jeporady Flashcards

1
Q

This can be seen in both female dogs and cats.

A

pyometra

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2
Q

This disease is seen often in puppies in our ES.

A

Parvoviral enteritis

feline panleukopenia- punched out peyers patch

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3
Q

Found during an autopsy of a pig.

can’t peel off

A

Fibrinous/fibrinosuppurative pleuropneumonia

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4
Q

From the lung of a foal (lesion and cause).

A

Pyogranulomatous pneumonia caused by Rhodoccous equi

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5
Q

A “surgical emergency”

A

Histocytoma

will regress on its own

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6
Q

When you find one congenital anomaly, always look for another…

A

Palatoschisis (cleft palate) and Ventricular Septal Defect (VSD)

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7
Q

Don’t look a gift horse in the mouth.

A

Severe malocclusion – “wave mouth”

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8
Q

Not all masses are bad.

A

Pancreas – nodular acinar/exocrine hyperplasia &

Liver – nodular hyperplasia

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9
Q

This represents a chronic lesion.

A

Liver – cirrhosis

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10
Q
A

Horse, penis – squamous cell carcinoma

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11
Q

This is commonly seen in older small dogs

A

Mitral valve endocardiosis (with left atrial fibrosis due to regurgitation)

smooth, shiny, nodular
murmur, jet lesions, atrial dilation: eccentric hypertrophy, thrombosis, chordae tendineae rupture, atrial rupture

DEGENERATIVE LESION

King Charles Cav or older dogs

ENDOCARDITIS= aortic, friable, red →thrombosis, bacterial/ inflammation
can lead to ischemia and infarct in kidneys

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12
Q

An important thing to notice in an abdominal explore

A

Peritoneal cavity – carcinomatosis

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13
Q

cat heart

A

Hypertrophic cardiomyopathy (HCM)

big heart
thick LV wall = ↓ volume (concentric hypertrophy- from pressure overload), left atrial dilation and thrombosis

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14
Q

Eating is important.

A

Bone marrow – serous atrophy of fat; Heart – serous atrophy of epicardial fat

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15
Q

Triangles of death.

A

Kidney and spleen – acute infarcts

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16
Q

One possible cause of this lesion.
big meaty

A

Diffuse splenomegaly (“meaty spleen”)–
chronic infectious disease, prolonged bacteremia/septicemia
chronic hemolytic anemia
diffuse granulomatous disease
disseminated follicular hyperplasia
neoplasia (lymphoma)
storage material
extramedullary hematopoiesis

17
Q

Cat. Write the pathogenesis. Start with the morphologic diagnosis of lesion A followed by the steps to get to lesion B. Use arrows!

A

Lesion A: unilateral thyroid gland hyperplasia (adenomatous hyperplasia)

Lesion B: aortic thromboembolism (“saddle thrombus”)

PG: Adenomatous thyroid gland hyperplasia – increased thyroid hormone production (hyperthyroidism) – increased production of myocardial contractile proteins – myocardial hypertrophy (thyrotoxic cardiomegaly) – turbulent blood flow – cardiac thrombosis - thromboembolism lodges in distal aorta – aortic (“saddle”) thromboembolism