Epi Mix O 2801-3000 Flashcards
Haemorrhagic septicaemia occurs only in tropical and subtropical countries
F
The morbidity of haemorrhagic septicaemia is high
T
There are no vaccines for the prevention of haemorrhagic septicaemia
F
Haemorrhagic septicaemia is caused by B and E types
T
Haemorrhagic septicaemia of cattle generally occurs in tropical countries
T
Haemorrhagic septicaemia infects cattle mainly from the environment
F
Haemorrhagic septicaemia of cattle’s endotoxin causes clinical symptoms
T
Pasteurella multocida is the agent of haemorrhagic septicaemia of cattle
T
The clinical signs of haemorrhagic septicaemia are caused by the dermonecrotoxin of the agent
F
Buffalos and cattle are the most susceptible species to haemorrhagic septicaemia
T
Haemorrhagic septicaemia is mainly seen in 1-2 months old animals
F
Bleeding is the most typical clinical sign of haemorrhagic septicaemia
F
Haemorrhagic septicaemia can be prevented with vaccination
T
Haemorrhagic septicaemia infected animals carry the bacteria for 1 year
F
Hyaluronic acid capsule is important for the bacteria’s ability to replicate unhindered
T
Haemorrhagic septicaemia is a slow and chronic disease
F
Endotoxins are important for the pathogenesis of the haemorrhagic septicaemia disease
T
Antibiotics are highly effective against haemorrhagic septicaemia
T
Attenuated vaccines can be used to prevent haemorrhagic septicaemia
T
After recovery from haemorrhagic septicaemia, cattle remain carriers
T
Haemorrhagic septicaemia of cattle occurs only in tropical countries
F
Secondary infection can occur with haemorrhagic septicaemia of cattle
F
Haemorrhagic septicaemia of cattle can be explained by endotoxin production
T
Haemorrhagic septicaemia is mainly seen in sheep and horses
F
Septicaemic ovine pasteurellosis is mainly seen in pregnant animals
F
Acute systemic pasteurellosis occurs mainly among 3-12 months old lambs
T
Bibersteinia trehalosi can cause acute systemic pasteurellosis in small ruminants
T
Cough and nasal discharge are clinical signs of ovine pasteurellosis
T
Acute systemic pasteurellosis is caused by Pasteurella multocida.
F
Acute systemic pasteurellosis does not occur in goats only in sheep
F
Septicemia ovine pasteurellosis is mainly seen in lambs younger than 3 months
T
Mastitis can be a clinical form of ovine pasteurellosis
T
septicemia can be a clinical form of ovine pateurellosis
t
septicemic pasteurellosis doesn’t occur in goats only in sheep
F
respiratory pasteurellosis occurs in goats
T
ewes have to be culled after mastitis caused by mannheimia haemolytica because the udder cannot regenerate
F
inactivated vaccines can be used for the prevention of ovine pasteurellosis
T
respiratory viruses can predispose sheep to pasteurellosis
T
mannheimia haemolytica cause mastitis in goats
T
fibrinous pleuropneumonia is a postmortem lesion of ovine pasteurellosis
T
Respiratory pasteurellosis can occur in goats
T
Septicaemia can be a clinical form of ovine pasteurellosis
T
Dermonecrotoxin producing Pasteurella multocida can cause irreversible lesions in the nose of the pigs
T
Mannheimia haemolyitica can cause Pasteurellosis in small ruminants
T
Bibersteinia trehalosi can cause Pasteurellosis in small ruminants
T
Enteritis is a frequent clinical sign of ovine pasteurellosis
F
Mannheimia haemolytica can cause respiratory pasteurellosis of sheep
T
Fibrinous pneumonia is a common post mortem lesion of respiratory pasteurellosis
T
Mannheimia haemolytica can cause mastitis of sheep
T
Respiratory pasteurellosis of sheep cannot be prevented with vaccines
F
Mannheimia haemolytica is an important agent of ovine respiratory pasteurellosis
T
Leukotoxin of M. haemolytica is responsible for the clinical signs of ovine respiratory pasteurellosis
T
Lesions of ovine respiratory pasteurellosis can be seen in the anterior lobes of the lungs
T
Macrolide antibiotics can be used to the treatment of ovine respiratory pasteurellosis
T
Acute systemic pasteurellosis occurs in lambs that are younger than 3 months
F
Acute systemic pasteurellosis is caused by Mannheimia haemolytica
F
Acute systemic pasteurellosis occurs in sheep but not goats
F
Respiratory form of ovine pasteurellosis can be seen only in suckling lambs
F
Leukotoxin produced Mannheimia haemolytica is responsible for ovine mastitis
T
Acute systemic pasteurellosis is caused by Pasteurella multocida
F
Lesions of ovine respiratory pasteurellosis are typically seen in the diaphragmatic lobe of the lungs
F
Mannheimia haemolytica can cause pneumonia in sheep
T
Mannheimia haemolytica can cause acute systemic pasteurellosis in sheep
F
Mannheimia haemolytica can produce dermonecrotoxin
F
Mannheimia haemolytica can cause septicaemia in suckling lambs
T
Acute systemic pasteurellosis is caused by Bibersteinia trehalosi
T
Acute systemic pasteurellosis occurs in sucking lambs and kids
F
In the case of acute systemic pasteurellosis bacterium, emboli are formed in the blood vessels
T
Nasal discharge and coughing for a week are the main clinical signs of acute systemic pasteurellosis
F
Acute systemic pasteurellosis is caused by type A and D of Pasteurella multocida
F
Necrosis of the mucous membranes and focal necrosis in the parenchymal organs are typical lesions of acute systemic pasteurellosis
T
Antibiotics can be used for the treatment of respiratory pasteurellosis of sheep
T
Respiratory pasteurellosis in sheep cannot be prevented with vaccination
F
Se-deficiency is a predisposing factor in sheep for pasteurellosis
T
Mannheimia haemolytica ́s endotoxin causes respiratory pasteurellosis in sheep
T
In respiratory pasteurellosis in sheep the cranial lung lobes are affected
T
Pasteurellosis does not cause septicaemia
F
Acute systemic pasteurellosis typically occurs in 3-12 months old small ruminants
T
Bacterium emboli are responsible for the clinical signs and lesions of acute systemic pasteurellosis
T
Fibrinous pneumonia is the main post mortem lesion of acute systemic pasteurellosis
F
Bibersteinia trehalosi is involved in development of acute systemic pasteurellosis
T
Serology is widely used to diagnose Pasteurella in sheep
F
Penicillin can be used to treat ovine pasteurellosis
T
The septicaemic form of pasteurellosis is most common in sheep above 1 year
F
You can see croupus pneumonia in case of ovine systemic pasteurellosis
F
Systemic pasturellosis is seen in 2-4-week-old lambs
F
Pasteurella causes septicaemia in lambs
T
In suckling lambs, acute systemic pasteurellosis may occur
F
Pasteurellosis in the sheep may cause interstitial pneumonia
F
Pasteurellosis in the sheep can occur in the form of mastitis
T
Pasteurellosis in sheep may occur as a septicaemia
T
Emboli is the cause of sudden death in acute systemic pasturellosis
T
Vaccines can be used to prevent pasteurellosis in sheep
T
In the respiratory form of ovine pasteurellosis, haemorrhagic pneumonia is seen
F
Mycoplasmas predispose pigs to pulmonary pasteurellosis
T
Pulmonary pasteurellosis is more frequent in suckling piglets than in adults
F
Bordetella bronchiseptica predisposes pigs to pulmonary pasteurellosis
F
Actinobacillus pleuropneumoniae predisposes pigs to pulmonary pasteurellosis
T
Actinobacillus pleuropneumoniae of pigs can be chronic
T
Pasteurella multocida can cause haemorrhagic septicaemia in pigs
T
Pulmonary pasteurellosis of pigs is mainly caused by Mannheimia hemolytica
F
respiratory pasteurellosis of pigs is generally an acute, generalized disease
F
Vaccines can prevent pneumonia of swine caused by pasteurella
F
P. multocida A is causative agent of pneumonia caused by pasteurellosis in swine
T
Rabbit pasteurellosis is a generalised disease
T
Pasteurella multocida A and D strains can cause pasteurellosis in rabbits
T
Clinical signs of pasteurellosis in rabbits are most severe in new-born animals
F
The agent of rabbit pasteurellosis can cause septicaemia
T
Nasal discharge is a typical clinical sign of rabbit pasteurellosis
T
Subcutaneous abscesses can be seen in the case of rabbit pasteurellosis
T
Otitis media can be a clinical sign of rabbit pasteurellosis
T
Rabbit pasteurellosis causes only respiratory clinical signs
F
rabbit pasteurellosis is more frequent in large scale farms than in small ones
T
Mannheimia haemolytica causes rabbit pasteurellosis
F
Pasteurellosis is limited to the respiratory tract in rabbits
F
Bibersteinia trehalosi is the causative agent pasteurellosis of rabbits
F
Serous pneumonia is typical in the case of pasteurellosis of rabbits
F
Pasteurellosis is limited to the respiratory track in rabbits
F
Neurologic signs can be seen in the case of pasteurellosis of rabbits
T
Pasteurellosis is common in suckling rabbits
F
The poor quality of the air can predispose to rabbit pasteurellosis
T
Purulent bronchopneumonia is a frequent post mortem lesion of rabbit pasteurellosis
T
Fibrinous pneumonia can be seen during necropsy in case of pasteurellosis in rabbit
T
Pasteurellosis in rabbit causes high amount of nasal discharge
T
Pasteurellosis in rabbit is caused by P. multocida A
T
Europe is free from atrophic rhinitis
F
Dermonecrotoxin producing Pasteurella multocida can cause irreversible lesions in the nose of pig
T
Dermonecrotoxin producing Pasteurella multocida can cause irreversible lesions in the nose of pigs
T
Toxoid vaccines are used for the prevention of atrophic rhinitis
T
The block of the lachrymal channel is a clinical sign of atrophic rhinitis
T
The turbinate bones can absorbed in the case of atrophic rhinitis
T
Overcrowding can predispose pigs to atrophic rhinitis
T
The maxilla can be shortened in the case of atrophic rhinitis
T
The most severe clinical signs of atrophic rhinitis can be seen in suckling piglets
F
Atrophic rhinitis can be prevented by vaccinating the pregnant sows
T
The lesions of atrophic rhinitis are examined after sawing the nose behind the first premolar teeth
T
Clinical signs of a atrophic rhinitis appear if piglets are infected in the first few weeks of their life:
T
The mortality and the economic impact of atrophic rhinitis are high
F
The endotoxin of the agent is responsible for the clinical signs and lesions of atrophic rhinitis
F
Atrophic rhinitis in fattening pigs is caused by Bordetella bronchiseptica
T
In 4-6 months old pigs B. bronchiseptica strains cause severe pneumonia
F
Dermonectotoxin is an important virulence factor of B. bronchiseptica
T
Atrophic rhinitis cannot be prevented with vaccination
F
Block of the lacrimal channel is a typical sign of atrophic rhinitis
T
The dermonecrotoxin producing Pasteurella multocida strains are responsible for the severe lesions of atrophic rhinitis
T
The dermonecrotoxin of P. multocida inhibits the activity of the osteoclast cells
F
Atrophic rhinitis can be prevented by vaccinating the day-old piglets
T
Atrophy of the turbinate bones is a typical lesion of atrophic rhinitis
T
Isolation of the causative agent from the nose confirm the diagnosis of atrophic rhinitis
F
The effects of the dermonecrotoxin produced by Pasteurella multocida are reversible
F
Bordetella bronchiseptica and Pasteurella multocida cause atrophic rhinitis
T
Bordetella bronchiseptica causes reversible lesions in the nasal cavity of pigs
T
Infection of pigs with Bordetella bronchiseptica and Pasteurella multocida at any age can result in atrophic rhinitis
T
The mortality of atrophic rhinitis can be 50-60%.
F
Clinical signs of atrophic rhinitis can be seen if piglets infected first week of life
T
The lesions of atrophic rhinitis are reversible
T
The lesions of atrophic rhinitis are caused by an endotoxin
F
Hungary is free from atrophic rhinitis of swine
F
The clinical form of atrophic rhinitis can be seen if the piglets were few weeks old when infected
F
Bordetella bronchiseptica causes reversible changes in swine
T
At atrophic rhinitis the conchae absorb
T
Isolating Pasteurella multocida from pigs’ noses proves atrophic rhinitis
F
Atrophic rhinitis can be prevented with toxoid vaccine given at weaning
F
PM lesions of atrophic rhinitis can be examined after transverse cut of the nose
T
Atrophic rhinitis is examined PM by a longitudinal section of the nose
F
Animals showing signs of atrophic rhinitis remain carriers
T
Atrophic rhinitis can be prevented by vaccinating the sow
T
Atrophic rhinitis cause disease in swine and calves
F
Dermonectotocin in case of atrophic rhinitis acts on the osteoblast cells
T
Atrophic rhinitis is caused by a synergistic interaction between B. bronchiseptica and P. multocida D
T
B. bronchiseptica can cause immunosuppression
T
Toxoid vaccines can be used for prevention of atrophic rhinitis
T
B. bronchiseptica strains producing toxins causing serious lesions
F
The typical PM lesions of atrophic rhinitis are caused by B. bronchiseptica strain
F
Atrophic rhinitis is a common disease that causes severe losses
F
Atrophic rhinitis only occurs pigs that were infected as suckling piglets
F
Atrophic rhinitis is proven by isolating P. multocida
F
Tetracyclines can successfully be used to treat atrophic rhinitis
T
Wrinkles and torsion of nose is the most prominent clinical sign of atrophic rhinitis
T
Fowl cholera is caused by Pasteurella multocida A, D and F strains
T
Fowl cholera can occur is ducks and geese
T
The agent of fowl cholera can survive in water for a few days
T
Fowl cholera is caused by Pasteurella gallinarum
F
The mortality of fowl cholera is low
F
Arthritis is a clinical sign of fowl cholera
T
The resistance of the agent of fowl cholera is low
T
Fowl cholera occurs mainly in the winter in Europe
F
Pasteurella multocida strains with lower virulence can cause chronic fowl cholera
T
The most severe form of fowl cholera occurs in day-old birds
F
Waterfowl is more susceptible to fowl cholera than hens
T
Focal inflammation and necrosis in the liver is a common postmortem lesion on of fowl cholera
T
Wild birds can introduce the agent of fowl cholera into a herd
T
Fowl cholera has an exponential mortality rate
T
Fowl cholera is a generalized disease it causes septicaemia
T
Animals recovered after fowl cholera remain bacterium carriers
T
The dermonecrotoxin of the agent is responsible for the clinical sign of fowl cholera
T
Vaccine for the prevention of fowl cholera provide type specific protection
T
The agent of fowl cholera is an obligate pathogenic bacterium
F
Antibiotics cannot be used for the treatment of fowl cholera
F
human can introduce the agent of fowl cholera into a herd
T