Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

22 fall: path > Test 2. 13 liver 2 > Flashcards

Test 2. 13 liver 2 Flashcards

1
Q
A

Peritoneal-pericardial diaphragmatic hernia (PPDH)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q
A

biliary cyst

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q
A

congenital biliary cyst

green and yellow filled with bile

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

amyloidosis deposits — material in the — and causes atrophy of —

A

eosinophilic material

space of Disse

hepatocytes and dilation of the sinusoids

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

— can be associated with inflammatory conditions in other organ systems

A

amyloidosis

Amyloid proteins are abnormal proteins the body cannot break down and recycle as it does with normal proteins

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

breed predisposition for amyloidosis

A

Chinese Shar-pei dogs and Abyssinian, Siamese, other oriental breeds)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what can happen when you biopsy amyloid liver

A

Spontaneous or biopsy-induced liver rupture with hemorrhage and hemoabdomen

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

amyloid is stained with Lugol’s and appears

A

blue to black

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

how common are Portosystemic shunts/vascular anomalies

A

common

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

how common are infarcts in the liver?

A

rare

many different blood supply from portal vein and hepatic artery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

how common is Peliosis hepatis (telangiectasia)

A

incidental vascular anomaly in the liver

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

R sided heart failure will do what to the liver

A

passive congestion

chronic will cause necrosis of the central vein

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

liver

A

chronic passive congestion

decrease in oxygen causes necrosis and fibrosis around the central vein

nutmeg liver

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q
A

porto systemic shunt

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Intrahepatic portosystemic shunts more frequently found in — breed dogs

A

large

patent ductus venosus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Extrahepatic portocaval and portoazygous shunts more frequent in — breed dogs

A

small
cats and ferrets

leads to hepatic hypoplasia/microhepatica

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

— shunts leads to hepatic hypoplasia/microhepatica

A

Extrahepatic portocaval and portoazygous shunts

liver is depenent on growth factors from the GI tract to grow to full size without them it will be small- hypoplasia

18
Q

what does portosystemic shunt do to liver

A

leads to hepatic hypoplasia/microhepatica

The liver is dependent on growth factors from the GI tract including
epithelial growth factor (EGF), hepatic growth factor (HGF), insulin like growth factor (IGF), amino acids, and hormones from the pancreas (insulin and glucagon)

19
Q

histo of portosystemic shunt

A

small lobules. Hepatic arteriolar hyperplasia
(compensatory) and bile duct hyperplasia (bystander effect)

20
Q
A

portocaval shunt

see how small the liver is

21
Q

liver secondary to cirrhosis

A

acquired(secondary) shunt associated with cirrhosis

22
Q

— shunt liver is small because of lack of growth factors. — shunt liver is small secondary to primary hepatic disease that causes fibrosis

A

congenital portosystemic shunt

secondary (acquired) shunt

23
Q

Primary portal vein hypoplasia is caused by

A

portal veins ending prematurely

can effect lobe or entire liver

can be asymptomatic with
enzyme or bile acid elevations to severe
signs associated with portal hypertension

24
Q

signs of primary portal vein hypoplasia

A

asymptomatic with
enzyme or bile acid elevations to severe
signs associated with portal hypertension

25
Q

other names for primary portal vein hypoplasia

A

microvascular dysplasia

non-cirrhotic portal hypertension

26
Q
A

Peliosis hepatis

incidental finding

27
Q
A

acute hepatitis from canine adenovirus (CAV-1)

vaccine DAPP2

28
Q

kidney dog

A

canine herpesvirus

29
Q

Feline Coronavirus is also called

A

Feline Infectious Peritonitis Virus

30
Q

Murine Coronavirus is also called

A

Mouse hepatitis virus

31
Q

cow liver

secondary to —

A

rumen acidosis

bacterial liver abscesses

32
Q
A

Necrobacillosis

33
Q

Tyzzers disease is caused by

A

Clostridium piliforme

cause liquefactive necrosis

gram - (usually gram+)

34
Q

gallbladder

A

Fibrinous cholecystitis Salmonella

35
Q

caused by

A

Hepatic necrosis (“Infarct”) Clostridium hemolyticum

Bacillary Haemoglobinuria- focally extensive- red water

Black disease (C. novyi)- multifocal necrosis

36
Q

Black disease (—) cause multifocal necrosis

A

C. novyi

37
Q

flukes will travel through liver and leave trails that are — that allow clostridium to grow

A

anaerobic

38
Q
A

fungal hepatitis

39
Q

rabbit

A

Rabbit- Coccidiosis
Eimeria stediae

40
Q

rabbit

A

Hepatic coccidiosis - rabbit

Eimeria stediae
follow bile ducts

22 fall: path (46 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions