl typhoid is caused by salmonella gallinarum
T
arthritis can be a clinical sign of fowl typhoid
T
the agent of fowl typhoid can infect newly hatched chicken in the hatchery
T
Isolation of the agent of fowl typhoid from dead eggs prove infection of the herd with the agent
T
Fowl typhoid can be spread by aerogenic infection
T
In the case of fowl typhoid there is a peak of death cases between days 3 and 5
T
Fowl typhoid is an acute disease of poultry
T
Fowl typhoid is more frequent in water fowl than hens
F
The susceptibility to fowl typhoid is increasing with age.
F
Fowl typhoid has a death peak on days 3-5.
T
Fowl typhoid has no effect on hatching %.
F
Unabsorbed yolk sack is a post mortem lesion of fowl typhoid
T
Fowl typhoid is caused by Salmonella Enteritidis
F
Aerogenic infection occurs in the case of fowl typhoid
T
Arthritis is a clinical sign of fowl typhoid
T
Germinative infection is important in the epidemiology of fowl typhoid
T
Salmonella gallinarum/pollorum is the causative agent of fowl typhoid
T
After recovery from fowl typhoid the animals do not remain carriers
F
Fowl typhoid generally does not occur in large scale poultry farms
T
In case of Salmonella Gallinarum, germinative and aerogenic infection can happen
T
In case of fowl typhoid, the death curve peak at age 5-6 weeks
F
In fowl typhoid there are two peaks during the epidemic
T
Fowl typhoid can’t spread in a germinative manner
F
Fowl typhoid can be diagnosed locally with agglutination methods
T
Fowl typhoid is caused by Salmonella avium.
F
The agent of fowl typhoid is shed in the faces in high number
T
Asymptomatic carriage of Salmonella can occur in poultry
T
Fowl typhoid have an exponential loss curve
F
Fowl typhoid can infect the ovaries
T
White diarrhoea is a sign in young chickens with fowl typhoid
T
We can use slide agglutination to detect fowl typhoid
T
We can use tetracyclines to treat fowl typhoid
T
Fowl typhoid is caused by facultative pathogenic Salmonella
F
Rotting eggs are an important clinical sign of fowl typhoid
F
Dead eggs can be seen in fowl typhoid
T
Fowl typhoid can be seen only in adult birds
F
Decreased hatchability is an important clinical sign of fowl typhoid
T
Fowl typhoid propagates by germinative infection
T
Diarrhoea is not a typical clinical sign of fowl cholera
F
Fowl typhoid is usually a disease of waterfowl
F
The highest infection rate of fowl typhoid is between days 8-10
F
The susceptibility to fowl typhoid increases with age
F
Clinical signs of fowl paratyphoid are frequently seen in the laying period
T
Fowl paratyphoid is limited to the gut, there is no septicaemia
F
Fowl paratyphoid has been eradicated in Europe
F
Agent of fowl paratyphoid can cause generalised disease
T
Some viral infections can predispose animals to fowl paratyphoid
T
salmonella avium is the main aetiological agent of fowl paratyphoid
F
Germinative infection can occur in the case of fowl paratyphoid
T
Fowl paratyphoid is caused by facultatively pathogenic salmonellae
T
Infectious bursitis can predispose chicken to fowl paratyphoid
T
Fowl paratyphoid is caused by Salmonella Gallinarum/Pullorum
F
Fowl paratyphoid alone occurs at any age
F
Fowl paratyphoid is a septicaemic disease in chicken
T
Salmonella gallinarum is the causative agent of fowl paratyphoid
F
Fowl paratyphoid cannot be seen in the European poultry flocks anymore
F
Germinative infection happens in the case of fowl paratyphoid
T
Lesions of fowl paratyphoid occur only in the intestinal tract
F
Salmonella Enteritidis can cause fowl paratyphoid
T
Fowl paratyphoid occurs typically in 2-5 months old poultry
F
Germinative infection does not occur in the case of fowl paratyphoid
F
There are no vaccines to prevent fowl paratyphoid
F
Salmonella derby is a frequent agent of fowl paratyphoid
F
Clinical signs of fowl paratyphoid are mainly seen in adult birds
F
Clinical signs of fowl paratyphoid can be successfully stopped with antibiotics
T
The agent of fowl paratyphoid are facultative pathogenic bacteria
T
The clinical signs of fowl paratyphoid are mainly seen in birds below 4 weeks of age
T
Antibiotic treatment can prevent the carriage of salmonella, after fowl paratyphoid
F
Paratyphoid of poultry only affects the intestines
F
Paratyphoid of poultry affects adult animals mainly
F
The clinical signs of fowl paratyphoid can be seen mainly in adult hens
F
Fowl paratyphoid is a generalized disease with septicaemia
T
Isolation of the agent of fowl paratyphoid from the parenchymal organs results in aetiological diagnosis
T
Fowl paratyphoid is a rare and sporadic disease
F
Fowl paratyphoid mostly occurs in 0-2 weeks old chickens
T
Fowl paratyphoid infection occurs by PO or germinative routes
T
Purulent conjunctivitis can occur as a clinical sign in ducks with fowl paratyphoid
T
Fluoroquinolones can be used to treat fowl paratyphoid
T
Fowl paratyphoid can be spread by rodents
T
Fowl paratyphoid can occur alone only in the first 2 weeks of life
T
Live and attenuated vaccines can be used to prevent fowl paratyphoid
T
Fowl paratyphoid usually causes generalized disease
T
Fowl paratyphoid causes disease mainly in water fowl
F
We can certify “Salmonella-free” status of poultry stocks with serological tests
F
Fowl paratyphoid causes high morbidity mostly in water birds
T
Abortion is the main clinical form of human salmonellosis
F
Vomiting and diarrhoea are frequent clinical signs of human salmonellosis
T
All serotypes of salmonellae can cause salmonellosis in humans
F
Food of animal origin is a frequent source of human salmonella infection
T
Salmonella typhimurium can infect humans
T
Humans are infected with salmonellae mainly per os
T
Enteritis is a clinical sign of Salmonellosis
T
All salmonella serotypes are zoonotic
F
Age can be a predisposing factor in human salmonellosis
T
Gastroenteritis is the most common form of human salmonellosis
T
Human salmonellosis is always limited to the gastrointestinal tract
F
Poultry meat and eggs are common source of human salmonella infections
T
In the case of diarrhoea caused by salmonella in humans, penicillin treatment is recommended
F
Septicaemia is the most common presentation of human salmonellosis
F
We use tetracyclines in the treatment of human gastroenteritis caused by Salmonella spp.
F
Salmonellosis mostly affect the elderly and young people (children
T
Asymptomatic carriage of Pasteurella multocida can occur in birds
T
Pasteurella multocida is highly resistant; it can survive in the environment for several months
F
Pasteurella multocida can cause pasteurellosis in small ruminants
T
Pasteurella multocida can infect humans
T
Primary pasteurellosis disease have no predisposing factors
F
Pasteurella multocida can complicate canine distemper
T
Leukotoxin (cytotoxin) production is an important virulence factor of Mannheimia haemolytica
T
The dermonecrotoxin of Pasteurella multocida damages the osteoclast cells
F
Primary pasteurellosis diseases are caused by obligate pathogenic Pasteurella bacteria
F
Dermonecrotoxin can be virulence factor of Pasteurella multocida
T
Dermonecrotoxin of pasteurella multocida causes lesions in the nasal cavity and on the skin of pigs
F
Capsule can be virulence factor of Pasteurella multocida
T
Enterotoxins are virulence factors of Pasteurella multocida
F
The agent of acute systemic pasteurellosis is zoonotic
F
Mannheimia haemolytica leukotoxin (cytotoxin) damages the macrophages
T
Pasteurella ovis can cause pasteurellosis in small ruminants
F
Arthritis can be a clinical sign of ovine pasteurellosis
T
Transportation is a predisposing factor of respiratory pasteurellosis of calves
T
Primary pasteurellosis diseases have non infectious predisposing factors
T
Emboli are responsible for the necrosis seen in the case of acute systemic pasteurellosis
T
Leukotoxin is a virulence factor of Mannheimia haemolytica
T
Leukotoxin is produced by the majority of Pasteurella multocida
F
Pasteurella multocida can cause disease only in ruminants
F
Mannheimia haemolytica can cause disease mainly in ruminants
T
Bibersteinia trehalosi can infect ruminants
T
Pasteurellaceae are resistant bacteria, they remain viable for a long time in the soil
F
The virulence factors of P. multocida are the capsule and dermonecrotoxin
T
Pasteurella are very resistant in the environment
F
Pasteurella are obligate pathogens
F
We can use inactivated bacteria as a vaccine for Pasteurella
T
Pasteurella bovis can cause pasteurellosis in cattle.
F
Diarrhoea is the main clinical sign of bovine pasteurellosis
F
Overcrowding is a predisposing factor of respiratory pasteurellosis of cattle
T
The lesions of respiratory pasteurellosis of calves are generally in the diaphragmatic lobes
F
Dermonecrotoxin of the agents is responsible for the clinical signs of respiratory pasteurellosis of cattle.
F
Respiratory pasteurellosis occurs only among cattle above 6 months of age
F
Toxoid vaccines can be used for the prevention of respiratory pasteurellosis of cattle
T
Pasteurella multocida can cause pasteurellosis in cattle
T
mannheimia haemolytica can cause respiratory pasteurellosis of calves
T
Mannheimia haemolytica can cause pasteurellosis in cattle
T
Respiratory pasteurellosis can occur after transport
T
Mannheimia haemolytica is an important agent of shipping fever
T
Leukotoxin of M. haemolytica damages the alveolar macrophages
T
Interstitial pneumonia is a frequent post mortem lesion of pulmonary pasteurellosis of cattle
F
Salmonellosis can predispose cattle to pasteurellosis
F
Transportation can predispose cattle to pasteurellosis
T
Bovine pasteurellosis cannot be prevented with vaccination
F
Respiratory pasteurellosis has been already eradicated in Europe
F
Mannheimia haemolytica is a causative agent of respiratory pasteurellosis
T
Mycoplasmas can predispose cattle to respiratory pasteurellosis
T
Respiratory pasteurellosis in cattle is caused by P. multocida A and M. haemolytica A
T
Most importance virulence factor of M. haemolytica is leukotoxin
T
Respiratory pasteurellosis in cattle is a common disease of young animals
T
Macrolides are recommended to use in case of respiratory pasteurellosis in cattle
T
Respiratory pasteurellosis of cattle appears in calves aged 1-3 months old
T
Respiratory pasteurellosis of cattle causes a fibrinous inflammation of serous membranes
T
Leukotoxin of M. haemolytica strains responsible for Pasteurella pneumonia attacks respiratory epithelium
T
Respiratory pasturellosis of cattle are mostly seen in the tropics
F
Respiratory pasteurellosis of cattle causes purulent pneumonia
T
We can use ELISA to diagnose respiratory pasteurellosis in cattle
T
A predisposing factor of respiratory pasteurellosis in cattle is transport
T
A predisposing factor of respiratory pasteurellosis in cattle is the presence of a viral infection
T
Vaccination is widely used for prevention of bovine respiratory pasteurellosis
T
Haemorrhagic septicaemia of cattle is generally a chronic disease
F
Pasteurella multocida strains are causative agents of haemorrhagic septicaemia of cattle
T
Antibiotic treatment at the time of appearance of the clinical signs of haemorrhagic septicaemia of cattle is generally late
T
Bleeding from the nose is a frequent clinical sign of haemorrhagic septicaemia of cattle
F
Haemorrhagic septicaemia occurs most frequently among 2-3 month old calves in endemic areas
F
The morbidity and mortality of Haemorrhagic septicaemia of cattle is low
F
Oedema formation can be a clinical sign of haemorrhagic septicaemia of cattle
T
Hemorrhagic septicemia mainly occurs in tropical and sub-tropical areas.
T
Pasteurella multocida B:2 and E:2 strains are causative agents of Haemorrhagic septicaemia
T
Monsoon can predispose to haemorrhagic septicaemia
T
Haemorrhagic septicaemia is mainly seen in pigs and horses
F
Haemorrhagic septicaemia is mainly an acute disease
T
After recovering from haemorrhagic septicaemia the animals do not shed the agent
F
Exotoxins are responsible for haemorrhagic septicaemia
F
The agent of haemorrhagic septicaemia can be carried in the tonsils
T
Haemorrhagic septicaemia cannot be prevented with vaccines
F
Haemorrhages on the heart are important post mortem signs of haemorrhagic septicaemia
T
Haemorrhagic septicaemia is caused by Pasteurella multocida
T
Haemorrhagic septicaemia is mainly seen in pigs
F
Oedema can be seen in the case of haemorrhagic septicaemia
T
Haemorrhagic septicaemia is endemic in several European countries
F
Exhausting work can predispose to haemorrhagic septicaemia
T
Re-convalescent animals can carry the agent of haemorrhagic septicaemia
T
The lesions of haemorrhagic septicaemia are caused by the exotoxin of the agent
F
Aerogenic infection is frequent in the case of haemorrhagic septicaemia
T
Haemorrhagic septicaemia is caused by Mannheimia haemolytica
T
The leukotoxin of the agent is responsible for the lesions of haemorrhagic septicaemia
F
Fever is a typical sign of haemorrhagic septicaemia
T
